3101AHS Medical Nutrition Therapy – Flashcards

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MNT Definition
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Therapeutic approach to treating medical conditions via use of diet devised by a medical doctor or registered dietitian
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ADIME Acronym
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Assessment Diagnosis Intervention Monitoring Evaluation
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5 Domains of Nutrition Assessment (ABCDN)
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Anthropometric measures Biochemical data / medical tests Client history Diet / food related history Nutrition focused physical findings
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Nutrition Diagnosis definition and domains
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Finds nutrition PROBLEM that can be treated alone by dietitian from 3 domains - intake, behaviour, clinical
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PES Statements
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Problem (P) related to etiology (E) as evidenced by signs and symptoms (S).
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Behavioural-Environmental Domain
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Problems that relate to knowledge, attitudes/beliefs, environment, food access or food safety
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Nutrition Intervention
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Actions to positively change a behaviour, environmental condition, or nutritional health status (work w/other health professionals) Direct interventions to etiology
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4 Domains of Intervention
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Food & nutrient delivery Nutrition education Nutrition counselling Co-ordination of nutrition care
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Four domains of monitoring and evaluation
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- Anthropometric measurements - Biochemical Data - Diet related history - Nutrition focused physical findings
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Search Terms - PICO
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Participants (patient* or inpatient* or hospital*) AND adults Intervention/control: (feed* or meal* or assistan*) Outcome
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Primary Deficiency Definition + two examples
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e.g Iron deficiency anemia, scurvy Caused by low level of nutrient in diet
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Secondary Deficiency Definition
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e.g lack of intrinsic factor Factors that can interfere with ingestion, digestion, absorption or utilisation
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Dietary Assessment Tools - Retrospective
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FFQ - measuring dietary patterns 24hr recall Diet history
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Dietary assessment tools - Prospective
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Food records Food surveillance (covert vs. known) Food inventory (plate waste)
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FFQ
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Semi-quantitative at best Most effective for assessing population intakes Recall bias
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24hr Recall
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Recall bias Underestimates by 10% Depends on regular eating habits
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Diet History
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Quantitative + qualitative, retrospective Reasonably accurate estimates Portion size difficult Recall bias
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Australian Dietary guideline 1
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To achieve + maintain healthy wt, be active and choose of nutritious food to meet energy needs.
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Australian Dietary guideline 2
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Enjoy wide variety of nutritious foods from five food groups every day + drink lots of water
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Australian Dietary guideline 3
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Limit intake of foods containing saturated fat, added salt, added sugars and alcohol
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Australian Dietary guideline 4
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Encourage, promote, support breastfeeding
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Australian Dietary guideline 5
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Care for your food - prepare and store safely
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EAR
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daily nutrient level estimated to meet requirements of 50% of healthy individuals
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RDI
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EAR+2SD avg. daily intake to meet nutrient requirements of almost all healthy individuals Don't use to assess groups
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AI
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Used when RDI can't be calculated. Avg. daily nutrient level based on observed/experimental estimates or estimates of a group of apparently healthy people that are assumed to be adequate
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Anthropometry Limitations
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Insensitive, unable to distinguish btwn disturbances from nutrient deficiencies or from imbalances in protein and energy
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Measurements for adults
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weight, height, BMI, WC, HC, WHR, MUAC, TSK
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Measurements for children
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weight, height, head circumference, HC for age, ht for age, wt of rage, wt for ht
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Measurements for sport
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sum of 7 skin folds, DXA, BIA
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Measurements for research
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DXA, BIA, Air displacement plethysmography
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How to interpret BMI
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Severely underweight <16kg/m² Moderately underweight 16 16.99kg/m² Mildly underweight 17‐18.49kg/m² Healthy weight 18.5‐25kg/m² Overweight 25‐30kg/m² Obese class I 30 - 35kg/m² Obese class II 35 - 40kg/m² Obese class III ≥40kg/m²
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BMI Doesn't reflect
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Distribution of body fat, Muscle Mass, Age, Family History, Gender, Ethnicity, Lost Height due to aging
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BMI unsuitable for
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Pregnant / lactating women, <18 years, ht 190cm, extreme obesity, eating disorders
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Wait Circumference Interpretation
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Men: >94cm = At risk > 102cm = High risk Women: >80cm = At risk > 88cm = High risk
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Waist Hip Circumference Ratio
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Distinguishes between fatness in the lower trunk and fatness in the upper trunk
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Waist Hip Ratio Interpretation
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Increased risk at Men >1.0 Women >0.85
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MUAC Average Values
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M - 29.3cm F - 28.5cm
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Tricep Skin-fold Interpretation
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Correlates with % of body fat Varies between 6-12mm in lean individuals 40-50mm in obese individuals
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Tricep Skin-fold Errors
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Parallax Error- if the assessor bends down to read caliper Intra-examiner- due to inexperience, skinfold site, difficulty in determining subcut fat from muscle in the subject
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Interpreting Child Growth Measurements
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Use reference data, previous measurements of same child, CDC / WHO charts usually used < 32 weeks = low birthweight chart 32-37 week plotted in 'pre-term chart' until 2 weeks after estimated due date
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Reason Child is >95th Percentile for Weight
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Tallness/Tall parents, Obesity, Oedema
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Reason Child is >95th Percentile for Height
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Tallness/Tall parents, Accelerated maturation, Marfan syndrome
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Reason Child is >95th Percentile for Weight vs Height
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Obesity, oedema, achondroplasia, hydrocephaly
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Achondroplasia characteristics
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Genetic disorder Affects bone growth Arms / legs v. short but normal torso
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Reason child <5th percentile in BMI
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dehydration, recent malnutrition, Marfan's
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Energy intake definition
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Energy of food calculated by adding the protein, fat, CHO and fibre.
