Nursing Care of the Patient with a Pregnancy Complication – Flashcards

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Danger Signals of Pregnancy
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Any bleeding from the vagina Gush of fluid from vagina (clear, not urine) Regular contractions occurring before completion of 37 weeks Severe headache or changes in vision Epigastric pain Vomiting that persists & is severe Change in fetal activity patterns Temperature elevation, chills, or "sick" feeling indicative of infection Swelling in upper body, especially face & fingers
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First Trimester Complications of Pregnancy
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Abortion (Miscarriage) Incompetent cervical OS Ectopic pregnancy Gestational trophoblastic disease (hydatidiform mole)
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Abortion (Miscarriage)
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Loss of pregnancy before viability of fetus, may be spontaneous (miscarriage), therapeutic or elective
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Abortion (Miscarriage) Assessment Findings
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Vaginal bleeding Contractions, pelvic cramping, backache Lowered hemoglobin if blood loss is significant Passage of fetus/tissue
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Nursing Intervention for Abortion (Miscarriage)
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Save all tissue passed Keep patient at rest & teach reason for bed rest Increase fluids PO or IV Prepare patient of surgical intervention if needed Provide discharge teaching about limited activities & coitus after bleeding ceases Observe reaction of mother & others, providing emotional support & give opportunity to express feelings of grief & loss
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Therapeutic Management of Abortion (Miscarriage)
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Depends on situation: Inevitable abortion Incomplete abortion
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Inevitable Abortion (Miscarriage)
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Vacuum curettage or dilation & curettage (D&C)
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Incomplete Abortion (Miscarriage)
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D&C with Pitocin or methergine
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Incompetent Cervical OS
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*Painless* condition in which cervix dilates without uterine contractions & allows passage of fetus, usually the result of prior cervical trauma/biopsy
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Medical Management of Incompetent Cervical OS
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May be treated with cerclage
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Cerclage
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Suturing of the cervix closed until patient is ready to give birth to term baby When the patient goes into labor, the suture is removed for vaginal delivery
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Incompetent Cervical OS Assessment Findings
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History or repeated, relatively *painless* abortions Early & progressive effacement & dilation of cervix Bulging of membranes through cervical OS
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Nursing Interventions for Incompetent Cervical OS
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Continue observation for contractions, rupture of membranes & monitor fetal heart tones Position patient to minimize pressure on cervix
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Ectopic Pregnancy
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Any gestation outside the uterine cavity Most frequently in the fallopian tubes, where the tissue is incapable of the growth needed to accommodate pregnancy, so rupture of the site usually occurs before 12 weeks Any condition that diminishes the tubal lumen may predispose a woman to ectopic pregnancy
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Classic Signs of Ectopic Pregnancy
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Missed menstrual period Positive pregnancy test Abdominal *pain* Vaginal spotting
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Other Assessment Findings of Ectopic Pregnancy
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History of missed periods & symptoms of early pregnancy Abdominal *pain*, may be localized to one side Rigid, tender abdomen, sometimes abnormal pelvic mass Bleeding, if severe may lead to shock Low hemoglobin & hematocrit, rising WBC HCG titers usually lower than in intrauterine pregnancy
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Nursing Interventions & Medical Management of Ectopic Pregnancy
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Giving Methotrexate to dissolve the tubal pregnancy if possible, least invasive Surgical intervention may be required
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Methotrexate
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Folic acid antagonist that interferes with the proliferation of trophoblastic cells
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Nursing Interventions & Surgical Management of Ectopic Pregnancy
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Prepare patient for surgery Institute measures to control/treat shock if hemorrhage is severe, continue to monitor postoperatively Allow patient to express feelings about loss of pregnancy & concerns about future pregnancies
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Gestational Trophoblastic Disease (Hydatidiform Mole or Molar Pregnancy)
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Proliferation of trophoblasts, embryo dies Unusual chromosomal patterns seen (either no genetic material in ovum or 69 chromosomes) The chronic villi change into a mass of clear, fluid-filled grapelike vessels Complete or partial Cause is essentially unknown Seems like a pregnancy but no fetus occurs Can set the patient up for cancer later in life
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Gestational Trophoblastic Disease (Hydatidiform Mole or Molar Pregnancy) Assessment Findings
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Absence of fetal heart sounds in the presence of other signs of pregnancy is a classic sign A uterus that is larger than expected for gestational age High levels of HCG with excessive nausea & vomiting (also seen in twins) Dark red to brownish vaginal bleeding (common signs) after 12th weeks (resembles prune juice) Anemia often accompanies bleeding Symptoms of preeclampsia before usual time of onset No palpation of normal fetal parts Ultrasound shows no fetal skeleton
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Gestational Trophoblastic Disease (Hydatidiform Mole or Molar Pregnancy) Nursing Interventions
