Lumbopelvic – Flashcards
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What are 4 components that may contribute to why LBP pts don't get better
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-core -multifidi -transversus -muscles that control specific segmental motion that regulates the shearing
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Threats to quality care for LBP
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-overuse of ineffective treatments -under-use of effective treatments -misuse of potentially effective treatments
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How can you correct overuse of ineffective treatments?
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decrease use which will improve quality of care
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How can you correct under-use of effective treatments?
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increase use which will improve quality of care
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How can you correct misuse of potentially effective treatments?
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improved decision making will improve quality of care
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Example of a treatment not often used for LBP that shows to be effective?
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high velocity manipulation
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STM/heat/US and LBP treatment effectiveness
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-not much evidence -should only use in conjunction with other treatments, to get tissue pliable, to calm system prior to exercise
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3 best treatments for LBP
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-mob/manipulation -direction specific exercise -stabilization approach
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Medical model of disease
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-pain is a reflex response to a physical stimulus -every symptom has an underlying stimulus -in order to alleviate symptoms, need to alleviate underlying stimulus
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Implications of LBP and imaging
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imaging may reveal a pathology that is not the direct cause of LBP that the pt presents with
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1st level of treatment-based classification
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is the pt appropriate for physical therapy management?
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2nd level of treatment based classification
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what is the level of acuity? staging of pt
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3rd level of treatment based classification
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what treatment should be used?
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3 options for the 1st level of classification
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-appropriate for PT (symptoms of mechanical origin) -requires consultation (med/psych) -requires referral (med/surg, psych)
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What happens if there are red flags on initial exam when classifying pt?
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referral/consult with medical specialist
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What happens if there are yellow flags on initial exam when classifying pt?
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fear avoidance? psych consult?
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How is acuteness determined?
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by the nature of the presenting symptoms and the goals for treatment
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Stage 1 second level classification characteristics (6)
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-acute -Oswestry >30 -unable to sit >30 min -unable to stand >15 min -unable to walk >1/4 mi
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Stage 2 second level classification characteristics (5)
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-subacute -Oswestry ~ 15-30 -able to sit, stand, walk -unable to perform complex task -no stage 1 findings
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Stage 3 second level classification characteristics (3)
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-Oswestry <15 -able to perform complex tasks -unable to perform demanding tasks
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Treatment goals for stage 1 second level classification
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-control pain -improve ability to perform basic mechanical functions -reduce disability
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Treatment goals for stage 2 second level classification
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-focus on impairment correction -further reduce disability -improve ability to perform complex functional tasks
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Treatment goals for stage 3 second level classification
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-restore higher level of function -improve ability to perform demanding activities
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Treatment for pt in stage 1 second level classifcation
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-mobilization -specific exercise -immobilization -traction
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Progression for pt in stage 1 to stage 2 second level classification
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-aerobic activity -eliminate impairments -trunk strengthening -general strengthening
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Manipulation classification characteristics
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-no symptoms below knee -onset 35
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Treatment for manipulation classification
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-manipulation -exercise
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Specific exercise classification characteristics
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-centralization phenomenon with movement exam -postural preference
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Specific exercise classification treatment
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-activities to promote centralization -ex. worse with sitting, better with extension => sit less, perform repeated extensions
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Stabilization classification characteristics
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-prone instability test -aberrant motions -hypermobility -age 91 -usually will be a young highly flexible pt
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Treatment for stabilization classification
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-stabilization ex. -turn on core muscles -decreased abnormal shearing in back -decrease pressure on pain producing structures
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Traction classification characteristics
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-neurological signs -leg symptoms -peripheralization with movement exam -crossed SLR
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Treatment for traction classification
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mechanical/auto traction to relieve neurological pressure
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What do you need to know in PMH for history of interventions?
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-have you had PT or other treatment before? -what did they do in PT/other treatment? -have they had injections?
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What to know about HPI
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-MOI -date of onset -has it happened before? -does it relate to time of day? -any correlation with eating? -meds
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Do you see a pt who is also seeing a chiropractor simultaneously?
