Emergency Medicine Shelf – Flashcards

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Leading cause of pediatric morbidity
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Falls. Most common from 0-14
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Leading cause of pediatric morality
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MVA
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Anatomic differences of kids
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1. more compact organs 2. relatively compact head 3. more elastic ligaments
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kids response to shock
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Normal BP w/ early shock. Different baseline HR, BP Greater propensity for spinal cord injury without radiologic abnormality (SCIWORA) More heat loss (sa)
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AVPU
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A= awake V=verbal stimulation response P= painful stimulation U= completely unresponsive
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Wadell triad
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1. closed head injury 2. intraabdominal injury 3. mid-shaft femur fracture
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What predicts intracranial injury risk (should CT)?
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Seizures, low CGS, scalp hematomas (parietal/temporal), altered mental status. NOT LOC.
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SALTER-Harris
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severity scale based on epiphysis fx S=straight across A=above epiphysis L=low or beLow epiphysis T=Two or Through ER=erasure of growth plate or crush
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Greenstick fx
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soft bones in which the bone bends and only partially breaks.
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Buckle (torus) fx
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longitudinal force on kid bones. easier to see on lateral.
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Boxer's fx
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4th or 5th metacarpal. Close fist and look at hand alignment and look for finger malrotation.
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CRITOE
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Ossification centers of the elbow: C-capitellum=1yr, R-radial head=3yr, I-internal medial epicondyle=5yr, T-trochlea=7yrs, O-olecranon=9yrs, E-external (lateral) epicondyle=11yrs
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Supracondylar fx
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FOOSH (fall on outstretched hand) Gartland 1: non-displaced Gartland 2: displaced but intact Gartland 3: displaced with disruption or ant/post periosteum. Neurovascular compromise/comapartment syndrome
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Toddler fx
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Spiral or oblique fx through distal 3rd of tibia non-displaced. -Etiology: Often un-witnessed fall or minor trauma. -Sx: Limp. Pain with gentle twisting of LE or heel tap. -Tx: short or long leg cast for 3-4weeks
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Avulsion fx
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sudden traction forces by muscles results in avulsion of apophysis (twisted ankle or ligaments/muscles attached)
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Heat related illness
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Edema Tetany malaria rubra syncope cramps exhaustion
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Cold related illnesses
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frostnip pernio-chillblains Kibe (chilblains on heel) Trench foot Surfer's ear Frostbite
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Heat Stroke
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Core temp >40C w/ CNS dysfx in setting of environmental heat load
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Drugs that cause heat stroke
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Diphenhydramine (Ach) phenothiazines (dopamine blockers) Ethanol Cocaine Ecstasy
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Frostbite risk factors
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smoking, prolonged hand/arm vibration, chronic dz affecting vasculature
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Frostbite - appearance -dx
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waxy, hard, insensate. Blisters containing blood or clear fluid. Don't rub affected areas. -dx: clinical, angiography, technetium99
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Mild Hypothermia
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32-35C (89.6-95) Shivering, hyper-reflexia, cold diuresis
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Moderate Hypothermia
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28-32 C (82.4-89.6) CNS depression, paradoxical undressing, cardiac dysrhythmias develop (sinus brady, afib), Osborn J waves.
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Severe
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<28C (82.4) Pulmonary edema, oliguria, loss of reflexes, acidosis, hypotension, coma, ventricular fibrillation, asystole.
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Passive external rewarming
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Remove wet garments, apply blankets, let them shiver
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Active external rewarming
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heating pads, radiant heat lamps, warm baths
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Active internal rewarming
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cardiopulmonary bypass, ECMO, warm pleural and peritoneal irrigation, arteriovenous rewarming, esophageal rewarming tubes, endovascular rewarming
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Shock
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physiologic state characterized by inadequate oxygen delivery. Delivery isn't keeping up with demand
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CO
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HR X SV
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How does inadequate volume cause shock?
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Decreased preload (ex: hemorrhage, dehydration)
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How does inadequate resistance cause shock?