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Provide pre-and postoperative care for evacuation of uterus *Teach contraceptive use so that pregnancy is delayed for at least one year due to increase risk of cancer* Teach patient need for follow up lab work to detect rising HCG levels indicative of choriocarcinoma Teach about risk of future pregnancies, if indicated
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Second Trimester Pregnancy Complications
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There are few unique causes of bleeding in the second trimester
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Third Trimester Pregnancy Complications
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Placenta previa Abruptio placenta Preterm labor Premature rupture of membranes Rupture of membranes Oligohydramnios Hydramnios (polyhydramnios)
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Preterm Labor
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Labor that occurs before the end of the 37th week of pregnancy Cause is frequently unknown
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Preterm Labor Prevention (Causes)
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Minimize or stop smoking (a major factor of preterm labor & birth) Minimize or stop substance abuse/chemical dependency Early & consistent prenatal care Appropriate weight gain Minimize psychological stressors Learn to recognize signs & symptoms of preterm labor
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Medical Management of Preterm Labor
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Unless labor is irreversible, or a condition exists which the mother or fetus would be jeopardized by the continuing of the pregnancy, or the membranes have ruptured, the usual medical intervention is to attempt to arrest the premature labor
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Tocolysis
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Use of medication in an attempt to stop labor May delay birth for 24-48 hours Important to administer betamethasone or transfer mother to a tertiary care facility
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Medication Treatments for the Medical Management of Preterm Labor
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Magnesium sulfate Nifedipine Indomethacin Beta-Adrenergic Drugs Betamethasone
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Magnesium Sulfate
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Stops uterine contractions with fewer side effects than beta-adrenergic drugs & interferes with muscle contractility Magnesium sulfate acts upon the myoneural junction, diminishing neuromuscular transmission It promotes maternal vasodilation, better tissue perfusion & have anticonvulsant effect
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Magnesium Sulfate Risks
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Magnesium toxicity PPH
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Magnesium Sulfate Administration
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Must be 1:1 with the nurse Give magnesium sulfate on an IV pump as secondary with fluids running as primary
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Magnesium Sulfate Nursing Responsibilities
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Monitor patients respirations, blood pressure & reflexes as well as I&O, flushing & other signs and symptoms of magnesium sulfate toxicity
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Magnesium Sulfate Side Effects
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Few serious side effects Initally patient feels hot, flushing, may cause headache, nausea, diarrhea, dizziness & lethargy *hypotonia & flushing* are the most common side effects
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Magnesium Toxicity
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Monitor respiratory rate *(<12 breaths/minute)* & hourly urine output *(<30 mL/hr)*
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Magnesium Sulfate Antidote
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Calcium gluconate
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nifedipine
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(Adalat/Procardia)
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nifedipine Drug Class
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Calcium Channel Blocker
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nifedipine Action
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Relax smooth muscles including the uterus by blocking calcium entry
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nifedipine Side Effects
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Hypotension Flushing
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indomethacin
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(Indocin)
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indomethacin Drug Class
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Prostaglandin synthetase inhibitor
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indomethacin Action
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Relaxes smooth muscle by inhibiting prostaglandins
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indomethacin Side Effects
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Nausea Vomiting Dyspepsia
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Beta-Adrenergic Drugs
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ritodrine (Yutopar) terbutaline (Brethine)
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ritodrine (Yutopar)
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Only beta-adrenergic approved by the FDA for tocolysis but it is infrequently used because of side effects & minimal increase in length of pregnancy
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terbutaline (Brethine)
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Not approved by the FDA for tocolysis but is more widely used
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Betamethasone
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Given when premature labor cannot or should not be arrested & fetal lung maturity needs to be improved Stimulates fetal lung maturation Used to cause fetal surfactant induction which helps to open the babies airways upon delivery
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Betamethasone Administration
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IM to the mother usually every 12 hours times 2 then weekly until 34 weeks gestation
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Nursing Interventions for Preterm Labor
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Keep patient at rest, side-lying position Hydrate the patient & maintain weight IV or PO fluids Maintain continuous maternal/fetal monitoring (maternal/fetal vital signs every 10 minutes) be alert for abrupt changes Monitor maternal I&O Monitor urine for glucose (diabetes) & ketones (dehydration) Watch cardiac & respiratory status carefully Evaluate lab test results carefully
