Praxis II (SLP) – Flashcards
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Sternocledomastoid
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Muscle of respiration
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Muscle which opposes velopharyngeal closure
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Lavator veli palatini
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Major acoustic characteristic of voiceless fricatives
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aperiodicity
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Front vowels characterized by
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High frequency second formant (F2)
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Technique which results in temporary facilitation of swallow reflex
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Thermal stimulation
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Stroboscopy
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Most efficient & effective instrument for viewing the vocal folds (VFs)
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Bulbar palsy characterized by
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Flaccid paralysis
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Independent variable
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manipulated to determine its effect on the dependent variable
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Within-subjects design
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dependent variables are measured repeatedly in the same subjects under different tasks/conditions
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between-subjects design
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each participant participates in one and only one group. The results from each group are then compared to each other to examine differences, and thus, effect of the IV. For example, in a study examining the effect of Bayer aspirin vs Tylenol on headaches, we can have 2 groups (those getting Bayer and those getting Tylenol). Participants get either Bayer OR Tylenol, but they do NOT get both
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Multiple baseline design
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The multiple baseline design allows for evaluation across clients, situations, or problems. It is a true experimental design in that it allows for causal inference. It is extremely useful for evaluating situations where an intervention would be likely to bring about enduring changes in the dependent variable
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Flaccid paralysis of soft palate remediated by
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palatal lift appliance to compensation for intact but non-functioning palate
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1st choice of tx for submucous cleft
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surgery
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1st choce for congenitally short palate
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surgery
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Best way to assess swallow (prior to providing speech tx)
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modified barium swallow
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ellipses
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omission of words or other linguistic units b/c they're unnecessary or contextually inferfed.
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deixis
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A word (such as this, that, these, those, now, then) that points to the time, place, or situation in which the speaker is speaking. Also known as deixis.
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Leading causes of aphasia
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CVA, TBI, seizures, tumors, neurodegenerative disorders (dementia & primary progressive aphasia)
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Fluent aphasias
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Wernicke's, conduction, transcortical sensory, anomic
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Anomic aphasia (location)
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tempo-parietal, angular gyrus; second temporal gyrus
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Anomic aphasia (characteristics)
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fluent aphasia, good comprehension & repeition. Word-finding difficulties, decreased output of nouns. Possible alexia & agraphia.
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Conduction aphasia (location)
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path between sensory and motor speech centers (arcuate fasciculus) or insula or deep to supermarginal gyrus
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Characteristics of conduction aphasia
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Fluent aphasia. Preserved comprehension with impaired repetition and literal paraphasias.
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Transcortical sensory aphasia location
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Posterior parieto-temporal, sparing Wernicke's area
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Transcortical sensory aphasia characteristics
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Fluent. Intact repetition, poor auditory comp, paraphasias
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Wernicke's aphasia location
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Posterior third of superior temporal gyrus
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Wernicke's aphasia characteristics
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Fluent but meaningless speech; severe auditory comprehension deficit; jargon, paraphasias, & neologisms; poor reading comprehension; writing deficits
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Nonfluent aphasias
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Broca's, transcortical motor, global aphasia, mixed nonfluent aphasia
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Global aphasia (common location of lesion)
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Left MCA (entire perisylvian region)
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Global aphasia (characteristics)
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Nonfluent aphasia. Ranges from mutism to total repetitive jargon or neologistic output (fluent but incomprehensible speech). Poor comprehension & repetition
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Transcortical motor aphasia location
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frontal lobe; anterior and superior to Broca's area
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Characteristics of transcortical motor aphasia
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Nonfluent. Intact repetition; lack of spontaneous speech; short, telegraphic sentences; agrammatism & paraphasias.
