Medsurg Exam 3: Chapter 10 Altered Fluid, electrolyte, and Acid-Base Balance – Flashcards

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Homeostasis
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-Ability to maintain internal equilibrium by adjusting physiologic processes -Reestablishment and maintenance a goal in managing fluid, electrolyte, and acid-base imbalance
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Water
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-transport of nutrients -medium for metabolic reactions -regulation of body temperature
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Electrolytes
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-water balance regulation -acid-base balance -enzyme reaction -neuromuscular activity
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Intracellular fluid (ICF)
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-found within cells -essential for normal cell division --40% of total body weight
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Extracellular fluid (ECF)
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-located outside of cells -interstitial fluid -intravascular fluid -transcellular fluid -20% of total body weight
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(ICF) contains solutes
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-Potassium -Magnesium -Phosphate -glucose -oxygen
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(ECF) contains solutes
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-sodium -chloride -bicarbonate
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Osmosis
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-concentration of a solution -osmotic pressure and tonicity
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Diffusion
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-simple -facilitated(carrier-mediated)
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filtration
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-balance of hydrostatic osmotic pressure
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active transport
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-sodium-potassium pump
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Thirst
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-primary regulator of water intake
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Kidneys
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regulates volume, osmolality of body fluids
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Renin-angiotensin-aldosterone system
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helps maintain intravascular fluid balance and blood pressure
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Antidiuretic hormone
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regulates water excretion from kidneys
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Atrial natriuretic peptide
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released by atrial muscle cells in response to distention from fluid overload
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The Patient with a Fluid Volume Deficit: Causes
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-vomiting and diarrhea -gastrointestinal suctioning, intestinal fistulas, and intestinal drainage -diuretics, renal disorders, and endocrine disorders -hot environment -hemorrhage
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The Patient with a fluid volume deficit: Patho
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-hypovolemia from loss of ECF -Third Spacing shift of fluid out of vascular space into unusable space triggered by stress hormones difficult assessment shock, multiorgan failure
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The Patient with a Fluid volume deficit: Manifestations
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-rapid weight loss and pale skin -decrease in skin turgor and urine output -Tachycardia -decreased systolic blood pressure and venous pressure
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The Patient with fluid volume deficit: Fluid management
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-oral rehydration(safest) -IV therapy
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the patient with fluid volume deficit: Interprofessional care
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-Diagnosis serum electrolytes serum osmolality hemoglobin and hematocrit urine specific gravity and osmolality hemodynamic pressures
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fluid volume deficit: health promotion
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-teaching to prevent fluid volume deficits -carefully monitor intake and output
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fluid volume deficit: Assessment
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-health history -physical assessment
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fluid volume deficit: Priorities of care
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-restoration of adequate fluid volume
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fluid volume deficit: diagnoses, outcomes and interventions, continuity of care
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-Deficient fluid volume -Ineffective Tissue Perfusion -Risk for Injury -assess patients understanding of the cause of the deficit
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fluid volume excess: manifestations and complications
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-Extracellular hypovolemia circulatory overload -Interstitial peripheral edema generalized edema -Complications CHF,pulmonary edema
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fluid volume excess: patho
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-heart or renal failure -cirrhosis of the liver -adrenal gland disorders -corticosteroid administration -stress conditions causing release of ADH and aldosterone -excessive sodium intake -medication side effects
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fluid volume excess: interprofessional care
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-Diagnosis serum eletrolytes, osmolality serum hematocrit, hemoglobin often decreased renal, liver function
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fluid volume excess: medications
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-Diuretics loop (furosemide) Thiazide-type (hydrochlorothiazide) Potassium-sparing (amiloride, spironolactone)
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fluid volume excess: treatments
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-fluid management -dietary management
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fluid volume excess: health promotion
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-relationship between sodium intake and water retention
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fluid volume excess: assessment
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-health history -physical assessment weight, vital signs, circulatory signs, lung sounds, dyspnea, cough urine output, mental status
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fluid volume excess: Priorities of care
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-supporting cardiovascular, respiratory function
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fluid volume excess: diagnosis, outcomes, interventions
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-fluid volume excess -risk for impaired skin integrity -impaired gas exchange
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fluid volume excess: continuity of care
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-manage cause of fluid volume excess -prevent future episodes of excess volume
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Sodium Balance: Normal levels,affects,organ is primary regulator and 3hormones
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-135-145mEq/L -affects distribution of fluids -kidney is primary regulator renin-angiotensin-aldosterone system antidiuretic hormone (ADH) atrial natriuretic peptide (ANP)
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Patient with hyponatremia: patho
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-excessive loss via kidneys, gi tract, skin, cerebral wasting, third spacing -water gains from systemic diseases -decrease in serum osmolality
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Patient with hyponatremia: manifestations
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-depend on rapidity, severity, and cause of imbalance
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Patient with hyponatremia: Diagnosis
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-serum sodium decreased -serum osmolality decreased -24hour urine specimen 40-220 mEq/L
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Patient with hyponatremia: medications
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-sodium containing fluids
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Patient with hyponatremia: Fluid and dietary mangement
