question

Epinephrine trade name

answer

Adrenaline

question

Epinephrine Pharmacological Class

answer

adrenergics

question

Epinephrine Therapeutic Classification

answer

Antiasthmatics, bronchodilators, vasopressors

question

Epinephrine Indications

answer

Management of reversible airway disease due to asthma or COPD, Management of severe allergic reactions, Management of cardiac arrest, management of upper airway obstruction and croup, and adjunct in the location/prolongation of anesthesia.

question

Epinephrine Action

answer

Results in beta1 (cardiac) adrenergic receptors and beta2 (pulmonary) adrenergic receptor sites.

question

Epinephrine Adverse reactions

answer

Nervousness, restlessness, tremor, angina, arrhythmias, HTN, tachycardia

question

Epinephrine Route

answer

Sub-q, IM, IV, inhalin, intracardiac, topical, intraspinal

question

Epinephrine Nursing implications

answer

Monitor BP, P, ECG, RR, I&Os, chest pain, arrhythmias, HR >110 BPM, signs and symptoms of HTN, and assess volume status.

question

Prazosin trade name

answer

Minipress

question

Prazosin Pharmacological class

answer

Peripherally acting adrenergics

question

Prazosin Therapeutic class

answer

Antihypertensives

question

Prazosin Indications

answer

Mild-Moderate HTN

question

Prazosin Action

answer

Dilates both arteries and veins by blocking postsynaptic alpha1-adrenergic receptors

question

Prazosin Therapeutic action

answer

decreased BP, decreased cardiac preload and afterload

question

Prazosin Adverse reactions

answer

Dizziness, HA, weakness, first dose orthostatic hypotension, paliptations

question

Prazosin Route

answer

PO

question

Prazosin Nursing Implications

answer

Assess for first dose othostatic reaction (dizziness, weakness) and syncope. May occur 30min-2hr after initial dose and occasionally thereafter. Monior I&Os and daily wt, and edema. Give at bedtime. May be used in combination with diuretics or beta blockers to minimize sodium and water retention.

question

Clonidine Trade name

answer

Catapres

question

Clonidine Pharmacological class

answer

Centrally acting Adrenergics

question

Clonidine Therapeutic class

answer

Antihypertensives

question

Clonidine Use- PO and Transdermal

answer

Mild-Moderate HTN

question

Clonidine Action

answer

Stimulates Alpha-adrenergic receptors in the CNS, which results in decreased sympathetic outflow inhibiting cardioacceleration and vasoconstriction centers.

question

Clonidine Therapeutic effects

answer

Decreased BP

question

Clonidine Adverse reactions

answer

Drowsiness, dry mouth, withdrawal phenomenon

question

Clonidine Route

answer

PO, transdermal, epidural

question

Clonidine Nursing implications

answer

Monitor I&Os, daily wt, and BP. Encourage patient to comply with additional interventions for HTN (wt reduction, decreased sodium diet, no smoking, moderation of alcohol consumption, regular exercise, and stress management).

question

Clonidine labs

answer

Decrease blood glucose, may cause weakly positive coombs test result

question

Captopril trade name

answer

Capoten

question

Captopril Pharmacological class

answer

Ace inhibitor

question

Captopril Therapeutic class

answer

Antihypertensive

question

Captopril Indications

answer

Mangement of HTN, HF, reduction of risk of death or development of HF following MI, decreased progression of diabetic neuropathy

question

Captopril Action

answer

ACE inhibitors block the conversion of angiotension I to the vasocontrictor angiostensin II. Ace inhibitors also prevent the degradation of bradykinin and other vasodilatory prostaglandins. Also increase plasma renin levels and decrease aldosterone levels. Net result is systemic vasodilation, lower BP, and decrease s/s of HF

question

Captopril adverse reactions

answer

Cough, hypotension, taste disturbances, angioedema, and neutropenia (captopril only)

question

Captopril Route

answer

PO

question

Capoten Nursing implications

answer

Monitor BP, P, signs of angioedema, and signs of fluid overload. Admin in one hour before or 2 hours after meal. Pill may be crushed.