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Broca's aphasia location
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Nonfluent. Lateral frontal, suprasylvian, pre-Rolandic, extending into adjacent subcortical periventricular white matter
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Broca's aphasia characteristics
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Agrammatism; effortful speech; short, telegraphic phrases; presence of apraxia; slow speech rate, lacking intonation; poor reading & writing ability; relatively good auditory comprehension
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Standardized aphasia tests
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Boston Diagnostic Aphasia Examination (BDAE), Western Aphasia Battery (BAD), Minnesota Test of Differential Diagnosis of Aphasia (MTDDA), Multilingual Aphasia Examination (MAE), Porch Index of Communicative Ability (PICA), Aphasia Diagnostic Profiles (ADP)
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Functional aphasia assessment tools
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Functional Communication Profile (FCP), Communicative Abilities in Daily Living (CADL), Communicative Effectiveness Profile (CETI), ASHA Functional Assessment of Communication Skills for Adults (ASHA-FACS
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Prognosis for tx of aphasia is better in patients who are:
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younger & healthier; better educated & in verbally demanding occupations; whose lesions are smaller; who have no othermedical or behavioral disorders; who have good hearing acuity; who have less severe aphasia; whose tx is initiated soon after onset; whose family members are `involved
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Incidence
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Number of new cases of a disorder within a specified period of time
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Commissural fibers
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Facilitates communication btw rt and lft hemispheres by connecting cortical areas of the 2 hemispheres
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Norm-referenced standardized assessments allow...
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comparisons of individual performance on a test w/ that of a defined group
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Reliability
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Results are replicable. If a test is reliable, it yields consistent results. Reliability is a prerequisite for measurement validity
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Split-half reliability
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Test's internal consistency. Scores from one half of the test should correlate with results from the other half the two halves give the same results
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Parallel-forms reliability
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Administering parallel forms of the test to the same group of people.
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Test-retest reliability
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Test's stability over time. Administer the same test multiple times to the same group and the scores should be similar
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Rater reliability
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Level of agreement among individuals rating a test. Intra-rater and Inter-rater
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Intra-rater reliability
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Results are consistent with the same person rating the test more than once
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Inter-rater reliability
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Results are consistent with more than one person rating the test.
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Validity
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A test is valid when it measures what it's supposed to
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Face validity
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It appears to measure what it's supposed to measure
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Content validity
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Tests contents are representative of the skill being assessed
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Construct validity
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Test measures theoretical construct which explains the behavior
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criterion validity
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Use external criteria- concurrent or predictive validity
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Concurrent validity
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Test's validity in comparison to a widely accepted standard
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Predictive validity
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Test's ability to predict performance (ex: GRE)
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CNs involved in swallowing
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Trigeminal (V), Facial (VII), Glossopharyngeal (IX), Vagus (X), Hypoglossal (XII)
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Stages of normal swallow
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Oral preparatory phase, oral phase, pharyngeal phase, esophageal phase
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Describe oral preparatory phase of swallow (adults)
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Food/liquid is manipulated in the oral cavity, chewed (if necessary), and made into a bolus, which is sealed with the tongue against the hard palate
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Describe oral phase of swallow
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Tongue moves food or liquid toward the back of the mouth (toward the anterior faucial pillars). To achieve this, the tongue presses the bolus against the hard palate and squeezes the bolus posteriorly
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Describe pharyngeal phase of swallow
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:Swallow reflex triggered and bolus is carried through the pharynx. These simultaneous actions occur: (a) the velopharyngeal port closes; (b) the bolus is squeezed to the top of the esophagus (cricopharyngeal sphincter); (c) the larynx elevates as the epiglottis, false vocal folds, and true vocal folds close to seal the airway; and (d) the cricopharyngeal sphincter relaxes to allow the bolus to enter the esophagus
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Esophageal phase
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Bolus is transported through the esophagus into the stomach
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Neurological causes associated w/ dysphagia
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CVA, TBI, muscular dystrophy, Parkinson's, myasthenia gravis, ALS, MS, CP
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Non-neurologic causes associated w/ dysphagia
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Head and neck cancer, GERD, esophageal tumors
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Diagnostic tests for dysphagia
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Videofluoroscopy, fiber-optic endoscopy, scintigraphy
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Videofluoroscopy
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e.g. modified barium swallow): a moving radiograph of the mouth, pharynx, larynx, and cervical esophagus during swallowing. can identify the specific nature of the oropharyngeal dysphagia; it can define abnormality of movements, trace progress of bolus, and demonstrate aspiration
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Fiber-optic endoscopy
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useful in assessing swallowing by providing direct observation of pharyngeal activity during the swallowing process
