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If a patient inherits or develops a mutation in a tumor suppressor gene, the complementary allele must be deleted/mutated before cancer develops. This is not true of oncogenes. What is the genetic term?
Loss of heterozygosity
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what is the main gene involved in bladder cancer? is it a tumor suppressor gene or oncogene?
RAS-oncogene RAS becomes an oncogene when it is locked into an active state and does not allow hydrolysis of GTP after tyrosine kinase receptor activation by growth factors.
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If a tumor suppressor gene becomes mutated, which of the following is not true?
A “gain-of function” mutation has occurred.
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Is the VHL gene an oncogene or tumor suppressor gene? How does it work?
VHL is a tumor suppressor gene (VEHLL THEN = suppressive). It degrades the protein HIF-1alpha which induces VEGF, CA-IX -> formation of vasculature -> progression of tumor
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What occur when proto onco or tumor suppressor gene are mutated and why that occur
1. Uncontrollable growth, cell prolifilation, transformation 2. excape Apoptosis 3. This occur because they are not responding to ligand that tell it when to turn on and off or sometime death signal
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Mutations in the proto-oncogene ras and the tumor suppressor gene p53 a.) increase protein synthesis by the cell. b.) can improve the chance of avoiding cancer as one ages. c.) disrupt normal regulation of the cell cycle. d.) can enhance further mutations, which can develop into cancer.
c.) disrupt normal regulation of the cell cycle.
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Which mutated tumor suppressor gene is most likely to contribute to many types of cancer, including bladder, breast, colorectal and lung? a. p53 b. APC c. BRCA1 D. BRCA2
Which mutated tumor suppressor gene is most likely to contribute to many types of cancer, including bladder, breast, colorectal, and lung? a. p53 b. APC c. BRCA1 d. BRCA2
a. Mutations in the p53 tumor suppressor gene have been found in many cancers, including bladder, breast, colorectal, esophageal, liver, lung, and ovarian cancers. APC gene mutations increase a person’s risk for familiar adenomatous polyposis, which is a precursor for colorectal cancer. BRCA1 and BRCA2 mutations increase the risk for breast and ovarian cancer.
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Define each of the following genes and their distinctions, then explain how each type of gene can participate in the pathogenesis of cancer: tumor suppressor gene; oncogene; and, DNA repair genes.
Tumor suppressor gene-normally suppressor tumors, pRB, P53, recessive trait, loss of function mutation Oncogene-as mutated this can change characteristics of growth factors and receptors. dominant gene. Helps with proliferation DNA repair gene-multi step, genes normally encode enzymes that can proof read, when we lose their effectiveness increased changes of cancer
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What does a tumor suppressor gene do?
(stop) cell division
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