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-high in sodium -fluid restriction
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Patient with hyponatremia: health promotion
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-teach manifestations -important to drink liquids with sodium, electrolytes when perspiring, in high temperature
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Patient with hyponatremia: Assessment
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-health history -physical assessment
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Patient with hyponatremia: Priorities of care
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-restoring sodium, water balance -preventing cerebral edema
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Patient with hyponatremia: Diagnoses, outcomes, and interventions
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-risk for imbalanced fluid volume -risk for ineffective cerebral tissue perfusion
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Patient with hyponatremia: continuity of care
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-figure out what is the underlying cause of sodium deficit -prevention -manifestations
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patient with hypernatremia: patho
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-excessive water loss -excessive sodium intake -increased osmolality
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patient with hypernatremia: manifestations
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-thirst, altered neurologic function -seizures,coma, and death
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patient with hypernatremia: Diagnosis
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-serum sodium elevated -serum osmolaltiy elevated
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patient with hypernatremia: medications
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-oral,enteral,iv fluid replacement
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patient with hypernatremia: health promotion
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-prevention -adequate water for patients receiving tube feedings
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patient with hypernatremia: Priorities of care
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-Mental status and brain function
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patient with hypernatremia: diagnoses, outcomes, and interventions
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-Risk for injury
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patient with hypernatremia: Continuity of care
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-respond to thirst and consume adequate fluids -low sodium diet -schedule for monitoring of serum electrolyte levels
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Hyponatremia: manifestations
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-plasma sodium <135 -decrease in serum osmolality -muscle cramps -headache -anxiety -nausea, anorexia,vomiting,hypotentions,shock
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Hypernatremia: manifestations
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-plasma sodium >145 -increase in serum osmolality -increase in thirst, oliguria, urine specific gravity -dry skin and mucous membranes and decreased skin turgor, dry mouth, headache, restlessness, seizures, coma, tachy, hypotention, vascular collapse
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Potassium balance
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3.5-5.3 mEq/L
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Potassium: functions
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-nerve impulses -cardiac rhythm -muscle contractions
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Potassium: regulator structures
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-sodium-potassium pump -kidneys and aldosterone
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patient w/hypokalemia: patho
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-inadequate potassium intake -excessive skin,renal or intestinal losses -redistribution between the ICF and ECF
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patient w/hypokalemia: manifestations
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-nausea and vomiting -muscle weakness and cramps -polyuria -decreased cardiac output
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patient w/hypokalemia: Diagnosis (what to tests to use)
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-serum potassium and electrolytes -arterial blood gases -renal function tests -ecf reading
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patient w/hypokalemia: meds
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oral and/or parenteral supplements
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patient w/hypokalemia: nutrition
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diet high in potassium rich foods
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patient w/hypokalemia: health promo
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balanced electrolyte solutions
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patient w/hypokalemia: assessment
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health history physical assesment
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patient w/hypokalemia: priorities of care
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cardiac impulse transmission cardiac and skeletal muscle function
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patient w/hypokalemia: Diagnosis, outcomes
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decreased cardia output activity intolerance risk for imbalance fluid volume
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patient w/hypokalemia: continuity of care
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recommended diet meds, supplements use of salt supplements
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patient w/hyperkalemia: pathophysiology
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inadequate renal excretion of potassium rapid iv administration acidosis, tissue trauma, chemotherapy, starvation
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patient w/hypokalemia: manifestations
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neuromuscular -muscle weakness -tremors gi discomfort cardia -ecg changes -dysrhythmias -cardiac arrest
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patient w/hypokalemia: diagnosis
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serum electrolytes, abgs, and ecg
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patient w/hypokalemia:
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calcium gluconate calcium chloride insulin and glucose hypertonic dextrose sodium bicarbonate sodium polystyrene sulfonate
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patient w/hypokalemia: Dialysis
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peritoneal or hemodialysis
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patient w/hypokalemia: health promo
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reading food and dietary supplement labels
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patient w/hypokalemia: priorities of care
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electrical conduction and contractility of the heart
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patient w/hypokalemia: outcomes
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risk for decreased cardiac output risk for activity intolerance
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patient w/hypokalemia: continuity of care
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diet restrictions manifestations otc medications to avoid
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Calcium balance: serum level
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9-11 mg/dl
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Calcium balance: functions
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stabilizing cell membranes muscle contraction and relaxation cardiac function and blood clotting
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Calcium balance: regulation
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parathyroid hormone, calicitriol, calcitonin, acid-base balance, plasma protein levels
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Hypocalcemia: risk factors
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Parathyroidectomy older women lactose intolerance alcoholism
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Hypocalcemia: patho
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pancreatitis hypoparathyroidism electrolyte imbalances alkalosis malabsorption disorders inadequate vitamin D medication
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Hypocalcemia: manifestations
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tetany convulsions
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Hypocalcemia: Diagnosis tests