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No - too much overlay; chances of hurting pt increase
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Visceral screen
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-urogenital -OBGYN -other viscera
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What are you screening for asking for change in b&b?
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cauda equina syndrome
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If the pt has a history that has restricted motion in joint opening or closing pattern, what do you suspect?
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facet syndrome
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If the pt has a history that has symptoms centralizing or peripheralizing with movement, how do you test and what do you suspect?
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-repeated motions testing -discogenic pain
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How will discogenic pain present with repeated motions testing?
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better in extension; worse in flexion
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If the pt has a history of pain in LEs that increases by extension and decreases by flexion, as well as increases in weight bearing positions, what do you suspect?
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spinal stenosis (unilateral = lateral; bilateral = central)
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If the pt has a history of sudden locking, catching, giving way during motion, or aberrant motions, what do you suspect?
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lumbar instability
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If the pt has a history of pain increased by stretching or muscular contraction with a history of trauma associated with it, what do you suspect?
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sprain/strain
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Cancer red flags
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-male > 50 yo -unexplained weight loss -prior history of cancer -poor response to conservative care -night pain that takes a long time to decrease upon waking
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Cauda equina syndrome red flags
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-L4-S1 dermatome and saddle region -progressive LE weakness (DF, PF, toe ext) -urinary/fecal incontinence, retention, sensory deficits
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Osteomyelitis red flags
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-immunosuppression -drug use -recent infection -increased body temp
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Fracture red flags
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-history of trauma -prolonged steroid use -> 70 yo
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Yellow flag
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-patients false beliefs about pain or injury, self efficacy, appropriate strategies -emotional distress -FABQ
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Blue flag
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-injured workers -low job satisfaction -conflicts with employer -conflicts with other workers -these may change how pt reacts to their pain
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Black flag
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-social issues -financial issues -incentive not to get better?
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0-20% on Oswestry
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minimal disability
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20-40% on Oswestry
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moderate disability
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40-60% on Oswestry
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severe disability
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60-80% on Oswestry
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crippled
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80-100% on Oswestry
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bed bound or exaggeration
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Minimal detectable change for Oswestry
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6 points
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When do you give a FABQ?
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to see if pt can fit in manipulation category
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When do you administer Roland-Morris disability questionnaire?
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in patients with LBP ONLY
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Score > 30 on McGill pain questionnaire
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symptom exaggeration
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Where is the location of stress found in spondylolisthesis, spondylolysis?
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pars articularis
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Why don't facet joints check AP shearing well?
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located in the sagittal plane mostly
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Are the IV foramen open or closed in flexion?
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open
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What happens to the annulus during flexion?
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pinches in the front, becomes taut in the back
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What happens to the nucleus pulposus during flexion?
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squishes posteriorly
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What happens to the spinal cord during flexion?
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elongates, increases dural tension
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Interspinous ligaments during flexion
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taut
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PLL in flexion
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taut; helps annulus keep disc in place
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Facet joints in flexion
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glide up and forward
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Facet joints when rotating R
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facet joints on R will separate and gap
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Type II lumbar SB/ROT and how the spine begins
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with a flexed lumbar spine, ipsilateral
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Type I lumbar SB/ROT and how the spine begins
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with a neutral or extended spine, contralateral
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How is the sacrum during R rotation?
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L side anterior, R side posterior
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Right on right torsion of ROA
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L side deep/stuck forward
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Where is right on right torsion of ROA more dysfunctional?
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lumbar flexion - sacrum moves backward and it becomes more asymmetrical
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Where is right on right torsion of ROA less dysfunctional?
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lumbar extension - sacrum moves forward and becomes more symmetrical
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Left on right torsion of ROA
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L side shallow/stuck backward
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Where is left on right torsion of ROA more dysfunctional?
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lumbar extension - sacrum moves forward, asymmetrical
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Where is left on right torsion of ROA less dysfunctional?