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Oligohydramnios
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Decrease amount of amniotic fluid between 32 to 36 weeks May be associated with placental insufficiency or fetal urinary tract abnormalities
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Oligohydramnios Risks
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Perinatal morbidity & mortality
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Oligohydramnios Interventions
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Amniofusion
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Oligohydramnios Assessments
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Fetal well being
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Potter's Syndrome
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Lack of urine into amniotic cavity Bilateral renal agenesis (BRA) or complete absence of the kidneys Fetus does not survive long after birth
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Hydramnios (Polyhydramnios)
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Too much amniotic fluid between 32 to 36 weeks Associated with fetal anomalies Increased incidence of preterm births, fetal malpresentation & cord prolapse Could indicate large baby
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Some Possibilities for Hydramnios
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Prenatal Bartter's Syndrome Poorly-controlled maternal diabetes Twin or multiple gestations Fetal abnormalities which make it difficult for the baby to swallow & process the fluid normally Rh blood incompatibility which can bring on fetal anemia & other factors
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Neonatal Bartter Syndrome
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The condition is caused by a defect in the kidney's ability to reabsorb sodium In most cases, Neonatal Bartter syndrome is seen between 24 & 30 weeks of gestation with excess amniotic fluid
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Characteristics of Neonatal Bartter Syndrome
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Hypokalemia Metabolic alkalosis Increased urinary excretion of sodium, potassium & chloride Normal blood pressure (?)
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Rupture of Membranes
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Loss of amniotic fluid, prior to term, unconnected with labor
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Dangers of Rupture of Membranes
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Prolapsed cord Infection Potential need for premature delivery
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PPROM
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Premature prolonged rupture of membranes Less than 37 weeks, longer than 18 hours
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Nursing Assessment for Rupture of Membranes
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Report from mother/family of discharge fluid Sterile speculum Nitrozene paper Ferning Pooling Monitor maternal/fetal vital signs on continuous basis, especially for maternal temperature (hourly) Calculate gestational age Observe for signs of infection & signs of onset of labor (may induce if there are signs of an infection) Observe & record color, odor, amount of fluid Delay vaginal exam unless fetal distress (prolapsed cord) Provide explanations of procedures & findings Support mother & family Prepare mother/family for early birth if indicated
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Nitrozene Paper
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Change of color to dark blue if positive
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Ferning
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Wiping fluid on slide to see if "ferns" appear when dried
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Possible Cause/Result of Rupture of Membranes
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Chorioamnionitis
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Placenta Previa
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Low implantation of the placenta so that it overlays some or all of the internal cervical OS Amount of cervical OS involved classifies placental previa as marginal, partial or complete
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Placenta Previa Etiology
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Cause is uncertain but uterine factors may be involved (scaring)
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Placenta Previa Assessment Findings
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*Bright red* vaginal bleeding after 7th month is cardinal indicator Bleeding may be intermitten, in gushes or continuous Uterus remains soft Fetal heart rate usually stable unless maternal shock present No vaginal exam by nurse Diagnosed by sonography
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Placenta Previa Nursing Intervention
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Ensure complete bed rest Maintain sterile conditions for any invasive procedures Make provisions for emergency C-section Continue to monitor maternal/fetal vital signs Measure blood loss carefully Assess uterine tone regularly
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Abruptio Placenta
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Separation of placenta from part or all of the normal implantation site, usually accompanied by *pain* Usually occurs after 20th week of pregnancy Seen frequently in women with hypertension, previous abruptio placentae, late pregnancies & multigravidas but cause essentially unknown
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Abruptio Placenta Assessment Findings
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*Painful* vaginal bleeding *Tender board like uterus* Fetal bradycardia & late decelerations, absent fetal heart tones in complete abruption Additional signs of shock
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Abruptio Placenta Nursing Interventions
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Ensure bed rest Check maternal/fetal vital signs frequently Prepare for IV infusions of fluids/blood as indicated Monitor urinary output Anticipate coagulation problems Provide support to parents as outlook for fetus can be poor Prepare for emergency surgery as indicated
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Other Complications with Pregnancy
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Hyperemesis gravidarum Preeclampsia Diabetes Placental Abnormalities Rh sensitization
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Hyperemesis Gravidarum