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Inappropriate low SVR (ex: SIRS, anaphylaxis, burns, Addisonian crisis)
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How does pump failure cause shock?
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Contractility or HR (ex: acute MI, arrhthymias, late sepsis)
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Normotensive Shock
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SBP40mmHg drop fro baseline. It's not about the pressure it's about the O2 demand
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Cryptic Shock
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Normotensive + high lactate = profound tissue hypoxia. SBP >90 or MAP >65. Lactate >4.
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SIRS criteria
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-Temp: 100.4 - HR: >90 - RR: >20 or PCO2<32 - wbc: 12 or bands >10%
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Severe Sepsis:
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Sepsis + Organ dysfunction (elevated Cr, INR, altered mental status, elevated lactate, hypotension that responds to fluids)
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Septic Shock
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Severe sepsis + hypotension. Hypotension does NOT respond to fluid (30cc/kg bolus)
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Early Goal-Directed Therapy (EGDT):
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Early antibiotics + resuscitation minimizes mortality in septic shock.
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Confusion
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Alteration in higher cerebral functions (memory, awareness, attention). Sx, not a dx
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Etiology of widespread cortical dysfunction --> confusion
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1. Primary intracranial disease 2. Systemic dz that affect CNS 3. Exogenous toxins 4. Drug withdrawl
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Coma
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complete failure of the arousal system with no sponatenous eye opening
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Coma causes?
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Brainstem dysfx and/or bilateral cortical dz
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Minimally conscious state
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inconsistent but discernable evidence of consciousness. Altered state. Able to follow commands/purposeful behaviors/simple commands
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Obtundation
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Awake but NOT alert. Psychomotor retardation
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Stupor
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Awaken with stimuli but little motor or verbal activity when aroused.
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Delerium
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Severe confusion associated with 1. Motor restlessness 2. Transient hallucinations 3. Disorientation 4. Delusions
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Delerium v Dementia
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Delerium always has an organic cause.
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Dementia
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stable course of confusion, slow onset, irreversible, progressive, no impairment of consciousness, alert, normal vitals
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Critical Life threatening AMS not to miss
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o Hypoxia/diffuse cerebral ischemia (CHF, MI, shock) o Systemic process: hypoglycemia o CNS infx o HTN encephalopathy o Elevated ICP
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Coma etiologies NOT to miss
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CNS: hemorrhage, stroke Infx: meningitis, sepsis Metabolic: hypoglycemia, DKA, HHNK
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"Big-Five" life threatening causes of chest pain
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1. ACS/MI 2. PE 3. PTX 4. Aortic dissection 5. esophageal rupture
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Spontaneous pneumothorax risk factors
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tall, young, thin, smoker
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PE risks
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long trips, pregnancy/OCP, cancer risks, virchow's triad (venous stasis, hypercoagability, endothelial damage) -- #1 risk factor= prior DVT/PE
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PE dx (ecg, xcr)
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- Ddimer: rule-in - ECG: sinus tachy! - XCR: normal or atelectasis. Wedge shaped infarcts hampton's hump and westermark's sign- pathopneumonic
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Aortic Dissection
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intimal tear with entry of blood into the media "dissects" between the intima and adventitia.
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Expressive aphasia, stroke (carotid ischemia), pain radiates to back, CXR: widened mediastinum
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Aortic dissection clinical picture
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Stanford classification (aortic dissection)
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A: involves Ascending aorta (w or w/o descending) 80% of dissections - older pts w/ HTN B descending aorta only - mar fan's, ehler Danlos, pregnancy
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Aortic Dissection physical exam
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unequal decreased or absent peripheral pulses, aortic insufficiency murmur
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Aortic Dissection treatment goals
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Fluid and BP control (beta blockers, nitroprusside)
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Cocaine-Related Chest pain -cause -tx
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-cause: coronary vasospasm - tx: benzodiazapem (NO beta blockers)
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Stable angina
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Transient, episodic chest discomfort that is predictable and reproducible
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Unstable angina (plaque rupture)
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Angina that is new in onset occurs at rest or is similar but somewhat "different".