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Scintigraphy
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produces an image of the swallowing mechanism by first covering the vocal tract with a specific nuclide and recording the distribution of the radioactivity w/ a scanning external scintillation camera
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Laryngeal penetration
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occurs when swallowed material penetrates laryngeal side of epiglottis, aryepiglottic folds, or spills over arytrenoid cartilages above level of true VFs
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Aspiration
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swallowed material has entered the trachea below the level of the true vocal folds
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Tx for dysphagia
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Lip exercises, tongue exercises, jaw exercises, swallowing exercises
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Postural tx methods for dysphagia
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Chin tuck, head turn, head tilt, head back, chin tuck w/ head turn
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Chin tuck
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pushes base of tongue towards pharyngeal wall; expands vallecular recesses; narrows entrance to laryngeal vestibule by moving epiglottis posteriorly. Used for delayed onset pharyngeal swallow; reduced base of tongue retraction to posterior pharyngeal wall approximation; decreased airway protection; aspiration DURING swallow
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Head turn (to weak side)
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blocks bolus from traveling down weak side by twisting the pharynx; applies pressure to the vocal fold to increase approximation; reduces resting pressure or the cricopharyngeus by pulling the larynx away from the posterior pharyngeal wall (increasing the space)used for unilateral pharyngeal weakness; unilateral laryngeal weakness; cricopharyngeal dysfunction
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Head tilt (to stronger side)
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directs bolus to stronger side of oral/pharyngeal cavities used for unilateral oral weakness; unilateral pharyngeal weakness
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Head back posture
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used for oral transit dysfunction. gravity helps clear the oral cavity
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Chin tuck w/ head turn
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increases epiglottic deflection to narrow the entrance to the laryngeal vestibule; increases VF approximation by applying extrinsic pressure used for reduced airway closure
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Swallow maneuvers (not appropriate for those w/ cognitive deficits)
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Supraglottic swallow, super-supraglotic swallow, Mendelsohn Maneuver, effortful swallow
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Supraglottic swallow
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Patient holds breath and coughs immediatly following a swallow to close VFs before and during swallow. Used when there is reduced airway protection (at the vocal fold level); Aspiration DURING the swallow
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Super-supraglottic swallow
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Patient holds breath, bears down, and coughs immediately following swallow and immediately swallows hard again. Used when there is reduced airway closure; aspiration BEFORE and DURING the swallow
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Mendelsohn Maneuver
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Increased laryngeal movement stretches/opens the CP; Prolonging hyolaryngeal elevation keeps the CP open longer. used for 1) Decreased range/duration hyolaryngeal elevation; (2) Decreased range/duration cricopharyngeal opening; (3) Decreased pharyngeal swallow coordination
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Effortful swallow
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Used to clear residue in valleculae. The increased effort increases the posterior movement of the base of tongue
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Shaker Exercise & Mendelsohn exercise
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Rehab swallowing exercises to improve function of strap muscles
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Gastrostomy
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creating an opening of a stoma in the stomach wall when normal food ingestion is not possible or ill-advised
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Odynophagia
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pain during swallow
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Surgical/medical tx to protect airway
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Stents, laryngotracheal separation, laryngectomy, trach tubes, feeding tubes
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Surgical treatments to improve glottal closure
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Medialization thyroplasty and injection of biomaterials
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Medialization thryoplasty
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surgical procedure which moves the paralyzed vocal fold closer to the mid glottis to allow better compensation by the unaffected fold
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Surgical/medical tx to improve opening of pharyngoesophageal segment
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Dilatation, Myotomy, Botox Injection
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Bedside swallow exam
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Obtain case history. Observe function of jaw, lips, tongue, phayrnx, larynx, and eating various textures
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Blom-Singer
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indwelling low-pressure voice prosthesis kit (laryngectomy)
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ProVox
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low-resistance indwelling prosthetic (laryngectomy)
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Servox
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transcervical electrolarynx
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Cooper-Rand
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intraoral electrolarynx
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HME device
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Heat and moisture exchanger. Filter placed on tracheostoma which heats and humidifies air. laryngectomy)
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7 Extrinsic muscles of larynx
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Digastric,Stylohyoid, Mylohyoid, Geniohyoid,Hypoglossus, Genioglossus, Thropharyngeus
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7 Intrinsic muscle of larynx
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Cricothyroid, Lateral cricoarytenoid(lateral), Posterior cricoarytenoid, Arytenoid, Thyroarytenoid,Aryepiglottis, Thyroepiglottis
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Muscle with greatest control of fundamental frequency
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Cricothyroid
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Muscle of abduction
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Posterior cricoarytenoid
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Muscles of adduction
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Lateral cricoarytenoid, transverse arytenoid
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Spastic dysphonia
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overadduction of VFs = strained, choked, or creaky voice
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Functional dysphonia
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Likely results in no voice due to underadducted VFs.