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lumbar flexion - sacrum moves backward, symmetrical
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Observation component of LBP exam
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-distress? -preferred posture -body type -posture assessment -sitting disposition -palpation for position -gait exam
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Grade 1 vertical compression test
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buckles with initial compression to bone pressure
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Grades 2-5 vertical compression test
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increase pressure by same increments
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Least specific to most specific assessment of lumbar mobility
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-AROM -PROM -PPIVM -PAIVM
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Capsular pattern of LS
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FB and SB contralateral; ROT ipsilateral
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Myosfascial pattern of LS
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FB and SB contralateral, more so than rotation
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What are you looking for with PSIS FB test?
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innominate hypomobility; tests mobility of the iliosacral joint
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What are you looking for with Marcher's, Gillet test?
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mobility of IS joint; ability of innominate to drop down and out when leg is lifted (if hypomobile, will stay still or go up)
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+ Flare test
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moves < 2.5 mm medial or lateral translation; reproduction of symptoms
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What does the leg swing assess?
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-innominate in the sagittal plane -hip flex/EXT -sacral FLEX/ext -L5 flex/EXT
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+ Slump sacral base test
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change in rotation/asymmetry or end feel *check thumb depth and spring *can also be done for innominate at PSIS
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If in extension, the L sacral base is shallow with hard end feel on the L and in flexion L sacral base is level with a soft end feel, what is the problem?
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left stuck on ROA
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If in extension, the L PSIS stops first with HEF and the R crest continues into anterior rotation, what is the problem?
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L innominate stuck posteriorly
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What is important to keep in mind about the sit/slump test for sacral base and innominate?
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-blockage can be at the hip -may need to raise table or slide to the edge if they have decreased hip flexion ROM -changes should occur in other positions as well (prone-->prone on elbow) -need to compare to other findings
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What is a significant leg length change in the supine to long sit test?
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at least 2.54 cm change
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If you have a right anterior rotation issue, what are your findings in supine?
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right leg longer
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If you have a right anterior rotation issue, what are your findings in long sit?
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right leg shorter
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If you have a right posterior rotation issue, what are your findings in supine?
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right leg shorter
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If you have a right posterior rotation issue, what are your findings in long sit?
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right leg longer
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If you have left anterior rotation issue, what are your findings in supine?
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left leg longer
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If you have left anterior rotation issue, what are your findings in long sit?
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left leg shorter
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If you have left posterior rotation issue, what are your findings in supine?
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left leg shorter
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If you have left posterior rotation issue, what are your findings in long sit?
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left leg longer
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What are lumbopelvic special tests based on?
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-static positional faults -movement deficits -symptom reproduction
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What type of test has the better interrater reliability for the SI joint?
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provocation tests
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What type of examination should you be doing for SI joint?
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-provocation testing -cluster testing
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What does a positive SI joint symptom reproduction test tell you?
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SI joint is the pain producing structure for your low back pain
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How do you identify stiffness dominant behavior?
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On a PA, identify R1 and R2
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How do you identify symptom dominant behavior?
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on a PA, if P1 and P2 comes before hitting R2; P1 is where they first notice pain and P2 is where they won't let you go any further
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Intervention protocol for symptom dominant behavior
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tissue health modalities; grade 1,2 mobs; no end range stretches
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What is the more reliable indicator for a problem when doing PAIVM testing?
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pain
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Neurological component to LBP exam
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-DTR -sensation -muscle test -nerve tension -gait -balance
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Indicator of neurological weakness
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-tested at end range -repeated testing has quick fatigability
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What can you test if unable to test great toe for neurological issues?
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hip abduction
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What if knee flexion and knee extension are weak?
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suspect musculoskeletal
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What if multiple myotomes are weak together in a group or near each other?
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space occupying lesion; cauda equina
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What does a positive crossed SLR test indicate?
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large herniated disc - poor prognosis
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Sciatic nerve bias position
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-hip flex -hip add -hip IR -knee ext -ankle DF
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Tibial nerve bias position
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-hip flex -hip add -hip IR -knee ext -ankle DF -foot eversion
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What is SLR a test for?