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MI (occlusion) or NSTEMI
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Sub-sternal chest discomfort > 15 min associated with dyspnea, diaphoresis, light-headedness, palpitations, n/v (12.5% of MIs are clinically silent)
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Atypical MI symptoms
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old people feel weak inferior wall MI; only with N/V - not chest pain
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Cardiac markers and sensitivities
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CK-MB: Sensitive >90% for MI 5-6 hrs after sx Troponin-I: Sensitive, duration for days Troponin-M: less sensitive, duration for days
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Chest pain immediate goals tx
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1. ABC/Stabilization/resuscitation ("Oh MY": IV, O2, monitor, pulse-ox) 2. ECG 3. +/-CXR
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Healing timeline
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Initial epitheliization: 24-48h Peak collagen synthesis 5-7d Strength of wound: 5% at 2 weeks, 35% at one month
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Contaminated wounds
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saliva, stool, dirt, wood or other organic matter. No visual debris
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Kraissel's lines
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lines of minimal tension. Important for wound contractures
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Regional block (amides: lidoaine, bupivicaine)
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large areas: when needed to avoid tissue distortion. Areas where infiltration is painful (ex: digital block, plantar surface)
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Lidocaine with epi. -don't use it? -treatment
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-ears, nose, penis and toes -phentolymine
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Wound preparation steps
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1. Cleansing (saline under pressure 8psi) 2. Debridement is critical 3. Exploration is critical
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Non-absorbable sutures
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Type: Monofilaments Subtypes: Ethilon (nylon,black) Prolene (polypropylene, blue, springy)
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Absorbable
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Types: Monofilaments Subtypes: Vicryl Dexon
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Tdap
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Once, 20-65 yo. Pregnant women after 20weeks.
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dT
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Every 10 years or booster. Okay in pregnant women
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TIG
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if not full immunized and current (within last 10 years)
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Abx prophylaxis
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Generally not necessary, except cat bites, certain hand injuries, foot punctures
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Gas permeable dressings
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Abrasions, burns, road rash, ulcers
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Difficult sutures
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Palms Shins Joint involvement vermillion border
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When to close? Golden hour.
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Depends on clinical scenario. 8-12hrs
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Primary closure
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Clean wound
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Delayed primary closure
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wound infx concerns
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Healing by secondary intention
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Dirty wound. Least favorable will make a big scar.
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Wound lecture pearls
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NO absolute golden hour, tap water is as good as NS, do NOT soak in betadine, non-sterile gloves ok, hand wounds less than 2cm> big, bulky dressings as good as sutures
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Wound lecture pitfalls
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always remember ABC, look for assoc injuries, don't miss high pressure injuries, always assess for foreign body, abx v delayed primary closure for high risk wound and or co-morbid conditions
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Primary EMBU applications
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1. Cardiac/IVC/Shock 2. FAST and pneumothorax 3. Aorta 4. Gallbladder and biliary 5. First trimester pregnancy and female pelvis 6. Evaluation for urterolithiasis and acute renal failure 7. Procedural guidance 8. Devaluation for DVT 9. Ultrasound of lungs and pleurae 10. Musculoskeletal and soft tissue evaluation
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CT objective
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Id of specific organ injury and or free fluid and/or retroperitoneal andmusculoskeletal injury → IR or OR
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DPL objective
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id of abnormal presence of blood, intestinal contents - old school
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Supine watersheds
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RUQ: 4 spaces LUQ: 4 spaces Subxiphoid:1 Suprapubic:1
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RUQ and LUQ spaces
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1. Pleural 2. Subphrenic 3. Splenorenal/hepatorenal (morrison's pouch) 4. Inferior pole
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Subxiphoid space
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pericardial space
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Suprapubic space
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retrovesicular (P of Douglas)
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Metabolic causes of AMS
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Hypoglycemia Hepatic encephalopathy Thyroid dyxfx Alcohol withdrawal
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CV causes of AMS
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HTN, enceophalopathy MI, CHF, PE Hypoxia Hypercarbia
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Infection causes of AMS
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Pneumonia UTI Sepsis Meningitis
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Neurological causes of AMS
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Siezure Stroke CNS mass
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Urospesis tx
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abx and fluid resuscitation, admission (River protocol for goal directed therapy)
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Occlusive Stroke -etiology -dx -tx
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-embolus from heart -dx: stat CT (rule out hemorrhage) -tx: tPA within 4hrs
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Anterior infarct sx
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lower extremity weakness> upper
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MCA infarct sx
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upper extremity> lower extremity; aphasia (left side), right sided hemi-paresis and sensory loss, left hemi anopsia, gaze preference toward the stroke.
