Medical Microbiology Flashcard
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Gram Positive Cocci |
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Gram Negative Cocci |
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Gram Positive Rods |
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Gram Negative Rods |
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Acid-fast Bacteria |
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Spiral Bacteria |
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Rickettsiae |
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Mycoplasmas |
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Fungal |
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Viral |
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Cold and upper respiratory tract infection,viral pneumonia, influenza, hepatitis, HIV/AIDS, measles, Rubella, mumps, polio, rabies, Encephalitis, chickenpox, Shingles, Herpes, yellow fever, dengue fever, hemorrhagic fever, warts, smallpox, infectious mononucleosis, cytomegalovirus, infection, respiratory syncytical cirus infections, gastroenteritis, some cancers (retrovirus associated leukemias), hantavirus, ebola, SARS |
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Bacterial |
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Prorozoan |
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Helminthic |
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Scientists and what did they do |
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Five States of immunity, how do eact occur, example of each |
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Infection Starts low #'s, undect-non comm. (incubation 13-17 day ) High #'s, dect. & comm (comm 10-12 days) disease- prodronal, acute, convaloscent, death High #'s Low #'s ends- Kills or is in latent stage
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List current vaccines and which type they are (toxiod, killed,"whole cell" bacterial, attenuated viral, genetically engineered, ect), which ones will have side effects |
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Smallpox still exist why or why not, what about the smallpox virus |
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difference between treatment for diphtheria and the prevention, what about tetanus |
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targets for antimicrobics which drug classes and examples of drugs in eah class that attacks that target |
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Properties of drugs that make them useful |
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Bacteriostatic Vs. Bacteriocidal |
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Broad spectrum vs. Narrow spectrum |
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drug synergism vs. antagonism |
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what is PDR and what information does it contain? |
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Generic Vs. Brand |
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types of transmission, and what is the main way they are transmitted |
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why and how are bacteria becoming resistatant to drugs |
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MRSA so important |
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main types of anti-viral drugs |
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HAART |
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difference in infection rate, morbidity rate, and mortality rate, how are they calculated |
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Difference between prevalence and incidence |
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Nosocomial infections and why are they so common, can they be prevented, and how? |
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Universal Percautions, meant to protect who, who enforces them? |
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normal flora in the human body and where ? |
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Types of symbiosis exist with normal flora |
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stages of infectio and what happens in each stage |
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Acute v. Chronic disease |
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Infectious disease v. communicable v. contagious |
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Active v. latent |
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Primary v. Secondary infection |
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what is zoonose, examples |
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pathogens actions to infect people and cause disease |
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3 classes based on where they multiply in the body, some examples of obligate intracellular parasies |
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Virulence factors involved in bacteria being able to invade and multiply in human tissues |
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difference between bacterial exotoxins and endotoxins |
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exotoxins are the following and what effect do the they on the body, Botulism toxin, plague toxin, staph. Aureus Leukocidin, cholera toxin, tetanus toxin,streptolysin O, diphtheria toxin |
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physical barriers to infection. Chemical defenses, Microbial factors, why are these considered manily "1st line" defense |
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WBCs originate |
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main types of leukocytes |
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difference between B and T cells |
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What type of T cell is called T4 cell, why is it important |
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phagocytic cells kill pathogens |
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five clinical signs of localized inflammatory response,which is due diedesis |
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human organs are incolced in blood "clearance", what happens when bacteremia or viremia overwhelms this system |
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5 types of Ig molecules and what do they do, which is the largest |
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what does IgG molecule look like, what is the difference between heavy chain and light chain |
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Constant region v. Variable region |
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antibody do if it is acting as an opsonin, antitoxin, agglutinin |
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how do antibodies activate complement, what does complement do when activated |
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doctors use the IgM/IgG serum ratio |
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somethings that can compromise host defenses |
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autoimmune diseases, which sex are they most common in |
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if an allergic reaction occurs with 15 mins of exposure, is the response antibody or T cell mediated |
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Chlamydiae |
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