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disc herniation
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Sural nerve bias position
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-hip flex -hip add -hip IR -knee ext -ankle DF -foot inversion
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Common peroneal nerve bias position
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-hip flex -hip add -hip IR -knee ext -ankle PF -foot inversion
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What does Brudzinski-Kernig sign assess?
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meningeal irritation, dural irritation, nerve root involvement positive in meningitis
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What can give you a "positive" prone knee bend test?
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-tight quads -limitations in lumbar ext -limitations in SIJ -limitations in APT -femoral nerve issue
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Lateral femoral cutaneous nerve issue
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-distinct bordered area of sensation loss -entrapment = myalgia paresthetica
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Lateral femoral cutaneous nerve bias position
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prone hip ext adduction knee flex
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Saphenous nerve bias position
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prone -hip ext -abd -ER -knee ext -ankle DF -eversion
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Components of slump test
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-trunk flex -cervical flex -knee ext -ankle DF
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+ Quadrant sign
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closes down foramen to increase radicular symptoms
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What does the bike test assess for?
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neurogenic vs. intermittent claudication
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+ Bike test
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onset of LE symptoms that decreases when pt slumps = neurogenic claudication
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What if there is no change in symptoms when doing the bike test?
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vascular issue
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Superficial tenderness Waddell sign
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tender to light touch over a broad area
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Non-anatomic tenderness Waddell sign
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deep tenderness over broad area
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Simulation components of Waddell sign
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-axial loading -rotation (hips with trunk)
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Distraction component of Waddell
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SLR findings not consistent with slump findings
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Regional component of Waddell (weakness and sensory)
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multiple spinal levels involved without a diabetes dx
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Overreaction component of Waddell
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overt upon observation during the exam
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5 categories of Waddell
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-tenderness -simulation -distraction -regional -overreaction
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What is a positive Waddell finding?
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>= 3/5 criteria positive
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When do you give sustained pressure during mobilization?
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-stretching -addressing connective tissue
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Benefits of doing oscillations during mobilization?
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-provides neurophysiological effect -pt may tolerate this better than sustained pressure
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Perform joint mob, pt gets worse, what do you do?
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-hold and monitor -decrease 1 variable and repeat and re-examine
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Perform joint mob, pt gets slightly better, what do you do?
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repeat, re-examine
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Perform joint mob, pt gets much better, what do you do?
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hold and monitor
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Perform joint mob, no change, what do you do?
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-increase 1 variable -repeat -re examine
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What do you want to eliminate when performing joint mobs?
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extension - won't be able to feel anything
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Where do you block during side bending with finger block mob?
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edge of superior SP
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Mechanical pain syndrome
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symptoms reproduced through movement or position and, therefore, deemed to be a condition that is amenable to PT intervention
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Centralization phenomenon
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symptoms migrate from a more distal to a more proximal location *indicator of favorable prognosis
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In what syndrome does centralization happen?
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derangement syndrome
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What does centralization occur in response to?
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loading strategies - repeated movements or postures
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What is leg pain at intake a significant predictor of?
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chronic pain and disability
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Competent disc
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annular wall of disc is intact, indicating the ability to reduce the disc and centralize symptoms; otherwise it is an incompetent disc
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Directional preference
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direction of movement which causes symptoms to centralize or improve
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What is important about directional preference in the initial stages of care?
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it is the primary focus of intervention
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Lateral shift aka sciatic scoliosis
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displacement of the upper body relative to the lower body in the frontal plane
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What is lateral shift defined by?
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the side to which the upper body is displaced relative to the lower body
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MDT syndromes
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-postural -dysfunction -derangement
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Postural syndrome characteristics (4)
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-normal structures under abnormal stress -pain from prolonged poor postures -reduction of symptoms with correction of posture -no loss of motion
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Dysfunction syndrome (4)
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-adaptive shortening of soft tissue; restricted movements -partial loss of motion -end range pain without peripheralization; local pain (unless nerve root is adhered) -pain reduced when stress is released
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How do you want to move in dysfunction syndrome?
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towards pain - stretch it!