question

Captopril labs

answer

Monitor CBC with diff prior to therapy, every two weeks for the first three months, and periodically for up to one year in patients at risk for neutropenia or at first sign of infection. D/C if neutrophil is <1000 mm3

question

Enalapril trade name

answer

Vasotec

question

Enalapril Pharmacological class

answer

ACE inhibitors

question

Enalapril Therapeutic class

answer

Antihypertensives

question

Enalapril Indications

answer

Management of HTN, HF, slowed progression of left ventriular dysfunction into overt heart failure.

question

Enalapril Action

answer

Block the conversion of angiotensin I into the vasocontrictor angiotensin II. Also prevent the degradation of bradykinin and other vasodilatory prostaglandins. Increases plasma renin levels and decreases aldosterone levels. Net result is systemic vasodilation and lower BP.

question

Enalapril Adverse reactions

answer

Cough, hypotention, taste disturbances, angioedema

question

Enalapril Route

answer

PO, IV

question

Enalapril Nursing implications

answer

Monitor BP, P. Assess patient for signs of angioedema (dyspnea, facial swelling). HF- monitor for signs of fluid overload.

question

Enalapril Labs

answer

Monitor BUN, creatinine, CBC, and electrolyte levels periodically during therapy. Potassium, BUN, and creatinine levels may be increased. Sodium levels may be decreased. May cause increased ALT, AST, alkaline phosphate, bilirubin, uric acid, and glucose.

question

Lisinopril Trade name

answer

Prinivil

question

Lisinopril Pharmacological class

answer

ACE inhibitor

question

Lisinopril Therapeutic classification

answer

Antihypertensives

question

Lisinopril Indications

answer

Mananement of HTN, HF, decreased risk of death or development of HF following MI.

question

Lisinopril Action

answer

Blocks the conversion of angiotensin I into the vasoconstrictor angiotensin II. ACE inhibitors also prevent the degradation of bradykinin and other vasodilatory prostaglandins. Also increases plasma renin levels and decreases aldosterone levels.

question

Lisinopril Adverse effects

answer

Cough, hypotension, taste disturbances, angioedema

question

Lisinopril Route

answer

PO

question

Lisinopril Nursing implications

answer

Monitor BP, P, signs of angioedema. HF: wt, assess for s/s of fluid overload (peripheral edema, rales/crackles, dyspnea, wt gain, jugular venous distention)

question

Lisinopril labs

answer

Monitor BUN, creatinine, electrolyte, and CBC levels.

question

Losartan Trade name

answer

Coozar

question

Losartan Pharmacological class

answer

Angiotensive II receptor blocker (ARB)

question

Losartan Therapeutic class

answer

Antihypertensive

question

Losartan Indications

answer

Alone or with other agents in the management of HTN. Treatment of diabetic neuropathy in patients with type to diabetes and HTN. Prevention of stoke in patients with HTN and left ventricular hypertrophy.

question

Losartan Action

answer

Blocks vasoconstrictor and aldosterone-producing effectss of angiotensin II at receptor sites, including vascular smooth muscle and the adrenal glands.

question

Losartan Adverse effects

answer

Dizziness, Hypotension, angioedema

question

Losartan Route

answer

PO

question

Losartan Nursing Implications

answer

Assess ortho BP, P. Assess for s/s of angioedema. HF: Daily wt and assess for s/s of fluid overload

question

Losartan Labs

answer

Monitor renal function and electrolyte levels. Potassium, BUN, and creatinine may increase. May increase uric acid, slight decrease in H&H and thrombocytopenia.

question

Atenolol Trade name

answer

Tenormin

question

Atenolol Pharmacological class

answer

Beta Blocker

question

Atenolol Therapeutic class

answer

Antianginals, antihypertensives

question

Atenolol Indications

answer

Management of HTN, angina pectoris, prevention of MI

question

Atenolol Action

answer

Blocks stimulation of beta1 (myocardial)-adrenergic receptors. Does not usually affect beta2 (pulmonary, vascular, uterine) receptor sites.