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1g protein
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17kJ
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1g fat
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37kJ
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1g CHO
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16kJ
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1g fibre
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8kJ
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1g alcohol
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29kJ
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Predictive Equations of Energy
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Harris Benedict - age / ht / wt Schofield - age wt
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Adjusted Body Weight Calculation
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[0.25 x (actual wt - weight @ BMI 25)] + weight @ BMI 25
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Energy Requirements for Sedentary + non-hypermetabolic
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100-125 kJ/kg
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Energy Requirements for Wt. Gain / mildly hypermetabolic
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125-145kJ/kg
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Energy Requirements for severely hypermetabolic
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145-165kJ/kg
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Protein Requirements Depending on Stress Levels
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Normal: 0.8 - 1.0 g/kg Minor stress: 1.0 - 1.2 Moderate stress/ dialysis: 1.2- 1.5 Severe stress/ major trauma/ burns: 1.5 - 2.0
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What does personal history include?
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General client info → age, gender, race, education, role in family
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What does medical history include?
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History of client, family history Should focus on things that have nutritional impact
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What does social history include?
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SES, living situation, community involvement
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Nausea / vomiting due to
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Antivirals, antifungals, antibacterials, penicillin, sulphamethoxazole, analgesics, narcotic analgesics
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Anti-emetics
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Granisetron, Tropisetron (for chemo / anaesthesia), Emetrol (for nausea / motion sickness), Maxalon / Dramamine ( gastroparesis), Domperidone (for gastroparesis), Metocloprimide (For chemo / radiation)
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Causes of overweight / obesity
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genetic, sociodemographic, neurobiological / endocrinological, microbiota
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Monogenic Obesity
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Extremely obese, absence of developmental delays, ↑ appetite, ↓satiety
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Syndromic obesity
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Clinically obese w/ mental retardation, developmental abnormalities
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Polygenic obesity
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General population, exposure to obesogenic environment required
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Medications Causing Obesity
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Hormones: corticosteroids, thyroid stimulating hormones Psychiatric medications: clozapine, lithium, beta adrenergic blockers, sodium valproate, tricyclic antidepressants Diabetic medications: Insulin, amitriptyline
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Risk Assessment for Obesity
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BP, thyroid function, HbA1c, BGL's, lipid profiles, nutrient deficiencies, BMI, WC, fatty liver test
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Obesity guidelines (1-5)
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1. Measure WC 2. Discuss readiness to change 3. Let know that small wt. loss improves health 4. Use multicomponent approaches 5. Refer appropriately 6. Support self- management approach and monitor
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Gastroplasty
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Gastric band reduces size of stomach, can be adjusted, filled with saline
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Gastric Bypass
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Reduces size through stapling procedure, requires lifelong follow-up by MD team, possible deficiencies in K⁺, Mg²⁺, folate, B12, Fe²⁺
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Orlistat / Xenical
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Inhibits GI lipase, ↓ 1/3 fat absorption, - GI side effects e.g feocal urgency, farting
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Long term weight management
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If weight regain - reassess energy intake and PA → reintervene with wt. loss strategies
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Diarrhoea side effects
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Loss of fluid / electrolytes / reduces nutrient absorption
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Diarrhoea caused by which medications?
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Anti-emetics, laxatives, diuretics, phosphate binders (Gaviscon), anti-thrombotic, cholesterol lowering, Metformin
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Anti-diarrhoea medications and side effects
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Immodium, codeine phosphate, lomotil Side effects: GI upset, constipation, dry mouth
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Constipation caused by which medications?
