First Aid p. 395 Heme synthesis, porphyrias, and lead poisoning Essay Example
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Lead poisoning
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Affected enzyme: Ferrochelatase and ALA dehydratase
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Ferrochelatase and ALA dehydratase
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Enzymes affected in lead poisoning
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Lead poisoning
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Accumulated substrate: Protoporphyrin, δ-ALA (blood)
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Lead poisoning
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Presenting symptoms: Microcytic anemia (basophilic stippling), GI and kidney disease.
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Lead poisoning
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Presenting symptoms: Children—exposure leads to mental deterioration. Adults—environmental exposure (e.g., batteries, ammunition) headache, memory loss, demyelination.
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Acute intermittent porphyria
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Affected enzyme: Porphobilinogen deaminase
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Porphobilinogen deaminase
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enzyme affected in Acute intermittent porphyria
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Acute intermittent porphyria
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Accumulated substrate: Porphobilinogen, δ-ALA, coporphobilinogen (urine)
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Acute intermittent porphyria
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Presenting symptoms: Painful abdomen Port wine-colored urine Polyneuropathy Psychological disturbances Precipitated by drugs (e.g., cytochrome P-450 inducers), alcohol, starvation
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Acute intermittent porphyria
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Treatment: glucose and heme, which inhibit ALA synthase
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glucose and heme, (which inhibit ALA synthase)
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treatment of Acute intermittent porphyria
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Porphyria cutanea tarda
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Affected enzyme: Uroporphyrinogen decarboxylase
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Uroporphyrinogen decarboxylase
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Enzyme affected in Porphyria cutanea tarda
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Porphyria cutanea tarda
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Accumulated substrate: Uroporphyrin (tea- colored urine)
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Porphyria cutanea tarda
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Presenting symptoms: Blistering cutaneous photosensitivity. Most common porphyria.
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Sideroblastic anemia (X-linked)
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Lead poisoning
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Acute intermittent porphyria
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Porphyria cutanea tarda
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Lead poisoning
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aminolevulinic acid synthase
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aminolevulinic acid dehydratase
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porphobilinogen deaminase
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uroporphyrinogen decarboxylase
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ferrochelatase
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