question

Atenolol Therapeutic effects

answer

Decreased BP and HR

question

Atenolol Side effects

answer

Fatigue, weakness, erectile dysfunction, bradycardia, HF, pulmonary edema

question

Atenolol Nursing implications

answer

Monitor P, BP, ECG, I&Os, daily wt, signs of HF. Take apical pulse before admin. If <50 bpm withhold and contact MD.

question

Atenolol Route

answer

PO

question

Atenolol Labs

answer

May increase BUN, serum lipoprotien, potassium, triglyceride, and uric acid levels. May increase ANA titer and blood glucose levels.

question

Atenolol s/s of overdose

answer

bradycardia, severe dizziness or fainting, severe drowsiness, dyspnea, bluish fingernails or palms, seizures

question

Metoprolol Trade name

answer

Lopressor

question

Metoprolol Pharmacological class

answer

Beta Blocker

question

Metoprolol Therapeutic class

answer

Antianginals, antihypertensives

question

Metoprolol Indications

answer

HTN, angina pectoris, prevention of MI and decreased mortality in patients with recent MI. Management of stable, symptomatic (class II & III) HF. Unlabeled use: Ventricular arrhythmias/tachycardia.

question

Metoprolol Action

answer

Blocks stimulation of beta1 (myocardial)-adrenergic receptors. Does not usually affect beta2 (pulmonary, vascular, uterine)-adrenergic receptor sites.

question

Metoprolol Therapeutic effects

answer

Decrease BP & HR

question

Metoprolol Adverse effects

answer

Fatigue, weakness, erectile dysfunction Serious: Bradycardia, HF, pulmonary edema

question

Metoprolol Route

answer

PO, IV

question

Metoprolol Nursing implications

answer

Take apical pulse before admin and if below 50 bpm hold drug and contact MD. Admin with meals. Monitor BP, P, ECG, P, I&O, daily wt, s/s of HF, and anginal attacks.

question

Metoprolol Labs

answer

May increase BUN, serum lipoprotein, potassium, triglyceride, and uric acid levels. May increase ANA titers and blood glucose levels.

question

Propranolol Trade name

answer

Inderal

question

Propranolol Pharmacological Class

answer

Beta Blockers

question

Propranolol Therapeutic class

answer

Antianginals, antihypertensives, antiarrythmics, vascular HA supressants

question

Propranolol Indications

answer

Management of: HTN, angina, arrhythmias, hypertrophic cardiomyopathy, thyrotoxicosis, essential tumors, and pheochromoctoma. Also used in prevention and management of MI and the prevention of vascular HA.

question

Proponolol Action

answer

Blocks stimulation of beta1 (myocardial) and beta2 (pulmonary, vascular, uterine)-adrenergic receptor sites.

question

Propanolol Therapeutic effects

answer

Decreased BP & HR

question

Proponolol Adverse effects

answer

Fatigue, weakness, erectile dysfunction Serious: Arrhythmias, HF, bradycardia, pulmonary edema, steven-johnson's syndrome, anaphylaxis

question

Propanolol Route

answer

PO, IV

question

Proponolol Nursing implications

answer

Monitor BP, P, taper drug to stop, assess for ortho hypotension, I&Os, daily wt, s/s of fluid overload & rash r/t steven johnson's syndrome. Admin with meals or directly after.

question

Propanolol Labs

answer

Increases BUN, serum lipoprotein, potassium, triglyceride, and uric acid levels.