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How is dysfunction syndrome named?
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direction in which symptoms are reproduced
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Derangement syndrome (4)
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-alteration in contents of the disc -peripheralization of symptoms with specific motions -loss of motion, spinal deformity -variable symptoms (sometimes it hurts sometimes it doesn't)
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How do you want to move in derangement syndrome?
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away from pain
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Example of dysfunction syndrome
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arm in a cast for 8 weeks
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Example of derangement syndrome
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-disc -bony derangement -locking knee with meniscus
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Age range for derangement
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20-55
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Pain rating/frequency for derangement
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sometimes yes, sometimes no for intermittent and constant
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Local or referred pain for pain location of derangement?
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sometimes yes sometimes no for both
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Acute, subacute, or chronic for derangement?
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all 3
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Gradual onset or sudden onset for derangement?
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either
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Motor/sensory deficits, abnormal reflexes, dural signs for derangement?
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can be all of them, but not necessarily
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Range of motion loss for derangement?
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yes - obstructed
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Changes in pain location with repeated movements for derangement?
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yes
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Pain during movement with repeated movements for derangement?
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yes
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End range pain with repeated movements for derangement?
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yes
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Age range for dysfunction
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over 30
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Intermittent or constant pain for dysfunction?
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intermittent
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Local or referred pain for dysfunction?
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both
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Acute, subacute, or chronic for dysfunction?
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chronic
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Gradual or sudden onset for dysfunction?
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gradual
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Motor/sensory deficits, abnormal reflexes, or dural signs for dysfunction?
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dural signs
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ROM loss for dysfunction?
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yes - restricted
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Changes in pain location for dysfunction with repeated movements?
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no
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Pain during movement for dysfunction with repeated movements?
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no
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End range pain with repeated movements for dysfunction?
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yes
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Age range for postural
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under 30
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Intermittent or constant pain for postural?
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intermittent
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Local or referred pain for postural?
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local
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Acute, subacute, or chronic for postural?
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acute
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Gradual or sudden onset for postural?
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gradual
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Motor/sensory deficits, abnormal reflexes, or dural signs for postural?
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none
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ROM loss for postural?
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no
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Changes in pain location with repeated movements for postural?
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no
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Pain during repeated movement for postural?
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no
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End range pain with repeated movement for postural?
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yes
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What is important when testing repeated motions?
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-need to set a baseline -need location and intensity of symptoms
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What positions do you do repeated movement testing?
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-standing -lying down
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Progression of forces for MDT intervention
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-patient generated -patient generated with overpressure -therapist generated
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Extension progression to provoke symptoms (8)
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-prone lying over pillows -prone lying -POE -extension in lying -extension in lying w overpressure -therapist PA mob -therapist manip -extension in standing
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Flexion progression to provoke symptoms (4)
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-flexion in lying -flexion in sitting (gradually straighten legs out) -flexion in step standing (good for adherent nerve root) -flexion in standing
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Lateral progression to provoke symptoms (7)
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-prone lying with hips offset -extension in lying w hips offset -extension in lying w hips offset and overpressure -rotation mob in ext -rotation manip in ext -side glide in standing -rotation mob in flexion
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Spinal instability
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based on spinal pathology associated with excessive movement at the intervertebral or segmental level - abnormal shearing forces
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Segmental instability
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failure of passive restraints that function to limit segmental motion
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What can abnormally large intervertebral motions cause?
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-compression and/or stretching of inflamed neural elements -abnormal deformations of ligaments, joint capsules, annular fibers, end plates *have high density of nocioceptors!
