ECG Graph paper, ECG waveforms,Cardiac Drugs
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There are ____ standard ECG leads
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12
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3 of the 12 leads are
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standard limb leads, I,II,III
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Where are standard limb leads placed?
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I right arm, III left arm, II left leg, there an electrode placed on the right leg to act as a ground.
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The placement of the standard limb leads forms what triangle?
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Einthoven Triangle
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The standard limb leads, I, II, III are called ___ ____ because they conduct between to designated electrodes
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bipolar leads
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Standard Lead I records electrical activity from the ___ ___ to the ____ ____
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right arm- to the left arm+
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Standard Lead II records electrical activity from the ___ ___ to the ___ ____
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Right arm - to the left leg+
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The Left Arm,Standard lead can be either ____ charged or ____ charged depending on the direction of the picture being taken
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Left are lead can be either + or _
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Standard Lead III records electrical activity from the ___ ____ to the ____ ____
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Left arm - to the Left Leg +
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Augmented leads are the 3 leads known as
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aVR, aVL and aVF
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aVR stands for
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augmented voltage right arm
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aVL stands for
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augmented voltage left arm
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aVF stands for
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augmented voltage foot
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Augmented limb leads are ____ because each lead uses only 1 electrode to record electrical activity generated from the heart
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unipolar
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aVR records activity from the Rarm and the electrical current generated from the heart flows ____ the positive right arm
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away
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aVL records activity form the left arm and the electrical current generated from the heart flows ___ the positive left arm
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toward
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ECG Graph paper moves at a standard speed of
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25 mm/sec
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Each small square on the ECG paper is equal to ____ seconds and is ___ in height and ___ wide
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0.04 seconds and is 0.1 mv in height, and is 1 mm high and 1mm wide
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there are ______ small squares /min
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1500 small squares per minute
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1500 small squares per minute is equal to
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.04 x 1500 = 60 seconds
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Every 5th line there is a bold line both horizontally and vertically forming a large square with is equal to how many seconds?
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0.2 seconds
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Each large square is 0.5 mv in height and is 5mm high by 5mmwide, there are howmany large squares per minute?
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300 large squares /min
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there are 300 large squares/min on ECG paper, this is figured by
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0.2 seconds x 300 large squares= 60 seconds
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There are vertical hash marks at the top of the paper to represent 1,2, or 3 seconds, but most commonly
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3 seconds
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if the hash marks are 3 seconds apart, there are how many large squares in that 3 second interval?
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0.2 seconds x 15 large squares = 3 second interval
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If the vertical hash marks are 3 seconds apart how many small squares are in each?
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3seconds x 0.04 seconds per small square = 75 small squares per each 3 second interval
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ECG paper - Voltage (Gain) is defined how?
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ECG recordings are standardized with a special signal inscribed into the recording to give an accurate voltage measurement.
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The most common voltage (Gain) on ECG paper is
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Standard I which is 1 mv = 10 mm high
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Standard II voltage ( Gain ) is used when
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the QRS complexes are small
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StandarII voltage(Gain) is equal to
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1 mv= 20 mm high
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Standard 1/2 Voltage (Gain) is used when the patient has?
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extreme ventricular hypertrophy
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Standard 1/2 Voltage(Gain) is measured
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1mv=5mm high
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To Calculate Rate most accurately, you should
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count the number of small squares between two consecutive R waves and divide that number into 1500
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you can count the large squares between two consecutive R waves to calculate rate and
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then divide into 300
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The easiest way to calculate the rate on an ECG strip is to
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count the number of QRS complexes in a 6 second strip and multiply by 10
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Normal Sinus Rhythm has a normal atrial and ventricular rate which is
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60-100
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The pace maker site for sinus tachycardia is
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the SA node
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in Sinus Tachycardia, the rhythm is normal but the
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rate is greater than 100 beats per minute
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What causes Sinus Tachycardia?
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Pain, Anxiety, Fever, Hypoxia, Shock,Medications, and stimulants such as caffeine and tobacco
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How do we treat Sinus Tachycardia?
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We treat the underlying cause, if it is symptomatic, we use beta blockerssuch as metoprolol & esmolol or calcium channel blockers such as Diltiazem or Verapamil
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Sinus Bradycardia is a normal rhythm but the rate is
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less than 60
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Sinus Bradycardia is caused by
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vagal stimulation, occurs normally in athletes and during sleep, hypothermia, and electrolyte imbalances
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We treat Sinus Bradycardia only if
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symptomatic with atropine and if severe, a pacemaker
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Sinus Arrhythmias are associated with what breathing pattern?
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Cheyne stokes respiration, where the heart rate is up while during inspiration and decreases during expiration
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A sinus arrhythmia can be identified because the
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rate is between 45-100 bpm with normal Pwave,normal PR interval and a normal QRS complex but the \"beats\" are not spaced evenly apart
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Atrial Fibrillation is caused by
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heart failure , CHF
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Atrial Fibrillation is treated by (medications aimed at)
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controlling the rate by use of calcium channel blockers such as Dilitiazem or Verapamil or the use of Beta Blockers such as Metoprolol or Esmolol IF UNSTABLE,SYCHRONIZED CARDIOVERTION
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What is Sychronized Cardiovertion?