question

Nifedipine Trade name

answer

Procardia

question

Nifedipine Pharmacological class

answer

Calcium channel blockers

question

Nifedipine Therapeutic class

answer

Antianginals, antihypertensives

question

Nifedipine Indications

answer

Management of: HTN (extended release only), angina pectoris, vasoplastic (prinzmetal's) angina

question

Nifedipine Action

answer

Inhibits calcium transport into myocardial and vascular smooth muscle cells, resulting in inhibition of excitation-contraction coupling and subsequent contraction

question

Nifedipine Therapeutic effects

answer

Systemic vasodilation, resulting in decreased BP, coronary vasodilation resulting in decreased frequency and severity of anginal attacks.

question

Nifedipine Adverse effects

answer

HA, peripheral edema, flushing Serious: Arrhythmias, HF, steven johnson's syndrome

question

Nifedipine Route

answer

PO

question

Nifedipine Nursing implications

answer

Monitor BP, P, ECG, I&O, daily wt, s/s of HF, assess for rash, and anginal pain.

question

Nifedipine Labs

answer

Calcium levels are unchanged. Potassium decreases will increase risk for arrhythmias so closely monitor and correct immediately. Increased hepatic enzymes and may cause positive ANA and direct coombs test results.

question

Verapamil Trade name

answer

Calan

question

Verapamil Pharmacological class

answer

Calcium Channel Blockers

question

Verapamil Therapeutic class

answer

Antianginals, antiarrhymics (class IV), antihypertensives, vascular HA suppressants

question

Verapamil Indications

answer

Management of HTN, angina pectoris and/or vasoplastic (primzmetal's) angina. Management of supraventricular arrhythmias and rapid ventricular rates in atrial flutter or fibrillation

question

Verapamil Action

answer

Inhibits the transport of calcium into myocardial and vascular smooth muscle cells, resulting in inhibition of excitation-contraction coupling and subsequent contraction. Decreased SA & AV conduction and prolongs AV node refractory period in conduction tissue.

question

Verapamil Therapeutic effects

answer

Decreased BP, ventricular rate, atrial flutter

question

Verapamil Adverse effects

answer

Generally well tolerated. Constipation being the cheif complaint. Serious: steven johnson's syndrome

question

Verapamil Route

answer

PO, IV

question

Verapamil Nursing implications

answer

Monitor BP, P, ECG, I&Os, daily wt, signs of HF, monitor for rash, assess anginal pain if present. Admin with meals or milk. Do not crush.

question

Amlodipine Trade name

answer

Norvasc

question

Amlodipine Pharmacological class

answer

Calcium channel blocker

question

Amlodipine Therapeutic class

answer

antihypertensive

question

Amlodipine Indications

answer

Alone or with other agents in the management of HTN, angina pectoris and vasoplastic (prinzmetal's) angina

question

Amlodipine Action

answer

Inhibits the transport of calcium into myocardial and vascular smooth muscle cells resulting in the inhibition of excitation-contraction coupling and subsequent contraction

question

Amlodipine Therapeutic effects

answer

Systemic vasodilation resulting in decreased BP

question

Amlodipine Adverse effects

answer

HA, peripheral edema

question

Amlodipine Route

answer

PO

question

Amlodipine Nursing implications

answer

Hold of systolic BP is >90 mm/hg. Monitor BP, P, ECG, I&O, daily wt, s/s of HF, and assess duration, locations, intensity, and precipitating factors of patients anginal pain.

question

Amlodipine Labs

answer

Serum calcium not effected.

question

Amiodarone Trade name

answer

Pacerone

question

Amiodarone Therapeutic class

answer

Antiarrhythmics (class III), Potassium channel blocker

question

Amiodarone Indications

answer

Life threatening ventricular arrhythmias-unresponsive to less toxic agents

question

Amiodarone Action

answer

Prolongs action potential and refractory period. Inhibits adrenergic stimulation, slows the sinus rate, increases PR and QT intervals, and decreases peripheral vascular resistance (vasodilation)

question

Amiodarone Therapeutic effects

answer

Suppression of arrhythmias

question

Amiodarone Adverse effects

answer

Dizziness, fatigue, malaise, corneal microdeposits, bradycardia, hypotension, anorexia, constipation, N, V, peripheral neuropathy, poor circulation, tremor Serious: ARDS, pulmonary fibrosis/toxicity, CHF, worse arrhythmias Rare: Toxic epidermal necrolysis

