assessing the need for O2 therapy – Flashcards

is an arterial hypoxemia that quickly responds to O2 Therapy and is often a result of V/Q mismatch

is an arterial hypoxemia but is not responsive to 02 therapy, which is indicated when there is a 20% increase of FiO2 that results in less than <10 mmHg increase of PaO2, and is a result of true shunting

Refractive Hypoxemia

True Shunting and V/Q mismatches

inadequate quantities of 02 at the arterial level

is inadequate quantities of O2 at the tissue level

Maintain adequate tissue oxygenation while minimizing cardiopulmonary workload

Tachycardia, hypertension, tachypnea and hyperpnea, pulomonary hypertension, vasoconstriction within extremities, vasodilation of the heart and brain, cyanosis, polycythemia, confusion, lethargy, somulance, lactic acid production when accompanied by poor perfusion

Develops after recieving high FiO2 / greater than 60% for 12-24 hours and is caused by overproduction of oxygen free radicals which inflict damage on the walls of the alveoli

substernal pain,decreased vital capacity,cough, signs of bronchopneumonia or tracheobronchitis, an increasing PAO2 and PaO2 with a decreasing diffusing capacity

use the lowest possible FiO2 that will meet the patient's tissue oxygenation needs

Oxygen Toxicity, Depression of Ventilation,Retinopathy of Prematurity, Absorption Atelectasis

in only some COPD patients with chronic hypercapnia, which causes high HCO3 retention.

Decreased RR and Vt, an increased PaO2,decreased pH, lethargy and confusion

Their drive for breathing has been suppressed because their disease state has change their physiologic drive to breathe from the normal drive to that of a hypoxic drive. Their normal has been reduced to a PaO2 of 50 to 65 mmHg and SpO2 of 85-90% , therefore, 100% iO2 caused their body to think they have more than enough oxygen so they don't need to take a breath therefore suppressing their spontaneous need for respiration

We keep THIS AT RISK COPD patient at the lowest possible FiO2 to bring them up to the rang of 50 to 65 PaO2

We can run an arterial blood gas to check for a high bicarbonate level

Retinal detachment in the premature or low birth weight neonate which is caused by vasoconstriction , hemmorahage, and the eventual scarring of the blood vessels of the eye which causes blindness

a High FiO2 in the premature or low birth weight neonate

below 80mmHg

depletion of nitrogen that typically holds the alveoli open, causing the alveoli to become poorly ventilated, or shuntlike by lacking the required nitrogen to keep them fully open. O2 is then removed in larger quantities than normal ventilation can replenish