Microbiology Cards – Flashcards

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Syphilis-agent
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Treponema pallidum
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Syphilis-Invasion
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Penetrates mucous membranes
Wounds/Hair follicles
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Syphilis Transmission
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Sex
congenital
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Syphilis Characteristics
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Primary-chancre
Secondary-Fever,jaundice
Tertiary-gummas, to brain
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Syphilis Test
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VRDL
(venereal disease research lab)
check for antibodies
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Syphilis Treatment
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penicillin (even though g-)
to affect protein synthesis
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Syphilis-how much reported
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one of the most reported
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Gonorrhea-agent
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Neisseria gonorrheae (g-)
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Gonorrhea Invasion
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Wounds
Attachment with pili to epithelial cells
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Gonorrhea Transmission
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Sex
fluid contact because very fragile diplococcus
passed to infants
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Gonorrhea Characteristics
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Discharge, burning, itching, sterility
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Infant Gonorrhea
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gonococcal opthalmia
can lead to blindness
treat with Silver Nitrate
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Chlamydial urethritis-agent
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Chlamydia tracomatis
(small intracell. parasite)
abnormal cell wall
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Chlamydia urethritis invasion
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Wounds
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Chlamydia urethritis transmission
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Sex
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Chlamydia Characteristics
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80-90% nonsymptomatic
discharge as gonorrhea
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Chlamydia in infants
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Chlamydial opthalmia and pneumonia
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Chlamydia Treatment
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Choroquinolones
erythromycin
Effect protein synth, and DNA gyrase
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Chancoid-Agent
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Haemophilus ducreyi
(g-) rod
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Chancoid Invasion
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Wounds
Contact with leisons
Bacteria go to lymph
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Chancoid Transmission
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Sex
Contact with leisons
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Chancoid Characteristics
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SOFT chancre
endemic in developing countries
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Lymphogora nuloma/venereum-agent
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Chlamydia trachomatis (different serotype)
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Lymphogora nuloma venereum Transmission
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Sex
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Lymphogora nuloma venereum Characteristics
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Fever, swelling, to lymph
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Contact Bacterial Diseases
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Transmitted by skin contact
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Leprosy-agent
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Mycobacterium leprae
(acid fast rod)
heat sensitive
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Leprosy-invasion
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Obligate intracell. parasite
grow inside macrophages
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Leprosy Transmission
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Skin contact, droplets
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Leprosy Characteristics
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Skin leisons
Inside bone in superficial regions
95% population has natural immunity
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Leprosy Treat
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2-3 antibiotics
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Leprosy Cultivation
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9 banded armadillos
hard to lab cultivate
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Abscess, boil Agent
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Stap aureus
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Abscess, boil Invasion
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Transient Normal flora
can enter wounds and become pathogenic
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Abscess, boil Transmission
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Skin contact wounds, spread by food handlers to be food poisioning
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Abscess boil Characteristics
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skin lesions, pus filled pockets
spread to blood and other organs
septicemia
can cause bone infection
can produce exotoxin (enterotoxin)
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Abscess Impetigo
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More in children with low immune system
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Abscess resistance
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MRSA- methicilin resistant s.a.
VISA- vancomycin resistamt s.a.
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TSS-agent
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Staph aureus strain that produces a toxin
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TSS invasion
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small wounds made with tampons
production of a toxin
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TSS characteristics
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Sunburn-like rash, fever, coma
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Bacterial Conjunctivitis-agent
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Haemophilus influenza III
(g-)rod
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Haemophilus influenzae III disease
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Pink eye
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Bacterial Conjunctivitis Transmission
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face to face contact, airborne droplets, towels
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Pink Eye characteristics
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very contagious
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Endogenous Bacterial Diseases
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normal flora mos become pathogenic in individuals with depressed immune systems
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Gangrene Agent
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Bacteroides fragilis
(g-) rod
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Gangrene Invasion
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Normally in large intestine
Injury goes to blood vessels
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Gangrene Characteristics
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Produces clots-necrosis
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UTI-Agent (primary)
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E.coli
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UTI Agents (secondary)
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Proteus, chlamydia
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UTI Invasion
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in urinary tract become pathogenic
belong in intestine
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UTI Characteristics
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Cystitis (bladder), urethritis (ureters), pyelonephritis (nephrons)
burn pain during urination
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UTI treat
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Sepro
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Dental Carries-Agents
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Strep mutans
Strep mitis
Strep salivaris
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Dental Carries-Invasion
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Biofilm (Plaque) formed on teeth
Bacteria produce acids that break enamel
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Dental Carries- Prevention
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Fluorides, decrease sugar in food (ferm produces acid)
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Stages of Biofilm Development
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Attach
Initial Colonization
Secondary Colonization
Mature Biofilm
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Nosocomial Infections
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acquired during hospitalization (caused by opportunistic organisms)
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Nosocomial Infecttions Passed by
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Hospital Staff
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3 Predisposing Factors for Nosocomial
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-wide variety mos present
-weak immune patients
-direct or indirect chain of transmission
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20% Nosocomial
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carried by hospital staff
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80% Nosocomial
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opportunistic
UTI/Wound infections
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