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Excess nausea & vomiting of early pregnancy leads to dehydration & electrolyte disturbances
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Hyperemesis Gravidarum Causes
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Possible severe reaction to HCG Not psychological Greater risk in conditions where HCG levels are increased HCG levels peak around 6 weeks after conception, plateau then begin to decline after 12th week Symptoms often improve later in pregnancy but may last entire time
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Hyperemesis Gravidarum Assessment Findings
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Nausea & vomiting, progressing to retching between meals Weight loss
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Hyperemesis Gravidarum Nursing Interventions
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Begin NPO & IV fluid & electrolyte replacement IV anti-nausea medication Monitor I&O Gradually re-introduce PO intake, monitor amounts taken & retained Monitor TPN & central line placement if unable to eat Provide mouth care Offer emotional support Refer to home health as appropriate for continued IV or TPN therapy
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Preeclampsia
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Refers to a condition unique to pregnancy where hypertension is accompanied by proteinuria & edema Maternal or fetal condition may be compromised
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Preeclampsia Onset
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After 12th week of pregnancy, may appear in labor up to 48 hours post partum
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Preeclampsia Characterization
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Wide spread vasospasm
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Preeclampsia Probable Cause
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Essentially unknown High in primigravidas, multiple pregnancies, hydatidiform mole, poor nutrition, essential hypertension, familial tendency Gradual loss of normal pregnancy related resistance to angiotensin 2 May also be related to decrease production of some vasodilating prostaglandins
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Usual Clinical Classification of Hypertensive Disorders in Pregnancy
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Preeclampsia Preeclampsia-Eclampsia Chronic hypertension Preeclampsia superimposed on chronic hypertension Gestational hypertension (does not become preeclampsia)
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Mild Preeclampsia Assessments
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Appearance of symptoms after 20th week of pregnancy Blood pressure >140 but 90 but 0.3g but <2g in 24 hour specimen (1+ or higher on dipstick) (?) All other assessments normal
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Nursing Interventions for Mild Preeclampsia
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Promote bed rest as long as signs of edema or proteinuria are minimal, preferably side-lying Provide well balanced diet with adequate protein & roughage, no sodium restrictions Explain need for close follow up weekly or twice weekly visits to physician
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Severe Preeclampsia Assessments
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Blood pressure of >160/110 or higher on 2 occasions at least 6 hours apart while on bedrest Proteinuria >5g/24 hours (3+ on dipstick) Pulmonary edema/heart failure Cyanosis may be present Headaches, nausea & vomiting, visual disturbances & irritability Epigastric or right quadrant pain Growth restriction, reduced amniotic fluid & fluid volume
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Severe Preeclampsia Nursing Interventions
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Minimize all stimuli/restrict visitors Check vital signs & lab values frequently Have airway, suction & oxygen equipment available Assess deep tendon reflexes & clonus Administer medication as ordered Continue observation for 24 to 48 hours postpartum Most likely start of magnesium sulfate up to 24 hours after delivery Promote best res, side-lying Carefully monitor fetal vital signs Monitor I&O Take daily weights Initiate seizure precautions Monitor for magnesium sulfate toxicity Administer sedatives as ordered
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Deep Tendon Reflexes
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Grade reflexes 4+ 3+ 2+ 1+ 0
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4+
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Hyperactivity, very brisk, jerky or clonic response, abnormal
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3+
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Brisker than average, may not be abnormal
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2+
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Average response, minimal
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1+
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Diminished response; low normal
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Clonus
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With a normal response, the foot returns to its normal position of plantar flexion Clonus is present if the foot "jerks" or "taps" against the examiner's hand Record the number of taps or beats of clonus, if none than indicate: no clonus
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HELLP Syndrome
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Laboratory diagnosis for a variation of severe preeclampsia, not a separate illness Possible life threatening complication *H*emolysis *E*levated *l*iver *e*nzymes *L*owered *p*latelets
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HELLP Syndrome Cure
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Delivery is the only known cure
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HELLP Syndrome Presentation
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Non specific
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Eclampsia Assessment Findings
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Increased hypertension precedes convulsion followed by hypotension & collapse Coma may ensue Labor may begin, putting fetus in great jeopardy Convulsions may recur
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SEIZURE
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Safety (bed railing pads) Establish/maintain airway IV bolus Zealous observation Uterine activity Rapid resuscitation Evaluate fetus
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Chronic Hypertension
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Associated with increased incidence of abruptio placenta & increased perinatal mortality Ideally management begins before pregnancy or when women is hypertensive before 20 weeks No signs preeclampsia (?)