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Vocal cord paralysis-vocal characteristics
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hoarse, breathy, decreased vocal intensity, loss of pitch range
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Ventricular dysphonia
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Patient adducts & vibrates ventricular bands instead or in addition to the vocal cords
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Laryngeal web
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Web grows btw VFs, usually triggered by mucosal surface laryngeal injury or irritation. Can cause severe dysphonia and shortness of breath but NOT total absence of voice
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Acute laryngitis
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person may lose the use of voice and may become aphonic during episode. **not approriate to provide voice tx to these individuals
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Vocal nodules characteristics
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hoarse voice quality, hard glottal attacks, and lowering of pitch
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Ventricular phonation
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Person uses false VFs- A rough type of phonation that, when used in conjunction with the true VFs, can result in diplophonia
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Spastic dysphonia
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involves aphonic breaks due to sudden over adduction or under adduction of VFs
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Contact ulcers
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stress, use voice extensively in daily life, has a tense, hard-driving personality, and exhibits glottal fry
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Glottal fry
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when VFs vibrate very slowly and the vibration causes a slow, low pitch vocal burst making the voice sound crackly or creaky, airflow rate and air pressure that produces the VF vibration are both low and lung volume is less
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Reflexive vocalizations
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0-1 months. Crying, coughs, hiccups, related to newborn's physical state
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Cooing
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2-3 mos. Sounds produced w/ a definite stop and start to oral movements. Back consonants and back and middle vowels w/ incomplete resonance
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Babbling
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4-6 mos. Greater independent control of tongue; prolonged strings of sounds; more labial sounds; experiments with sound
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Canonical babbling
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6-10 mos. Repetitive syllable production; increased lip control; labial and alveolar plosives /p, b, t, d/, nasals, and /j/ begin to emerge
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Jargon/1st words
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11-14 mos. Greater variation in the sequences of syllables, creating so-called diverse babbling (e.g. ma-moo-mee); elevates tongue tip; intonational patterns; consistent forms (sound-meaning relationships); predominance of /m, w, b, p/; first words emerge - consist primarily of CV, VC, CVCV reduplicated, and CVCV patterns
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Piagetian theory
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cognitive developmental theory which describes "ages and stages" components that predicts what children can and cannot understand at different ages, and a theory of development that describes how children develop cognitive abilities
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Piaget's stages
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sensorimotor (birth-2yrs), preoperational (2-7 yrs), concrete operational (7-11 yrs), formal operational (11-18+ yrs)
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Information-processing theory
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Humans process the information they receive, rather than merely responding to stimuli. This perspective equates the mind to a computer, which is responsible for analyzing information from the environment
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Social learning theory
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States that people learn within a social context. It is facilitated through modeling and observational learning
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Nativist theories
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Nature. Include Chomsky's transformational grammar (or generative grammar and the theories of Jerry Fodor and Eric Lenneberg. These theories view the acquisition of language as being based more on inherent abilities or mechanisms than on environmental influences
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Behavioral theories
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Nurture. Such as Skinner's find language acquisition to be a form of operant conditioning in which linguistic behavior is shaped by the consequences of verbal responses.
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Empiricist theories
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Recognizes the interaction between nature and nurture, but puts more emphasis on the role of learning, or nurture, and finds that the inherent, or nature, part is a general cognitive learning mechanism
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Emergentist theories
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(e.g. MacWhinney's Competition Model) Such theories claim that language acquisition is a cognitive process emerging from the interactions of biology and the environment.