question

Amiodarone Route

answer

PO, IV, intaosseous (children)

question

Amiodarone Nursing implications

answer

Monitor ECG, BP, HR and rhythm, bradycardia, and increase in arrhythmias report immediately. Assess for s/s of pulmonary toxicity, ARDS, neurotoxicity, and thyroid dysfunction

question

Digoxin Trade name

answer

Lanoxin

question

Digoxin Pharmacological class

answer

digitalis glycosides

question

Digoxin Therapeutic class

answer

antiarrhythmics, inotropics

question

Digoxin Indications

answer

HF, atrial fibrillation, and atrial flutter (slows ventricular rate). Paroxysmal atrial tachycardia

question

Digoxin Action

answer

Increases the force of myocardial contraction. Prolongs refractory period of the AV node. Decreases conduction through SA and AV nodes

question

Digoxin Therapeutic effects

answer

Increases cardiac output (positive inotropic effect) and slowing of the HR (negative chronotropic effect)

question

Digoxin Adverse effects

answer

Fatigue, bradycardia, anorexia, N, V Serious: Arrhythmias

question

Digoxin Route

answer

PO, IV

question

Digoxin Nursing implications

answer

Monitor apical pulse for 1 full minute before admin. Hold if <60 BPM. Hypokalemia, hypomagnesemia, or hypercalcemia increase susceptibility to digitialis toxicity. Monitor labs and digoxin levels (normal 0.5-2 ng/ml)

question

Digoxin Overdose

answer

admin: digoxin immune fab (digifab)

question

Spironolactone Trade name

answer

Aldactone

question

Spironolactone Pharmacological class

answer

Potassium sparing diuretics

question

Spironolactone Therapeutic class

answer

diuretics

question

Spironolactone Indications

answer

Counteract potassium loss caused by other diuretics. Used with other agents (thiazides) to treat edema or HTN. Primary aldosteronism. Unlabelled use: Management of HF (low doses).

question

Spironolactone Adverse effects

answer

Hyperkalemia Serious: Steven johnson's syndrome, toxic epidermal necrolysis

question

Spironolactone Route

answer

PO

question

Spironolactone Nursing implications

answer

Monitor I&Os, daily wt, BP(for adjunct with antihypertensive), monitor for s/s of hypo/hyperkalemia. Assess frequently for rash. May increase digoxin levels. Admin in AM to avoid interrupting sleep pattern

question

Spironolactone Labs

answer

Monitor potassium and urinary calcium excretion levels, magnesium, BUN, creatinine, can increase. May decrease sodium levels.

question

Furosemide Trade name

answer

Lasix

question

Furosemide Pharmacological class

answer

Loop diuretics

question

Furosemide Therapeutic class

answer

Diuretic, antihypertensive

question

Furosemide Indications

answer

Edema due to: HF, hepatic or renal disease, HTN, unlabeled use: hypercalcemia of malignancy

question

Furosemide Action

answer

Inhibit the reabsorption of sodium, and chloride from the loop of henle and distal renal tubule. Increases renal excretion of water, sodium, chloride, magnesium, hydrogen, and calcium. May have renal and peripheral vasodilatory effects

question

Furosemide Adverse effects

answer

Dehydration, hypochloremia, hypokalemia hypomagnesemia, hyponatremia, hypovolemia, metabolic alkalosis Seious: Steven johnson's syndrome, toxic epidermal necrolysis

question

Furosemide Route

answer

PO, IV, IM

question

Furosemide Nursing implications

answer

Monitor I&Os, daily wt, edema, lung sounds, skin tugor & mucous membranes, BP, P, assess for digoxin toxicity, allergy and rash.

question

Furosemide Labs

answer

May increase BUN, serum glucose, creatinine, and uric acid levels. Decreases potassium, calcium, and magnesium levels.