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Chronic Hypertension Treatment
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aldomet (Methyldopa)
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aldomet (Methyldopa)
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Most often recommended for chronic hypertension after lifestyle changes
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Chronic Hypertension Treatment Goal
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Maintain a diastolic below 90 mmHg
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Chronic Hypertension with Superimposed Preeclampsia
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In women with hypertension before 20 weeks & new-onset proteinuria In women with both hypertension & proteinuria before 20 weeks & significant increase in hypertension plus one of the following: new onset of symptoms, thrombocytopenia, elevated liver enzymes Increased morbidity for both the mother & fetus
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Gestational Hypertension
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Transient elevation of blood pressure occurs for the first time after mid pregnancy without proteinuria or other signs of preeclampsia If preeclampsia does not develop & blood pressure returns to normal by 12 weeks post partum If blood pressure elevation persists after 12 weeks post partum, the women is diagnosed with chronic hypertension
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Diabetes Mellitus
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Gestational diabetes mellitus is any degree of glucose intolerance with its onset or first recognition during pregnancy
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Gestational Diabetes Mellitus Onset & Reversal
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Onset during pregnancy & reversal after termination of pregnancy
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Gestational Diabetes Mellitus Risks
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Increased risk of adult onset diabetes later in life
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Significance of Diabetes in Pregnancy
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Interaction of estrogen, progesterone & cortisol raise maternal resistance to insulin If the pancreas cannot respond by producing additional insulin, excess glucose moves across the placenta to fetus where fetal insulin metabolizes it & acts as growth hormone, promoting macrosomia Maternal insulin needs to be carefully monitored during pregnancy to avoid widely fluctuating levels of blood glucose Dose may drop during first trimester then rise during second & third trimesters Higher incidence of fetal anomalies & maternal hypoglycemia
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Assessment Findings for Gestational Diabetes Mellitus
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Polyuria Polydipsia Weight loss Polyphagia Elevated glucose levels in blood & urine 1 hour glucose tolerance test at 24 to 28 weeks 3 hours glucose tolerance test used if results from 1 hours GTT>180 mg/dl
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Gestational Diabetes Mellitus Nursing Interventions
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Teach patient the effects & interactions of diabetes & pregnancy & signs of hyper & hypoglycemia Teach patient how to control diabetes in pregnancy Monitor fetal status throughout pregnancy & usually will be referred to endocrinologist Assess status of mother & baby frequently Monitor carefully fluids, calories, glucose & insulin during labor & delivery May start on insulin drip during labor & delivery Continue careful observation in post delivery period High risk for hypoglycemia for newborn
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Infections (TORCH)
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Toxoplasmosis Other infections Rubella Cytomegalovirus Herpes Devastating to the fetus causing abortions, malformations & even fetal death
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Infections Nursing Interventions
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Instruct the pregnant woman in signs & symptoms that indicate infections Caution women to avoid obviously infected persons & other sources of infections May affect delivery options
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Placental Abnormalities
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*Developmental problems of the placenta:* Placental lesions Succenturiate placenta Circumvallate placenta Battledore placenta *Degenerative changes:* Infarcts Placental calcifications
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Succenturiate Placenta
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One or more accessory lobes of fetal villi will develop on the placenta
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Succenturiate Placenta Maternal Complications
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Post partum hemorrhage if placenta is retained
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Succenturiate Placenta Fetal Complications
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No fetal-neonate complications as long as all parts of the placenta remain attached until after birth of the fetus
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Circumvallate Placenta
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A double fold of chorion & amnion form a ring around the umbilical cord, on the fetal side of the placenta
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Circumvallate Placenta Maternal Complications
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Increased incidence of late abortion, antepartum hemorrhage & preterm labor
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Circumvallate Placenta Fetal Complications
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Intrauterine growth restriction Prematurity and/or fetal death
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Battledore Placenta
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The umbilical cord is inserted at or near the placental margin
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Battledore Placenta Maternal Complications
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Increased incidence of preterm labor & bleeding complications
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Battledore Placenta Fetal Complication
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Prematurity & fetal stress
