Medical Microbiology Flashcard
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| Acid-fast Bacteria |
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| Spiral Bacteria |
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| Rickettsiae |
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| Mycoplasmas |
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| Fungal |
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| Viral |
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| Cold and upper respiratory tract infection,viral pneumonia, influenza, hepatitis, HIV/AIDS, measles, Rubella, mumps, polio, rabies, Encephalitis, chickenpox, Shingles, Herpes, yellow fever, dengue fever, hemorrhagic fever, warts, smallpox, infectious mononucleosis, cytomegalovirus, infection, respiratory syncytical cirus infections, gastroenteritis, some cancers (retrovirus associated leukemias), hantavirus, ebola, SARS |
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| Bacterial |
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| Prorozoan |
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| Helminthic |
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| Scientists and what did they do |
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| Five States of immunity, how do eact occur, example of each |
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Infection Starts low #'s, undect-non comm. (incubation 13-17 day ) High #'s, dect. & comm (comm 10-12 days) disease- prodronal, acute, convaloscent, death High #'s Low #'s ends- Kills or is in latent stage
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| List current vaccines and which type they are (toxiod, killed,"whole cell" bacterial, attenuated viral, genetically engineered, ect), which ones will have side effects |
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| Smallpox still exist why or why not, what about the smallpox virus |
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| difference between treatment for diphtheria and the prevention, what about tetanus |
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| targets for antimicrobics which drug classes and examples of drugs in eah class that attacks that target |
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| Properties of drugs that make them useful |
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| Bacteriostatic Vs. Bacteriocidal |
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| Broad spectrum vs. Narrow spectrum |
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| drug synergism vs. antagonism |
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| what is PDR and what information does it contain? |
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| Generic Vs. Brand |
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| types of transmission, and what is the main way they are transmitted |
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| why and how are bacteria becoming resistatant to drugs |
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| MRSA so important |
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| main types of anti-viral drugs |
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| HAART |
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| difference in infection rate, morbidity rate, and mortality rate, how are they calculated |
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| Difference between prevalence and incidence |
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| Nosocomial infections and why are they so common, can they be prevented, and how? |
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| Universal Percautions, meant to protect who, who enforces them? |
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| normal flora in the human body and where ? |
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| Types of symbiosis exist with normal flora |
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| stages of infectio and what happens in each stage |
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| Acute v. Chronic disease |
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| Infectious disease v. communicable v. contagious |
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| Active v. latent |
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| Primary v. Secondary infection |
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| what is zoonose, examples |
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| pathogens actions to infect people and cause disease |
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| 3 classes based on where they multiply in the body, some examples of obligate intracellular parasies |
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| Virulence factors involved in bacteria being able to invade and multiply in human tissues |
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| difference between bacterial exotoxins and endotoxins |
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| exotoxins are the following and what effect do the they on the body, Botulism toxin, plague toxin, staph. Aureus Leukocidin, cholera toxin, tetanus toxin,streptolysin O, diphtheria toxin |
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| physical barriers to infection. Chemical defenses, Microbial factors, why are these considered manily "1st line" defense |
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| WBCs originate |
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| main types of leukocytes |
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| difference between B and T cells |
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| What type of T cell is called T4 cell, why is it important |
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| phagocytic cells kill pathogens |
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| five clinical signs of localized inflammatory response,which is due diedesis |
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| human organs are incolced in blood "clearance", what happens when bacteremia or viremia overwhelms this system |
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| 5 types of Ig molecules and what do they do, which is the largest |
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| what does IgG molecule look like, what is the difference between heavy chain and light chain |
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| Constant region v. Variable region |
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| antibody do if it is acting as an opsonin, antitoxin, agglutinin |
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| how do antibodies activate complement, what does complement do when activated |
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| doctors use the IgM/IgG serum ratio |
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| somethings that can compromise host defenses |
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| autoimmune diseases, which sex are they most common in |
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| if an allergic reaction occurs with 15 mins of exposure, is the response antibody or T cell mediated |
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| Chlamydiae |
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