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/p, m, h, w/ typically mastered
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by age 3
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/b, k, g, t, f, n/ and "ng" typically mastered
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by age 4
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/d/ typically mastered
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by age 5
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/l/ typically mastered
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by age 6
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/s, z, v/, "ch", "sh", "j", voiceless "th", and "zuh" typically mastered
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by age 7
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sounds mastered latest
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/r/ sounds and voiced "th"
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Parents bring their 4-year-old daughter in for SLP evaluations. They are concerned about /r/ sound production and periods of stuttering for the last 18 months. SLP observes that child exhibits sound-syllable repetitions on 30 percent of words. Best action for SLP to take?
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Provide stuttering intervention but no articulation intervention for /r/ at the present time because /r/ typically isn't mastered until after age 7
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Brown's stage I
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Age: 1:0-2:2 yrs MLU: 1.0-2.0
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Brown's stage II
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Age: 2:3-2:6 yrs MLU: 2.0-2.5
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Brown's stage III
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Age: 2:7-2:10 yrs MLU: 2.5-3.0
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Brown's stage IV
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Age: 2:11-3:4 yrs MLU: 3.0-3.75
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Brown's stage V
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Age: 3:5-3:10 yrs MLU: 3.75-4.5
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Stage II- Brown's grammatical morphemes
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Age: 2:3-2:6 yrs Morphemes: Present progressive -ing Prepositions in & on Plural -s Irregular past tense verbs (e.g. I ran)
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Stage III- Brown's grammatical morphemes
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Age: 2:7-2:10 yrs Morphemes: Possessive -'s Uncontractible copula be (e.g. He was sad).
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Stage III-IV Brown's grammatical morphemes
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Age: 2:7-3:4 yrs Morphemes: Articles a, the, an
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Stage V Brown's grammatical morphemes
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Age: 3:5-3:10 yrs Morphemes: Regular past tense -ed Regular 3rd person singular -s (e.g. daddy drives fast). Irregular 3rd person singular (e.g. We did it). Uncontractible auxiliary (e.g. Mommy was sleeping). Contractible copula be (e.g. She's funny. They are funny) Contractible auxiliary (e.g. He's eating. They are eating). *auxiliaries (e.g. will, have, must, would)
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Operant conditioning
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form of learning in which an individual's behavior is modified by its consequences; the behavior may change in form, frequency, or strength
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Classical conditioning
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form of learning in which one stimulus, the conditioned stimulus or CS, comes to signal the occurrence of a second stimulus, the unconditioned stimulus or US
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Waveform
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graph of the amplitude of particle movement (vertical axis) as a function of time (horizontal axis)
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Source-filter theory
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Energy from a sound source is modified by the resonating system (a filter) to yield the acoustic signal of speech. From vowels, the energy source is usually VF vibration and resonating system in vocal tract
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Harmonic
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a whole-number multiple of a fundamental component. For human voice, fundamental component is fundamental frequency of vocal cord vibration (125 Hz for men and 225 Hz for women).
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Formant
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vowels are associated w/ high-energy dark bands that run horizontally. These bands are resonances of the vocal tract called formants
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whispered speech
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aperiodic
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T/F consonants have formants
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False, because they are not periodic (i.e. they are aperodic)
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Spectrogram
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a graphic or photographic representation of a spectrum. 3-D analysis printed on 2-D surface. Horizontal dimension = time; vertical dimension = frequency; darkness = intensity. Each vertical line on a spectrogram represents a vocal fold pulse (single vibration)vowels vary widely in direction
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Velopharyngeal insufficiency
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VP insufficiency occurs due to an anatomical or structural defect. This is most commonly associated with cleft palate. The primary effects of the VP insufficiency are air-flow escape and hypernasality. Secondary effects are disorders in speech articulation (distortions, substitutions and omissions)
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Major factor underlying speech impairment in persons w/ cleft palate
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Palatopharyngeal insufficiency
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Velopharyngeal incompetence
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VP incompetence describes dysfunction of an anatomically intact VP mechanism as in patients with neuromuscular disorders. VP incompetence is traditionally managed by a palatal lift prosthesis.