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Velamentous Insertion of the Umbilical Cord
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The vessels of the umbilical cord divide some distance from the placenta in the placental membranes
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Velamentous Insertion of the Umbilical Cord Maternal Complications
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Hemorrhage if one vessel is torn
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Velamentous Insertion of the Umbilical Cord Fetal Complications
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Fetal stress Hemorrhage
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Placental Accreta
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Abnormal adherence Placenta grows into the uterine wall The incidence of placenta accrete also significantly increases in women with previous C-section compared to those without prior surgical delivery
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Rh Sensitization
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Rh-negative women who become pregnant with an Rh positive fetus may become sensitized to the Rh antigen if there is any accidental contact between maternal & fetal blood Can occur also during amniocentesis of other invasive procedure
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Complications of Rh Sensitization
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Sensitized Rh negative women develop anti-Rh antibodies which may cross the placenta in subsequent Rh-positive pregnancies & attack & destroy the fetal RBC's
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Newborn Effects of Rh Incompatibility & Sensitivity
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Progressively severe Erythroblastosis fetalis Hydrops fetalis
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Erythroblastosisi Fetaslis
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The antibodies from the mom cross the placenta & attach to fetal red blood cells & destroy them
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Hydrops Fetalis
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Severe anemia that results in heart failure
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Rh Sensitization Assessment
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All pregnant women should be tested for blood group, Rh factor & antibody screening, a history of previous miscarriage, blood transfusions or infants experiencing jaundice should be noted
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Rh Sensitization Intervention
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Unsensitized Rh-negative patients should be given 300mg of Rh immune globulin (RhoGAM) IM at *28 weeks & within 72 hours of delivery* RhoGAM is not given to mothers who are already sensitized & have antibodies Rh immune globulin is also given after abortion, ectopic pregnancy, amniocentesis & any other situation that might result in maternal exposure to the fetal Rh antigen Kleihauer-Betke test
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Kleihauer-Betke Test
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Test to see if there has been any maternal & fetal blood mixing
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Fetal Demise
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First trimester fetal loss Fetal anomaly Neonatal death Stillbirth or intrauterine fetal demise
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First Trimester Fetal Loss
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Ectopic pregnancies Elective termination
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Fetal Anomaly
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The loss of their "perfect child" Intensity of grief may be affected by the type & severity of the anomaly Anticipatory grieving
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Neonatal Death
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Death within the first month of life Typically related to congenital defect, sepsis, prematurity or SIDS
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Stillbirth/Intrauterine Fetal Demise
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Fetal demise in utero after 20 weeks gestation
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Delivery of Stillbirth/Intrauterine Fetal Demise
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Most mothers spontaneously begin labor within 2 weeks after IUFD, if labor does not ensue, they need to go to the labor & delivery unit
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First Symptom of Fetal Death
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Absent movement (?)
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Nursing Care during Perinatal Loss
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Planning & implementation with grieving parents Follow up after discharge
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Induction of Labor
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Prostaglandins
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Prostaglandins
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Vaginal suppositories 12 to 24 weeks dinoprostone (Prostin E2/Cervidil) misoprostol (Cytotec)
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dinoprostone (Prostin E2)
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Suppositories for 14 to 28 weeks
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misoprostol (Cytotec)
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Intravaginal tablets for 2nd & 3rd trimester fetal demises & for termination of 2nd & 3rd trimester pregnancies
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Nursing Care for Fetal Demises
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Communicating & caring techniques Options for parents
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Communicating & Caring Techniques
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Actualize the loss Provide time to grieve Allow for individual differences, cultural, spiritual & physical needs of the parents
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Options for Parents
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Seeing & holding Bathing & dressing Privacy Visitation with other family members Religious rituals/funeral arrangements Special memories Memory box & pictures
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