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Where a patient with an unstable heart rhythm recieves electrical therapy at the top of each R wave. There MUST BE A RATE to use Sych Cardiovert
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Atrial Fibrillation occurrs when the atrial rate(pace maker sites are) is determined by
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350-600 ectopic foci, which leads to a PR interval that cannot be measured and a QRS complex that is Narrow to normal in duration
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Normal QRS duration is
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.06 -.12 seconds
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A normal PR interval should have a duration of
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.12-.2 seconds
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the normal P wave should precede each
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QRS complex
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Atrial Flutter is caused by
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heart failure and CHF
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How do we treat Atrial Flutter?
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Treatment is aimed at controlling the rate through calcium channel blockers such as Diltiazem or Verapamil or through Beta Blockers such as Metoprolol or Esmolol if the patient is UNSTABLE, SYCHRONIZED CARDIOVERTION
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Atrial Flutter is accompanied by a rapid ventricular response and a reduced cardiac output,this causes the Pwave to have a
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sawtooth wave known as Flutter Waves and a PR INTERVAL that includes a usually constant R wave. The QRS is narrow to normal
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Atrial Flutter has an atrail rate of
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250-350 beats per minute with a regular ventricular rate
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Unifocal Premature Ventricular Contractions are also known as
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Unifocal PVC
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Unifocal PVC's are caused by
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( OCCUR NORMALLY) ,stress, exercise, stimulants, electrolyte imbalances, CAD, heart disease, hypoxemia, and digitalis toxicity
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We treat unifocal PVC's only if symptomatic with
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IV antiarrhythmics such as amiodarone and lidocaine
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Unifocal PVC's originate from what pace maker site?
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the same ectopic focus, so the each look similiar in appearance. They are a regular irregular ventricular beat.
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During the premature beat of a Unifocal PVC, the Pwave is ____,the PR interval ____
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P wave is missing , the PR interval is not measurable, the QRS complex is wide, bizarre, distorted but is in the same shape, because each originates from the same ectopic focus
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Just as with a Unifocal PVC there is no visible P wave in the premature beats of a Multifocal PVC, but the QRS complexes, although still wide, bizarre and distored, they
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have a different shape because they originate is different places.
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Just like Unifocal PVC's, Multifocal PVC's are caused by
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Stress exercise, simulants, electrolyte imbalances, CAD, heart disease, hypoxemia and idopathic (OCCUR NORMALLY)
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Ventricular Bigeminy is noticed by a PVC that occurs rhythmically every
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every other beat, due to one ventricular ectopic focus
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Just like unifocal and multifocal PVC's , Ventricular Bigeminy is caused by
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Stress, exercise,stimulants, electrolyte imbalances, CAD, Heart disease, hypoxemia and idiopathic (OCCUR NORMALLY)
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We treat Ventricular Bigeminy only if symptomatic with
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IV antiarrhythmic medications such as amiodarone and lidocaine.
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We treat Multifocal PVC's only if symptomatic with IV
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Antiarrhythmic medications such as amiodarone and lidocaine
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Ventricular Trigeminy like Unifocal PVC's, Multifocal PVC's, Ventricular Bigeminy occurs normall but is also caused by
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stress , exercise, stimulants, electrolyte imbalances, CAD, heart disease, hypoxemia and idiopathic
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We treat Ventricular Trigeminy ( like all of the other PVC's) only if symptomatic, when symptomatic we administer IV Antiarrhytmics such as
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amiodarone and lidocaine
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We can identify Ventricular Trigeminy when we see that every
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third beat is a wide bizarre distorted QRS complex ( signifying a PVC)
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The PVC site for Ventricular Trigeminy is
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ectopic ventricular focus
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Ventricular Tachycardia is caused by
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acute myocardial ischemia/infarction , electrolyte abnormalities and advanced cardiomyopathy.
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Ventricular Fibrillation is caused by
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acute myocardial ischemia/infarction, electrolyte abnormalities, advanced cardiomyopathy, severe acidosis, electrical shock
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with Ventricular Fibrillation, the waveform is identified by the
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absence of both atrial rate and ventricular rate, the absence of an atrial rhythm and ventricular rhythm, and the absence of identifiable P wave, PR interval and QRS complexes.
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Ventricular Fibrillation is basically just
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impulses from many ectopic foci resulting a chaotic twitching of the ventricles
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Ventricular Fibrillation is treated IMMEDIATELY with
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HIGH QUALITY CPR, DEFIBRILLATION, and medications such as epinephrine or vasopression, amiodarone or lidocaine
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We treat Ventricular Tachycardia
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WHEN NO PULSE, WE TREAT V-TACH THE SAME AS V-FIB! if there IS A PULSE PRESENT, we use antiarrhythmics such as amiodarone or lidocaine or IF UNSTABLE, WE SYNCH CARDIO VERT .