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Palatal lift
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appropriate when the VPI is caused by poor movement of the velum. The anterior, retaining portion clasps to the teeth and a posterior tailpiece pushes the soft palate up into position to obturate the nasopharynx
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Obturator
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appropriate when the VPI is caused by a deficiency in the length of the soft palate to close the velopharyngeal port.
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Pharyngeal flap
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raises a vertical flap of tissue from the posterior pharyngeal wall. The flap is pulled across the nasopharynx and sutured into the velum. This leaves two openings on either side of the flap that are closed during speech by the inward movement of the lateral pharyngeal walls
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Instruments used to assess velopharyngeal function
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Flexible endoscopy, Videofluoroscopy Cephalometric radiography
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Tx approaches for remediation of speech resonance disorders
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Surgery such as a pharyngeal flap or sphincter pharyngoplasty, may be recommended to correct moderate to severe velopharyngeal dysfunction. Speech therapy is a great tool for children who have speech errors and may be an effective treatment option for mild resonance disorders. The goal of therapy is to help a child learn to use his/her tongue, lips, and velopharyngeal valve correctly
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8-yr-old with repaired palatal cleft has received speech intervention for 2 years to reduce moderately severe hypernasality and nasal emission. Cognitive and linguistic skills are normal. Data supports that abnormal speech characteristics are result of inadequate velopharyngeal mechanism. There have been small improvements, but none in past 6 - 9 months. Which of following is most appropriate course of action for SLP to take at this point
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Refer child to cleft palate tame for consideration of surgical or prosthetic management
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2 months after undergoing surgery to improve velopharyngeal function, a client continues to exhibit nasal airflow and articulatory compensations in production of pressure consonants. The SLP would most appropriately
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provide speech treatment
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8-yr-old with moderately severe hypernasality has persistent nasal regurgitation following and adenoidectomy performed 6-months earlier. Child's speech is likely to show greatest improvement
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with pharyngeal flap surgery
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Substitutions most likely to occur in conjunction with hyponasality
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/b/ for /m/
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Dysarthria
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Neurologically based speech disorder. Many different types. Common to all: impaired muscular control of the speech mechanism and peripheral or central nervous system pathology
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Communication problems in dysarthria include
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respiratory, articulatory, phonatory, resonatory, and prosodic disturbances caused by weakness, incoordination, or paralysis of speech musculature
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Ataxic dysarthria
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Results from damage to cerebellar system. Characterized predominantly by articulatory & prosodic problems
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Ataxic dyarthria physical characteristics
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gait disturbances, over- or undershooting of targets; uncoordinated, jerky, inaccurate, slow, imprecise movemnts
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Ataxic dysarthria communication characteristics
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Artic: imprecise consonants; irregular artic. breakdowns & vowel distortions Prosody: excessive & even stress; prolonged phonemes and intervals btw words or syllables; slow rate of speech. Phonatory: monopitch, monoloudness, and harshness Speech quality: drunken sounding
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Neuropathology of ataxia
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cerebellar lesions, Friedrich's ataxia, TBI, alcohol and drug abuse, meningitis and encephalitis (inflammatory conditions)
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Flaccid dysarthria
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LMN damage. Results from damage to motor units of cranial or spinal nerves that supply speech muscles
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Flaccid dysarthria neuropathology
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myasthenia gravis; vascular diseases & brainstem strokes; infections (e.g. polio and AIDS); dymyelinating disease (e.g. Guillain-Barre syndrome); progressive bulbar palsy & ALS (degenerative diseases); surgical trauma during brain, laryngeal, facial, or chest surgery
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Specific CNs which may be involved in flaccid dysarthria
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trigeminal (V), facial (VII), glossopharyngeal (IX), vagus (X), and hypoglossal (XII) nerves
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Physical characteristics of flaccid dysarthria
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weakness, hypotonia, atrophy, diminished reflexes; twitches of resting muscles (fasciculations) and contractions of individual muscles (fibrillations); rapid and progressive weakness w/ use and recovery with rest
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Communication characteristics of flaccid dysarthria