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serum calcium total albumin magnesium phosphate parathyroid hormone ecg
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Hypocalcemia: health promotion
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deficit associated with aging female gender
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Hypocalcemia: nursing diagnosis
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risk for injury
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Hypocalcemia: continuity of care
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educate on risk factors, meds, dietary recommendations, manifestations
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hypercalcemia: patho
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hyperparathyroidism and malignancies prolonged immobility excessive intake of vitamin d
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hypercalcemia: manifestations
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subtle and mild at first decrease in neuromuscular excitability cns symtoms cardia effects
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hypercalcemia: Diagnosis
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serum pth levels ecg test bone density test
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hypercalcemia: meds
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loop diuretics bisphosphonates sodium phosphate gulcocorticoids
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hypercalcemia: Fluid management
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isotonic saline
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hypercalcemia: helath promotion
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promote mobility fluid intake
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hypercalcemia: priorities of care
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safety
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hypercalcemia: outcomes
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risk for injury risk for fluid volume excess
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hypercalcemia: continuity of care
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limiting intake meds increase fluid intake weight bearing exercise
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Hyocalcemia: manifestations
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serum <9 numbness and tingling muscle cramping hyperactive reflexes tetany carpopedal adn larygeal spasms positive chvostek and trousseau hypotention ventricular dysrhythmias bone pain and fractures
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hypercalcemia: manifestations
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serum >11 increased thrist anorexia nausea and vomiting constipation muscle weakness increased bp av block lethargy coma
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Magnesium balance: normal serum
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1.8-3.0
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Magnesium: functions
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intracellular processes neuromuscular transmission cardiovascular function
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hypomagnesmia: risk factors
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alcoholism protein malnutrition diabetic ketoacidosis kidney disease meds
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hypomagnesmia patho
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relates to hypokalemia, hypocalcemia
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hypomagnesmia: manifestations
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tremors hyperrecative reflexes positive chvostek, trousseau signs tetany paresthesias seizures
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hypomagnesmia: assessment
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manifestations of hypokalemia,hypocalcemia gI function
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hypomagnesmia: Diangonis
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monitor serum mag, potassium, calcium cardiac status dioxin toxicity deep tendon reflexes
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hypomagnesmia: continuity of care
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discuss alcoholism treatment options educate on mag sources
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hypermagnesemia: patho
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renal failure nausea and vomiting interfered neuromuscular transmission depressed CNS
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hypomagnesmia: patho and manifestations
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hypotention flushing, sweating bradydysrhythmias respiratory depression
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hypo mag nes emia: interprofessional care
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hemodialysis or peritoneal dialysis calcium gluconate mechanical ventilation
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hypomagnesemia: manifestations
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canges in personality nystagmus positve babinski and chvostek hypertension tachy cardiac dysrhythmias
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hypermagnesemia: manifestations
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confusion and lethargy hypotension cardic dysrthythmias coma cardiac arrest
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Phosphate balanced level
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2.5-4.5
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phosphate functions:
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production of atp red blood cell, nervous system, muscle function metabolism acid-base balance`
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hypophosphatemia: manifestations
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decrease gi absorption increased renal excretion refeeding syndrome meds
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hypophosphatemia: patho
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alcoholism hyperventilation respiratory alkalosis diabetic ketoacidosis
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hyperphospatemia: manisfestations
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paresthesias muscle weakness nausea and vomiting dysphagia tetany decreased bp cardiac dysrhythmias
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hypophosphatemai
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intention tremor confusion bone pain joint stiffness bleeding impaired wbc seizures
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nonvolatile acids:
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lactic acid hcl acid sulfuric acid
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First line of defense:
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buffer systems bicarbonate phosphate protein
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Second line of defense: respiratory
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hyperventilation hypoventilation (hours) to days
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Renal system: third line of defense
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days to weeks kidneys
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ph
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7.35-7.45
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paco2
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35-45
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pao2
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80-100
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serum bicarbonate
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22-26
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base excess
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-3.0to3.0
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HCo2
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22-26
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metabolic acidosis: patho
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accumulation of metabolic acids excess loss of bicarbonate increase in chloride levels anion gap
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metabolic acidosi: diagnosis
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abgs serum electrolytes, ecg, blood glucose, renal function
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metabolic acidosis: meds
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sodium bicarbonate iv insulin and fluid for dka saline solutions and glucose etoh
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Metabolic alkalosis: meds
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potassium chloride sodium chloride acidifying solution for severe cases
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acidosis: meds
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bronchodilators antibiotics
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