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During V-Tach, there is no measurable atrial rate, but the ventricular rate
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100-250 bpm, AND A VENTRICULAR RHYTHM WITH A REGULAR R to R, there is NO P WAVE with wide QRS complexes
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ASYSTOLE is caused by
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H's and T's, and is treated with HIGH QUALITY CPR, epinephrine or vasopressin, we search for a reversible cause NEVER DEFIBRILLATE THIS RHYTHM!
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during Asystole, there is a cardiac standstill, where there is
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NO ELECTRICAL IMPULSE
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PULSELESS ELECTRICAL ACTIVITY can be any electrical conduction pattern except
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V-Fib and Pulseless V-tach that exists without a pulse
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Pulseless Electrical Activity is caused by
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the H's and T's, is treated with High Quality CPR , medications such as epinephrine and vasopressin, atropine, and searching for a reversible cause DO NOT DEFIBRILLATE THIS RHYTHM
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DISORDERS OF CONDUCTION INCLUDE
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First - degree AV Block, Second-degree AV Block, Mobitz 1, Second-degree AV Block, Mobitz II, and Third degree AV block
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We can identify the First Degree AV Block by
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noticing the constant prolonged P-R interval, the block is present at the AV node
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We treat a first degree heart block with
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atropine only if symptomatic.
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a First Degree Heart Block is caused by
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Drug effects or toxicity , increase of vagal tone, hyperkalemia, MI and myocarditis
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We can identify a Second - degree MOBITZ I
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by the progressive prolongation of the P-R interval until the QRS complex is absent (dropped)
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We treat a second degree heart block Mobitz I only if symptomatic, with
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atropine, TCP/TVP ( trancutaneous pacing, transvenous pacing)
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A SECOND DEGREE MOBITZ I is caused by
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drug effect/toxicity, excessive vagal tone, ischemic heart disease, myocarditis
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A Second Degree heart block MOBITZ II is identified by the normal or prolonged BUT CONSTANT PR INTERVAL until
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the QRS complex is dropped
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a Second Degree heart block MOBITZ II is caused by
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AMI, drug effect/toxicity, valvular disease
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A Second Degree heart block Mobitz II can progress easily to
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a complete heart block
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IF SYMPTOMATIC, treat a Second Degree heart block Mobitz II with
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atropine, TCP/TVP
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A third- degree heart block can be identified due to its complete Atrial and Ventricular Disassociation, in other words,
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the atrial and ventricular contractions occur independantly, therefore, they march out evenly to their own rhythms,There is no relationship between the P waves and QRS complexes
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Circulatory Volume is equal to
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Venous Return
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Contractile force is the same as
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Stroke Volume
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Stroke Volume x Rate =
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Cardiac output
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Cardiac output +Systemic Vascular Resistance =
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Mean Arterial Pressure
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Venous Return ( circulatory volume) has a direct affect on
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cardiac output
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Mean Arterial Pressure directly affects
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tissue perfusion
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What 3 factors affect cardiac output?
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Circulatory Blood Volume(venous return), Heart rate,and Contractile force of the heart ( stroke volume)
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HR x SV=
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Cardiac output
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What 2 factors affect Systemic Vascular Resistance?
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The RADIUS of the VESSEL (vascular tone) and the VISCOSITY OF THE BLOOD
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What regulates vascular tone? or in other words What controls the radius of the vessel?
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The sympathetic nervous system and circulating hormones such as epinephrine, angiotensin and vasopressin
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What is Mean Arterial Pressure?
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The typical measure of Blood Pressure as it is RELATES to PERFUSION
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What affects Mean Arterial Pressure?
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Cardiac Output and Systemic Vascular Resistance
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CO x SVR = MAP or
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HR x SV X SVR= MAP
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MAP is also figured by
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[systolic + diastolic(2)] / 3
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What maintains Tissue Perfusion?
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adequate blood pressure
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Chronotropic is defined as
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Agent affecting the rate of cardiac contraction * very low rates and very high rates lowre cardiac output and can cause arrhythmias
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Inotropic
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agents affecting the force of cardiac contraction
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Preload
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load or tension on the cardiac muscle as it begins to contract
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Afterload
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amount of resistance against which the heart must pump
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Cardiac Glycosides
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increase the force of cardiac contraction = are cardiotonic(inotropic) agents
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Digoxin and Digitoxin are
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cardiac glycosides= are cardiotonic agents ;Inotropic
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Beta-Adrenergbic Cardiac Stimulants are
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Catecholamines such as dobutamine, dopamine(dose specific), Isoproterenol, Epinephrine
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Dopamine is a dose specific inotropic agent, a low dose=
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renal perfusion (dopamenergic receptors)
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a moderate dose of Dopamine =
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increase HR and contractility targeting the beta 1 receptors
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a High dose of dopamine targets alpha receptors and
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causes peripheral vasoconstriction
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Do NOT USE Isoproterenol in
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heart failure because of the increased myocardial oxygen consumption
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Do NOT use Epinephrine in heart failure because
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of the increased myocardial oxygen consumption
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Dobutamine increases the force of contraction without
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increasing blood pressure (beta 1)
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Phosphodiesterase inhibitors are cardiotonic ( Inotropic) agents
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such as Amrinone and milrinone and work by increasing levels of cAMP and increases intracellular calcium