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Respiration: weakness in combination w/ cranial nerve weakness; Phonatory: breathy voice, audible inspiration, short phrases; Resonance: hypernasality, imprecise consonants, nasal emission, short phrases; Phonatory-prosodic: harsh voice, monopitch, and monoloud
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Hyperkinetic dysarthria
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Results from damage to basal ganglia (extrapyramidal system). Associated w/ involuntary movements and variable muscle tone. Prosodic disturbances are dominant
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Causes of Hyperkinetic dysarthria
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etiology often unknown; include vascular, traumatic, infectious , neoplastic, and metabolic factors; Huntington's disease
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Physical characteristics of hyperkinetic dysarthria
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abnormal & involuntary movements of orofacial muscles; myclonus (involuntary jerks), tics of face and shoulders, tremor, chorea; abrupt & severe contractions of the extremities; writhing, involuntary movements (athetosis); spasms; dystonia (contractions of antagonistic muscles); spasmodic torticollis; blepharospasm
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Communication characteristics of hyperkinetic dysarthria
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depends on dominant neurological condition (e.g. chorea, dystonia, athetosis, etc.) Phonatory: voice tremor, intermittently strained voice, voice stoppage, vocal noise, harsh voice Resonance: predominantly intermittent hypernasality Prosody: slower rate, excess loudness variations, prolonged intervals within words, equal stress Respiratory: audible inspiration and forced and sudden inspiration or expiration Artic: inconsistent problems, including imprecise consonant productions and distortions of vowels
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Hypokinetic dysarthria
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Results from damage to basal ganglia (extrapyramidal system)
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Causes of hypokinetic dysarthria
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Parkinson's, Alzheimer's, multiple or bilateral strokes, repeated head trauma, inflammation, tumor, antipsychotic or neuroleptic drug toxicity, hydrocephalus
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Physical characteristics of hypokinetic dysarthria
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Resting tremors of face, mouth, and limb muscles which DIMINISH when moved voluntarily; mask-like face w infrequent blinking and no smiling; micrographia (small writing); walking disorders (slow to start, then short, rapid, shuffling steps); postural problems such as involuntary flexion of head, trunk, arm, and difficulty chanting positions; decreased swallowing (drooling)
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Communication characteristics of hypokinetic dysarthria:
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Phonatory: monopitch, low pitch, monoloudness, harsh and continuously breathy voice Prosody: reduced stress, inappropriate silent intervals, short rushes of speech, variable and increased rate in segments, short phrases Artic: imprecise consonants, repeated phonemes, resonance disorders and mild hypernasality Respiratory: reduced vital capacity, irregular breathing, faster rate of respiration
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Spastic dysarthria
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Results from bilateral damage to upper motor neurons. Lesions in multiple areas, including cortical areas, basal ganglia, internal capsule, pons, and medulla are common
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Physical characteristics of spastic dysarthria
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spasticity and weakness, especially bilateral facial weakness, though jaw strength may be normal and lower face weakness may be less severe; reduced range and slowness of movement, loss of fine and skilled movement and increased muscle tone; hyperactive gag reflex; hyperadduction of VFs and inadequate velopharyngeal port closure
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Communications characteristics of spastic dysarthria
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Prosody: excess & equal stress, slow rate, monopitch, monoloudness, reduced stress, and short phrases Artic: imprecise consonants & distorted vowels Phonatory: continuous breathy voice, harshness, low pitch, pitch breaks, STRAINED-STRANGLED quality, short phrases, slow rate Resonance: predominant hypernasality
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Mixed dysarthrias
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Combination of two or more pure dysarthrias. Most common mixed types are: flaccid-spastic and ataxic-spastic
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Mixed flaccid-spastic
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associated with ALS
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Characteristics of mixed flaccid-spastic
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imprecise consonants, hypernasality, harsh voice, slow rate, monopitch, short phrases, distorted vowels, low pitch, monoloudness, excess & equal stress and reduced stress, strained-strangled quality, breathiness, etc
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Mixed ataxic-spastic
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associated with MS
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Characteristics of mixed ataxic-spastic
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impaired loudness control, harsh voice quality, imprecise articulation, impaired emphasis, hypernasality, inappropriate pitch levels, and sudden artic breakdowns
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Unilateral Upper Motor Neuron Dysarthria
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Result from damage to upper motor neurons that supply cranial and spinal nerves involved in speech production
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Causes of UUMN dysarthria
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vascular disorders produce lft-hemisphere lesions may coexist w/ aphasia or apraxia; dysarthria due to rt-hemisphere lesions may coexist with rt hemisphere syndrome