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Tip of the basilar stroke sx
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locked in syndrome
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Acute Bacterial Meningitis bugs
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young people: strep pneumo and neisseria meningitis old people: listeria
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Acute Bacterial Meningitis -dx -tx
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-dx: lumbar puncture (careful not to herniate) -tx: abx and dexamethasone
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Subarachnoid hemorrhage -sx -dx
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-sx: worst HA of life -dx: head CT/LP
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AAA surgery indication
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3cm=aneurysmal 5.5 cm elective surgery recommended
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AAA RF
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over 50yo, atherosclerosis, peripheral vascular disease (PVD), first degree relative with AAA
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AAA classic triad
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(1) abdominal pain (2) hypotension (3) syncope (more in thoracic)
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Arterial occlusive Acute Mesenteric Ischemia -etiology -sx -dx -tx
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-origin of the SMA -sx: pain out of proportion to exam -dx: angiography "thumbprinting" -tx: heparin, glucagon, intraarterial papaverine, laporatomy
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Venous occlusive Acute Mesenteric Ischemia -etiology -sx -tx
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-etiology: Starts in venous arcades and progresses to SMV -sx: slower onset than arterial -tx: Heparin, IV thrombolytics, thrombectomy
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Non-Occlusive acute mesenteric ischemia -etiology -sx -tx
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-etiology: "intestinal angina" after eating. -sx: Anorexia in elderly people or critically ill -tx: difficult. vasodilation, anticoagulate
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Melenemesis
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coffee ground emesis, partially digested blood
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UGIB causes
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PUD, gastric erosions, variceal bleeding, Mallory-weiss tear, esophagitis, duodenitis, aortoenertic fistula, renal disease
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UGIB high risk (re-bleed, mortality)
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Hgb: 110-120 BPM Age >60 Coagulopathy Co-morbidities such as cancer
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UGIB dx
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NG lavage: EXCEPT known esophageal varices, active PUD, Mallory-weiss tears, gastric bypass surgery Endoscopy: 12-24 hours if bleeding stops
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LGIB dx
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Anoscopy: localizes lesion to rectum Colonoscopy: procedure of choice, difficult to perform without bowel prep or if active bleeding Nuclear red blood cell scan Angiography
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Define fever in kids
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>38C (100.4F) - infant 38.5 (101.3F) -older kids
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Reasons for fevers
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Host defense Endogenous pyrogens (IL-1, IL-6): rheumatic disease, Exogenous pyrogens: LPS, cocaine, anti-cholinergics
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Hyperpyrexia
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extreme elevation of body temp ≥ 41.5°C due to high set point
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Hyperthermia
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results from uncontrolled temperature regulation (e.g. heat stroke), body temp rises above set point
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Febrile infant (<2yrs) agents
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E. coli, Group B strep, listeria monocytogenes, HSV 1, HSV 2
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Fever without source
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temp >39, normal physical exam (well appearing, no obvious bacterial illness, petechiae -- occult)
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Occult bacteremia
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this is rarely happens anymore because of vaccines (h flu, strep pneumo)
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Occult UTI -agents -rf
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-agents: E coli, gnr, enterococcus -RF: age < 12mos, duration of fever 2+days, uncircumsized males
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Fever Unknown Origin (FUO)
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elevated temperature but despite investigations by a physician no explanation has been found
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Septic workup in kids
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CBC, UA, CSF, CXR
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Febrile infant (29-56 days) mgmt
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Complete septic workup 28-56d: admission "Low risk" criteria: withhold abx "High risk" criteria: abx
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Febrile young child (2mos-3yrs) mgmt
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Well appearing: urine dip, UCx Ill-appearing non-toxic: CBC, urine dip Toxic appearing: septic work-up, immediate abx
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Febrile child (3+yrs)
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Similar to adolescents and adults