question

Hydrochlorothiazide Trade name

answer

Apo-hydro, microzide, urozide

question

Hydrochlorothiazide Pharmacological class

answer

Thiazide diuretics

question

Hydrochlorothiazide Therapeutic class

answer

Antihypertensives, diuretics

question

Hydrochlorothiazide Indications

answer

Management of mild-moderate HTN. Treatment of edema associated with HF, renal dysfunction, cirrhosis

question

Hydrochlorothiazide Action

answer

Increases excretion of chloride, potassium, magnesium, and bicarbonate ions. May produce arteiolar dilation

question

Hydrochlorothiazide Therapeutic effects

answer

Decrease in BP in HTN pts and diuresis with mobilization of edema

question

Hydrochlorothiazide Adverse effects

answer

Acute angle-closure glaucoma, acute myopia, hypokalemia Serious: Steven johnson's syndrome

question

Hydrochlorothiazide Route

answer

PO

question

Hydrochlorothiazide Nursing Implications

answer

Monitor BP, I&O, daily wt, check for edema, and if taking digoxin check for toxicity. Check for rash and if hypokalemia occurs may give supplement or decrease dose. Give with food in the AM.

question

Hydrochlorothiazide Labs

answer

May increase serum and urine glucose in diabetics. Increase in serum bilirubin, calcium, creatinine, uric acid, and decrease in magnesium, potassium, sodium, and urinary calcium concentrations.

question

Vancomycin Trade name

answer

Vancocin

question

Vancomycin Therapeutic class

answer

anti-infectives

question

Vancomycin Indications

answer

Treatment of potentially life threatening infections when less toxic anti-infectives are contraindicated. Particularly useful in staph infections.

question

Vancomycin Action

answer

Inhibits the second stage of cell wall synthesis of susceptible bacteria

question

Vancomycin Adverse Reactions

answer

Nephrotoxicity, phlebitis Serious: Hypersensitivity reactions including anaphylaxis, chills, fever, red man syndrome (with rapid infusion), superinfection

question

Vancomycin Route

answer

IV, IT, PO

question

Vancomycin Nursing implications

answer

Check patency of IV line, draw vancomycin troph and peak, monitor for red man syndrome

question

Enoxaparin Trade name

answer

Lovanox

question

Enoxaparin Pharmacological class

answer

antithrombotics

question

Enoxaparin Therapeutic class

answer

anticoagulants

question

Enoxaparin Indications

answer

Prevention of venous thromboelbolism (VTE), deep vein thrombosis (DVT), and/or pulmonary embolism (PE) in surgical or MED patients. Treatment of DVT with or without PE.

question

Enoxaparin Action

answer

Potentiates the inhibitory effect of antithrombin on factor Xa and thrombin

question

Enoxaparin Adverse effects

answer

bleeding, anemia

question

Enoxaparin Route

answer

Sub-q, IV

question

Enoxaparin Nursing implications

answer

Assess for s/s of bleeding, hemorrhage, additional or increased thrombosis, anginal pain, hypersensitivity reactionsw

question

Enoxaparin Labs

answer

aPTT is not necessary. Monitor CBC, platelet count, and stools for occult blood. If thrombocytopenia occurs, monitor closely. If hematocrit decreases unexpectedly, assess patient for potential bleeding sites.

question

Heparin Trade name

answer

Hep-Lock

question

Heparin Pharmacological class

answer

Antithrombotics

question

Heparin Therapeutic class

answer

Anticoagulants

question

Heparin indications

answer

Prophylaxis and treatment of: venous thromboembolism, pulmonary emboli, atrial fibrillation with embolism, acute and chronic consumptive coagulopathies, peripheral arterial thromboembolism

question

Heparin Action

answer

Potentiates the inhibitory effect of antithrombin on factor Xa and thrombin

question

Heparin Adverse effects

answer

Bleeding, anemia, heparin induced thrombocytopenia (HIT) with or without thrombosis

question

Heparin Route

answer

IV, SUB-Q

question

Heparin nursing implications

answer

Protamine sulfate is the antidote to overdose (1mg per 100 units heparin)

question

Heparin Labs

answer

Measurements of aPTT are normally 40 seconds. Heparin increases levels to 60-80 seconds. Should be made every 4-6 hours. Once therapy is established once a day will suffice.