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Physical characteristics of UUMN dysarthria
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unilateral lower face weakness, unilateral tongue weakness, unilateral palatal weakness, and hemiplegia/hemiparesis
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Communication characteristics of UUMN dysarthria
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Artic: imprecise consonants and irregular artic breakdowns Phonatory: harsh voice, reduced loudness, strained harshness Prosody: slow rate, increased rate in segments, excess and equal stress, monopitch, monoloudness, low pitch, and short phrases Resonance: predominantly hypernasality Other: dysphagia, aphasia, apraxia, and rt hemisphere syndrome
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Assessment of dysarthria
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1) Record extended conversational speech and reading sample; 2) Use variety of speech tasks (e.g. imitations of syllables, words, phrases, and sentences), sustained vowel prolongation; 3) Diadochokinetic rate or alternating motion rations and sequential motion rates; 4) Assess speech production mech during nonspeech activities (e.g. oral mech exam, coughing, nasal airflow); 5) Assess respiration, phonation, articulation, prosody, and resonance, and intelligibility; 6) Standardized tests (e.g. Assessment of Intelligibility of Dysarthric Speakers & Frenchay Dysarthria Assessment)
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Treatment of dysarthrias
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techniques to modify respiratory, phonatory, articulatory, and resonatory problems are all necessary
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Tx goals in dysarthria
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modification of respiratory, phonatory, articulatory, resonatory, and prosodic problems and increasing the efficiency, effectiveness, and naturalness of communication; increase physiological support and teach self-monitoring skills
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Tx procedures
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Intensive, systematic, and extensive drill, instruction, demonstration, modeling, shaping, prompting, fading, differential reinforcement, and other proven behavioral management procedures. When necessary, phonetic placement and instrumental feedback or biofeedback may be used
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Dementia is associated with
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Wernicke-Korsakoff syndrome (associated with alcoholism), Alzheimer type, frontotemporal dementia, Parkinson's, Huntington's infection, and other (vascular disease, multiple CVAs, TBI)
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Rt hemisphere syndrome
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Symptoms: attentional and affective symptoms; communication deficits found in 50% of cases. Treatment often addresses problems such as impaired attention, impulsive behavior, pragmatic communication impairments and visual neglect
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Tx of patients with TBI
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Cognitive rehabilitation and direct communication training`
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Cognitive rehab following TBI
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Clinicians may train the following: attention, visual processing, and memory
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Communication Tx following TBI
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Involves direct behavioral procedures. Systematic reinforcement of attending behaviors, appropriate discourse, topic maintenance, self-correction, etc. to decrease inappropriate behaviors. Goals should be functional w/ initial focus on effectiveness of communication NOT grammatical correctness. Family members should be involved.
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Apraxia is often associated with lesions in what area?
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Broca's
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Porch Index of Communicative Ability
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Samples speech & language skills to only a limited extent
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Apraxia of speech (AOS)
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Neurogenic speech disorder. A basic disorder of volitional movement in the absence of muscle weakness, paralysis or fatigue.
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Locus of AOS
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caused by damage or injury to speech-motor programming areas (e.g. Broca's) in the dominant hemisphere
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T/F patients with AOS have rapid rate of speech?
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False. They typically have a reduced rate
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Characteristics of AOS
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Consonants more impacted than vowels; increased frequency of errors on longer words; groping behaviors; automatic productions easier than volitional productions; awareness of errors; speech sound substitutions common, particularly substitution of voiceless phoneme for voiced (e.g. "pet" for "bet"); may make several unsuccessful attempts to self-correct
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Assessing AOS
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Repetitive production of single- and multiple syllables (e.g. "puh-ta-kuh", multisyllabic words); imitative production of progressively longer words, phrases, and sentences; picture description tasks; assess oral reading; assess overall movement of limbs; administer standardized tests (e.g. Apraxia Battery for Adults)
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Tx of AOS
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Auditory-visual stimulation, oral-motor repetition, & phonetic placement
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Progression of Tx of AOS
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Carefully sequenced to move from automatic simple productions to less automatic, more spontaneous productions