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Croup
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laryngotracheobronchitis always caused by a virus (paraflu) in pharyngeal epithelium that spread to larynx and produce mucus and edema. Sx: Seal-barking cough, steeple sign on xcr
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Croup tx
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1. cool mist/hot shower 2. dexamethasone 3. racemic epinephrine nebulized
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Asthma
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airway hyperresponsiveness --> dead space ventilation. hypercapnea & respiratory acidosis, end-expiratory wheeze
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Asthma tx
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Albuterol (beta 2 agonist) Ipratroprium (anticholinergic agent) Corticosteroids: use in ALL acute attacks Magnesium sulfate Terbutaline/Epinephrine: parenteral beta-agonists
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Bronchiolitis
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RSV invades nasopharyngeal epithelium cell-cell transfer to lower airway and cause mucus plugging, and edema sx: grunting, nasal flaring
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Bronchiolitis tx
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-Supportive care: O2, hydration, nasal suctioning, pulmonary toilet, ventilator -Albuterol, maybe? -Racemic epinephrine - Corticosteroids
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Pneumonia
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aspiration or hematogenous spread to lungs of organsism that cause exudative inflamm response, fibrin deposition. Sx: pleuritic chest pain
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Foreign body aspiration
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younger children into R main-stem bronchus, ball-valve effect sx: classic triad: wheezing, sudden cough, choking, diminished unilateral breath sounds
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Opioid drugs
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morphine derivatives (hydrocodone, methadone, meperidine, propoxyphene, fentanyl) tx: narcan/naloxone
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Opioid-like drugs
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tramadol, clonidine, imidazolidines
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Opioid toxidrome
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Pinpoint pupils, respiratory depression, lethargy to coma, bradycardia, hypothermia, borderline, hypotension
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Anticholinergic toxidrome
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block acetylcholine transmission Mad as a hatter, blind as a bat, red as a beet, hot as a hare, dry as a bone, full as a tick
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Anticholinergic drugs
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Benadryl diphenhydramine (OTC cold, sleep meds), antiparkinson, anticholinergic meds, benzotropine, Jinsom weeds, atropine tx: neostigmine
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Sympathomimetic toxidrome
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tachycardia, elevated bp, dilated pupils, hyperactive bowels, diaphoresis
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Sympathomimetic drugs
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Cocaine, amphetamines, anorectics, otc stimulants, herbal, PCP (nystagmus)
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Cholinergic toxidrome
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DUMBELS D: Diarrhea, diaphoresis U: urination M: miosis B: bradycardia, bronchorrhea E: emesis L: lacrimation S: salivation, seizures
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Cholinergic drugs
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organophosphates/carbamate pesticides, carbamate medicinals (donepezil, physotigmine, pyridostigmine), nerve gas agents
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Certified First responders
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first on scene firefighter, police etc. 45hrs of training. CPR, AED, splint and bleeding control.
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EMS curriculum
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Set by the Federal Department of Transportation
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EMS scope of practice
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set by each state
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EMS length of training
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minimums set by the DOT, but overseen by the states
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EMS certification
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states with local credentialing
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Emergency Medical Dispatcher
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24hrs of training. Provide "pre-arrival instructions" Medical priority dispatch.
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EMT-basic
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120+ hours of training. All CFR skills. O2 admin, basic extrication skills. Assist with patient's meds (NTG, MDI)
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EMT-I (intermediate)
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80-1000hrs (vary by state). IVs, some medications, advanced airway skills (ET, combi-tube)
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EMT-paramedic
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EMT-B training plus 1000+ hrs of training. Clinical rotations in ED, ICU, CCU, labor & delivery and anesthesia. Strict CME requirements. ET intubation, surgical airway, chest decompression, EKG
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EMS personnel
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EMT-B EMT-I EMT-P
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