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Function of passive subsystem/spinal column
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enough soft tissues/ligamentous structures to hold spine together in alignment and prevent excessive movement
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Function of active subsystem/spinal muscles
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stabilizing muscles that can turn on and control and create a force against abnormal shearing in a way that protects the spine
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Function of control subsystem/neural
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sensitivity and firing capability from a neurological perspective to control everything to protect the spine
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Components of Panjabi model
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-control subsystem -active subsystem -passive subsystem
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Clinical instability
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-significant decrease in the capacity of the stabilizing system of the spine to maintain the IV neutral zones within physiological limits which results in pain and disability -dysfunction in one or more of the stabilizing subsystems leading to an increase in size of the neutral zone
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Neutral zone
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part of range of physiological IV motion, measured from neutral position, within which spinal motion is produced with a minimal internal resistance
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Elastic zone
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after the neutral zone, where tissues begin to stretch and cause resistance to motion (R1)
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Hypermobility instability
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larger area where shifting/shearing occur before resistance is given
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Goal if spine is hypermobile
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increase stiffness for spine to protect itself from unstable movement
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Things that can affect passive subsystem
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-traumatic injury -lax ligaments -degenerative disc
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Things that can affect active subsystem
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disuse - not exercising or using muscles, so they become weak
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Things that can affect control subsystem
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-unable to recruit segmental mm so larger mm take over and cause strain -abnormal firing pattern resulting in inhibition (can be caused by pain) -altered motor programs that prevent accessing inner mm
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Flow sheet for a dysfunctional spine
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injury/degeneration/disease --> decrease passive and active stability --> decrease neural control --> guarding --> accelerated degeneration/abnormal mm loading/muscle fatigue --> chronic dysfunction/pain
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Indicators for neural control subsystem issues
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-changes in mm onset timing -changes in patterns of mm recruitment determined by EMG -changes in mm activation and spinal stiffness determined by biomechanical modeling -changes in kinematic patterns of spinal movement determined by visual observation or instrumented motion analysis
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Examination predictors for success for pts with spinal instability
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- 91 deg
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Examination predictors for some improvement for pts with spinal instability
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- + prone instability test -aberrant movement -hypermobility with lumbar spring test -FABQ physical activity subscale <9
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Examination predictors for no improvement for pts with spinal instability
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- - prone instability test -no aberrant movement -no hypermobility with lumbar spring test -FABQ physical activity subscale > 9
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What 3 components provide compressive force and increased stability to spine?
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-thoracolumbar fascia -transversus -multifidus
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Transversus abdominis function
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attachment to lateral raphe of thoracolumbar fascia allows it to apply compressive force to fascia
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Multifidus function
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exerts a pushing force on thoracolumbar fascia and produces a hydraulic amplifier mechanism
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The cylinder, with top and bottom, components of spinal stabilizers
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-diaphragm -transversus + thoracolumbar fascia + multifidus -pelvic floor
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What happens to other parts of the cylinder if one component of the horizontal portion is having an issue?
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loss of pelvic floor control
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(+) passive lumbar extension test
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pain or feeling of instability
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What does active SLR test test?
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pelvic instability
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(+) active SLR test
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symptom reproduction with active SLR; symptom reduction with active SLR + directed force through ilia
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In LPM, what is the diagonal direction determined by?
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what foot is forward
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Where do you apply pressure for flexion LPM?
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coracoid
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Where do you apply pressure for extension LPM?
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upper scapula area
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What do proprioceptors need to do in LPM?