question

Warfarin Trade name

answer

Coumadin

question

Warfarin Pharmacological class

answer

coumarins

question

Warfarin Therapeutic class

answer

anticoagulants

question

Warfarin Indications

answer

Prophylaxis and treatment of: Venous thrombosis, pulmonary embolism, atrial fibrillation with embolization,. Management of MI: decrease risk of death, decrease risk of subsequent MI, decreased risk of future thromboembolic events. Prevention of thrombus formulation and embolization after prosthetic valve placement.

question

Warfarin Action

answer

Interfers with hepatic synthesis of vitamin k dependent clotting factors (II, VII, IX, X)

question

Warfarin Adverse effects

answer

Serious: Bleeding

question

Warfarin Route

answer

PO, IV

question

Warfarin Nursing implications

answer

Assess for signs of bleeding and hemorrhage and 4 evidence of more or increased thrombosis

question

Warfarin labs

answer

Monitor pt/INR frequently during therapy. Normal INR is 0.2-1.2. An INR of 2.5-3.5 is recommended for patients at very high risk of embolization. Monitor hepatic function and CBC before and periodically throughout therapy

question

Warfarin Overdose

answer

Antidote is vitamin K

question

Acetylsalicylic acid Trade name

answer

Aspirin

question

Aspirin Pharmacological class

answer

Salicylates

question

Aspirin Therapeutic class

answer

antipyretics, nonopioid analgesics

question

Aspirin Indications

answer

Inflammatory disorders including: Rheumatoid arthritis, osteoarthritis. Mild to moderate pain, fever, and prophylaxis of transient ischemic attacks and MI.

question

Aspirin Action

answer

Produce analgesia and decrease inflammation and fever by inhibiting the production of prostaglandins. Decrease platelet aggregation

question

Aspirin Adverse effects

answer

Dyspepsia, epigastric distress, N Serious: allergic reactions including anaphylaxis and laryngeal edema, GI bleeding

question

Aspirin Route

answer

PO, Rectal

question

Aspirin Nursing implications

answer

Assess for bleeding, pain.

question

Aspirin Labs

answer

Monitor hepatic function. May increase serum AST, ALT, and alkaline phosphate. Prolongs bleeding time for 4-7 days.

question

Aspirin toxicity

answer

Monitor for onset of tinnitus, HA, hyperventilation, agitation, mental confusion, lethargy, D, and sweating

question

Clopidogrel Trade Name

answer

Plavix

question

Clopidogrel Pharmacological class

answer

Platelet aggregation inhibitors

question

Clopidogrel Therapeutic class

answer

Antiplatelet agents

question

Clopidogrel Indications

answer

Reduction of atherosclerotic events (MI, stroke, vascular death) in pts at risk for such events including recent MI, acute coronary syndrome (unstable angina/non-Q-wave MI), stoke, or peripheral vascular disease

question

Clopidogrel Action

answer

Inhibits platelet aggregation by irreversibly inhibiting the binding of ATP to platelet receptors

question

Clopidogrel Adverse effects

answer

Serious: GI bleeding, bleeding, neutropenia, thrombotic thrombocytopenic pupura (TTP)

question

Clopidogrel Route

answer

PO

question

Clopidogrel Nursing Implications

answer

Monitor pt for s/s of stoke, peripheral vascular disease or MI, also for signs of TTP (thrombocytopenia, neurological findings, renal dysfunction, fever)----Requires prompt treatment

question

Clopidogrel labs

answer

Monitor bleeding time, CBC with diff, and platelet count

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