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Codeine, panadeine, tramadol, di-gesic Morphine, oxycontin, oxycodone, endone
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Laxative medications:
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benefibre, metamucil, senokot, sennetabs, senna plus, coloxyl with Senna, lactulose, movicol and agarol
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Motility Agent Medications and Side-effects
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Buscopan / atrobel / setacol Side effects: GI spasms, enuresis, dry mouth, thirst Alvercol / Zelmac Side Effects: IBS, constipation
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Diuretic Medication as well as treatment and side - effects
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Allopurinol for gout, reduces urea Side effects: sore throat / nausea Frusemide for oedema, ht Side effects: constipation, diarrhoea, high uric acid Spironolactone Side effects: cramps, hyperkalemia, hyponatremia
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Dry Mouth caused by which medications?
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Narcotic analgesics, calcium channel blockers, anti-reflux meds, diuretics, antihistamines, decongestants
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Inflamed gums caused by which medications?
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anti epileptics, erythromycin, Zoloft, anti-hypertensive, verapamil
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Inflamed tongue caused by which medications?
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Valium, Zoloft, prozac, Nurofen, Voltaren, Penicillin, anti-epileptics
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Oral thrush caused by which medications?
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inhaled steroids used for asthma, COPD, zyprexa, omeprazole (for GERD)
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Burning mouth caused by which medications?
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ACE inhibitors (captopril), penicillin, prozac, Zoloft, HRT
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Oral Ulcers caused by which medications?
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Bisphosphonates (for osteoporosis), NSAIDs, ACE inhibitors, cytotoxic meds, antipsychotics, tetracycline, erythromycin
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Altered taste or smell caused by which medications?
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ACE inhibitors, Metformin, antibiotics, anti-epileptics, lithium, anti-psychotics, tricyclic antidepressants, antihistamines, decongestants, colchicine, calcium channel blockers, cholesterol ↓ meds, methotrexate
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Absorption issues are caused by which medications?
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Metformin (B12) Proton pump inhibitors (B12, Ca²⁺) Anti-epileptics (Vit D, folic acid) Bile acids / resins (Fe²⁺, folate, Vit A) LT broad spectrum antibiotics (Vit K) Steroids (Ca²⁺) Diuretics (Na⁺, K⁺, Mg²⁺)
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Anti-reflux agents / antacids and their side effects
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Aluminium hydroxide: constipation, diarrhoea, renal stones, Ca²⁺, P malabsorption Calcium carbonate: GI upset, Malabsorption B12, Fe²⁺ Sodium bicarbonate: Excess Na, oedema Losec HP7: dry mouth, confusion, tiredness
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Blood Tests for Diabetes:
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Blood glucose levels Overnight glucose tolerance test (overnight fast) HbA1c (average)
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For every gram of glucose how much water?
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4g
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Hypoglycemia blood levels
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< 3.5mmol/L
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Hyperglycemia blood levels
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>10mmol/L (glucose >12mmol/L will be excreted in urine)
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Desirable Cholesterol levels
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<4.0 mmol/L
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Desirable LDL levels
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< 2.0 mmol/L
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Most prevalent micronutrient deficiency
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Iron deficiency anemia (infants, kids, pregnant women more at risk)
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Absolute iron deficiency
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Stores inadequate to support bone marrow requirements
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Functional iron deficiency
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Stores are adequate but iron can't be released or supplied to marrow fast enough to meet demand
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Tests for anaemia
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Iron studies + full blood count
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Changes in blood due to iron deficiency
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Serum Ferritin ↓ reflects iron stores Serum transferrin ↑ is total iron binding capacity Transferrin Saturation↓ = %iron binding capacity used
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Iron deficiency stages
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Stage 1: Iron stars gone, measured by ↓ serum ferritin Stage 2: Serum transferrin ↑ total iron capacity, transferrin saturation ↓ Stage 3: Hb production ↓
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Normal Hb levels
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M 13-18 g/L F 11-16 g/L
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Full blood count includes
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Hb concentration, Haematocrit, Erythrocyte count, RBC Protoporphyrin, Mean corpuscular volume, Mean corpuscular Hb, Mean corpuscular Hb Conc, Platelet count, Red cell distribution width,
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Main type of CVD's in Australia
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Coronary Heart Disease, Stroke, Heart Failure, Peripheral Vascular Disease
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Deaths due to CVD in Aus
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43,603 in 2013 30% all deaths 1 every 12 mins (although fallen by 8%)
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Most common form of CVD and outcomes?