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detect abnormal shearing force and respond to protect before reaching an abnormal position
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Grade 0 LPM
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no/poor initiation
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Grade 1 LPM
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lumbar shears with initial compression to bone pressure
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Grades 2-5 LPM
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increase pressure by same increments
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Muscle recruitment phase of TA drawing in
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deep stabilizers only
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Force output phase of TA drawing in
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6 mmHg - 8 mmHg
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Endurance phase of TA drawing in
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10 sec x 10 reps
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Position phase of TA drawing in
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prone, supine, sit, stand
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Contraction phase of TA drawing in
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isometric to isotonic
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Extremity activity phase of TA drawing in
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UE, LE, superficial movers
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Function phase of TA drawing in
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critical ADL
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Formal skill training for the TA
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drawing in the abdomen with concurrent multifidus contraction
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Integration into dynamic function for the TA
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deep muscle provide support while superficial muscles perform movement
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Incorporation of skill into heavy tasks for the TA
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deep muscles stabilize while focus is on restoring superficial muscle function
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Type of exercise to isolate LMS
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local segmental control
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Type of exercise to train LMS control
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closed chain segmental control
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Train LMS functionally
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open chain segmental control
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Components to assess stability
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-upper ab MMT -lower ab MMT -trunk extensor MMT -TADIM -multifidus test -aberrant movement? yes or no -prone instability test -SLR -active SLR
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Diagnoses that are flexion biased
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-MDT flexion dysfunction -MDT anterior derangement -TBC flexion syndrome -central stenosis -lateral stenosis -spondylolisthesis/lysis -paravertebral myofascial pain syndrome -facet joint spondylosis -opening/tension syndrome with low reactivity -closing/compression syndrome with high reactivity -degenerative disc disease without HNP
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Progression for flexion-biased regimen
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unloaded --> partially loaded --> loaded --> lateral component
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Diagnoses that are extension biased
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-MDT postural syndrome -MDT extension dysfunction syndrome -MDT posterior derangement syndrome -TBC extension syndrome -bulging or HNP -closing/compression syndrome with low reactivity -opening/tension syndrome with high reactivity
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Progression for extension biased regimen
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unloaded --> partially loaded --> loaded --> lateral component
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Examples of unloaded flexion biased exercises
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-PPT -SKTC -DKTC
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Examples of partially loaded flexion biased exercises
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-cat stretch -prayer stretch -seated PPT -seated flexion
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Examples of loaded flexion biased exercises
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-standing PPT -standing flexion
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Examples of lateral component of flexion biased exercises
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-LTR -prayer stretch 3 ways -seated flexion with SB -standing flexion with SB -side glide in stand -SL stretch with bolster -million dollar roll
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Examples of unloaded extension biased exercises
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-prone lying -APT (rarely) -POE -PPU
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Examples of partially loaded extension biased exercises
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-cow -seated APT -seated extension
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Examples of loaded extension biased exercises
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-standing APT -standing extension
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Examples of lateral component of extension biased exercises
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-LTR -prone lying with hips offset -POE with hips offset -PPU with hips offset -side glide in stand -SL stretch with bolster -million dollar roll
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What does the fascial system do?
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-ensheathes and permeates all tissues and structures -supplies mechanical supportive framework that holds and integrates body together and gives it form -provides for the space and lubrication between bodily structures -creates pathways for nerves, blood, lymphatic vessels
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What do fascial cross links do?
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immobilize what used to be a functional joint - limits extensibility
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How do soft tissue restrictions occur?
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-cross linking -scar tissue -dehydration of ground substance -lymphatic stasis -neuro changes (mechanoreceptors) throughout system -cellular electrochemical changes (cellular function, membrane permeability) -pain (at injured site, referral patterns)
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What do golgi mechanoreceptors respond to?
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-GTO muscle contraction -other golgi receptors - strong stretch
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Possible techniques to affect golgi mechanoreceptor
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-active contraction -STM with active movement or dynamic stretching
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Know results of stimulation of golgi mechanoreceptor
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tonus decrease
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What do Pacini mechanoreceptors respond to?
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-rapid pressure changes -vibration -fast stimulation
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Possible techniques to affect Pacini mechanoreceptors
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-HVLA -oscillatory mob
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Known results of stimulation to Pacini mechanoreceptors
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increased sense of kinesthesia
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What do Ruffini mechanoreceptors respond to?
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-sustained pressure -tangential forces (lateral stretch)
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Possible techniques to affect Ruffini mechanoreceptors
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-trigger point release -deep fascial mobilization
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Known results of stimulation for Ruffini mechanoreceptors
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inhibition of sympathetic activity and increased local proprioception attention
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What do interstitial mechanoreceptors respond to?
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-rapid and sustained pressures -even split between high and low mech. threshold units
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Possible techniques to affect interstitial mechanoreceptors
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-low level group = gentle massage -high level group = deep fascial work, HVLA
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Known results of stimulation of interstitial mechanoreceptors
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-vasodilation -increase in plasma extravasation