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Failure of coronary arteries to supply blood to myocardium → angina, MI, stroke
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Peripheral Vascular Disease and outcomes
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Reduced circulation to other body parts (not heart or brain) Stenosed vessels → functional impairment, limb ischemia, ulcers, amputation
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Chronic / Congestive Heart Failure and outcomes. + mortality rate
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Underlying structural abnormality or cardiac dysfunction impairs ability of heart to fill or eject blood → shortness of breath, chronic fatigue, oedema, thickened mitral valve / chordae tendinae 5yr mortality rate 50%
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Nutrition Therapy in CHF
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Maintain healthy weight low salt diet to reduce oedema
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Stroke definition and outcomes
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artery supplying blood to brain suddenly becomes blocked or bleeds → impairment of speaking / thinking / movement
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Thrombogenesis
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Thrombus formed at site of large atheromatous plaque Can totally occlude vessel Can lead to MI, stroke
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Five Phases of Atherogenesis
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Phase 1: No symptoms Small fatty streaks Phase 2: Plaque with high LDL content that enters injured endothelial wall Phase 3: Lesion ruptures and forms non-occlusive thrombus Phase 4: Lesion ruptures and is occlusive Associated with angina, MI, stroke Phase 5: Fibrotic / occlusive lesions
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Aneurysm Definition and Risk Factors
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Result of weakened vessel wall Risk of rupture → internal bleeding Risk factors - obesity, diabetes, HTN, smoking, alcohol, age
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Metabolic Syndrome Caused by
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Any three of: Central obesity, hypertension, high BGL, high TG levels, low HDL
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Stress
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Activates neurohormonal response → ↑HR, ↑BP
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Cholesterol
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Waxy, transported by lipoproteins, produced in liver
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Hyperlipoproteinaemia
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One or more classes of lipoproteins elevated
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Desirable Triglyceride Values
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< 1.5 mmmol/L
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Diagnosis of hypertension
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2 or more blood pressure readings on 2 or more separate occasions
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CVD Diagnostics
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ECG, chest x-ray, ultrasound, coronary angioplasty, plasma enzymes
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CABG procedure
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Blocked arteries bypassed by section of pt's saphenous vein
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PTCA procedure
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Catheter with balloon directed to position of stenosis + gradually inflated
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Coronary stent procedure
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Stent mounted on delivery balloon which is dilated; adapts to artery and remains in place
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Lifestyle Modifications to Manage Hypertension
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Less salt, DASH diet (dietary approaches to stop hypertension), exercise, stop smoking, limit alcohol, have healthy weight
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DASH diet
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fruits, veges, whole-grains, poultry, fish + low-fat dairy foods Replace sat + trans fats with poly / monounsaturated fats Decrease salt <1500mg ↑Fibre
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How much can dietary intervention decrease LDL by?
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12%-16%
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Every 1mmol/L decrease in total cholesterol reduces risk of coronary event by how much?
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20%
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Top 3 sources of saturated fats
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Milk + dairy products (as well as ice cream) Biscuits, cakes, pastries Meat + poultry
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Sources of trans fats
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Margarine, snack foods, packaged baked goods + fried fast food
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Sources of monounsaturated fats
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Avocados, nuts, flaxseed, sunflower seed, olives, tahini, oils from plants or seeds, lean meats, eggs
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Omega-6 includes which acids and how much to consume?
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Linoleic acid (essential) Arachidonic acid (non-essential, pro-inflammatory) 5%-10% total energy intake
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Omega-3 includes which essential acids?
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ALA, EPA, DHA
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Desirable ratio of N-3 and N-6
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< 5:1 is ideal
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EPA Function
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Replaces arachidonic acid in platelets, ↓ thromboxane production → inhibits platelet aggregation → slows growth of atherosclerotic plaques
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Sources of Omega-6
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Sunflower oils, margarines, tahini, flaxseed, nuts
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Sources of Omega-3
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ALA - walnuts, pecans, seed oils EPA + DHA - oily fish Recommended 2-3 fish meals weekly
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Recommendations to Lower Risk of CHD
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2-3 serves of fish (250-500mg n-3), 1g ALA / day
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Daily serve of nuts
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30g
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Fibre is classed as? Does it lower or increase cholesterol?
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pectins, gums, mucilages Lowers cholesterol
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Salt recommendation
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<4g salt/d in hypertension <6g salt/d in general population
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How much of our body weight is water?
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65%
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Mediterranean diet includes which food groups?
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rich in legumes, red wine, fish and fruit Low in meat
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Homocysteine / folate link with CHD?
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High levels = toxic effects on vascular endothelium By-product of folate pathway if deficiency exists
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Sterols role in CHD?
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Found in plants, similar to cholesterol but poorly absorbed ↓ cholesterol by competition 2-3g or 2tsp / day
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Antioxidant role in CHD?
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Prevents lipoprotein oxidation + restores normal endothelial function + protects myocardium from ischaemic damage
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