Nursing care of the newborn with special needs – Flashcards

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Appropriate for gestational age (AGA)
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a newborn weight that falls within the 10th-90th percentile for that particular gestational age -80% of newborns, lowest risk
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Small for gestational age (SGA)
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newborns typically weight less than 2500gr (5 lb 8oz) at term due to less growth than expected in utero; at or below 10th percentile as correlated with the number of weeks of gestation
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Large for gestational age (LGA)
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newborns whose birthweight is above the 90th percentile on a growth chart and who weigh more than 4000gr (8 lb 13oz) at term due to accelerated growth for length of gestation; may be preterm, postterm, or full term
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Marginal weights at birth and of any gestational age
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Low birth weight: less than 2,500 g (5.5 lb) Very low birth weight: less than 1,500 g (3 lb 5 oz) Extremely low birth weight: less than 1,000 g (2 lb 3 oz)
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What condition does SGA newborns have?
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intrauterine growth restriction (IUGR); intrinsic or environmental causes to SGA
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SGA infants are associated with
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-increased neonatal morbidity and mortality -short stature, -cardiovascular disease, -insulin resistance, - diabetes mellitus type 2, -dyslipidemia, -end-stage renal disease in adulthood. -decreased levels of intelligence and cognition
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Potential factors contributing to the birth of SGA newborns
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Maternal causes: Chronic hypertension Diabetes mellitus with vascular disease Autoimmune diseases Living at a high altitude (hypoxia) Smoking or exposure to passive smoke Periodontal disease of the mouth Maternal age of >20 or <35 years old Failure to seek any prenatal care Substandard living conditions Low socioeconomic status Abuse and violence Placental factors: Abnormal cord insertion Chronic abruption Decreased surface area, infarction Decreased placental weight Placenta previa Placental insuficiency Fetal factors: Trisomy 13, 18, and 21 Turner's syndrome Chronic fetal infection (cytomegalovirus [CMV], rubella, syphilis, toxoplasmosis) Congenital anomalies (heart, diaphragmatic hernia, tracheoesophageal fistula) Radiation exposure Multiple fetal gestation
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Symmetric IUGR
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-fetuses with equally poor growth rates of the brain, the abdomen, and the long bones and is thought to result from an early global insult -occurs at less than 28 weeks -poor prognosis
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Asymmetric IUGR
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refers to infants whose brain growth is spared compared to their abdomen and internal organs -occurs at more than 28 weeks -better prognosis
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Glucose concentration for hypoglycemia
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at or below 40 mg/dL
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Typical characteristics of SGA
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head larger than rest of body (asymmetric), wasted appearance of extremities, reduced subcutaneous fat stores, decreased amount of breast tissue, sunken abdomen, wide skull sutures secondary to inadequate bone growth, poor muscle tone over buttocks and cheeks, loose and dry skin that appears oversized, thin umbilical cord
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Signs of hypoglycemia
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lethargy, tachycardia, respiratory distress, jitteriness, drowsiness, poor feeding, feebrl sucking, hypothermia, temperature instability, diaphoresis, weak cry, seizures, hypotonia, blood glucose levels <40 for term newborns, <20 for preterm newborns
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Who is at risk for polycythemia (elevated RBC count)?
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SGA newborns, infants of diabetic mothers, and multiple births; screening should be done at 2, 12, and 24 hrs of age
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Hyperviscosity
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increased resistance to blood flow and decreased oxygen delivery; can cause hypoglycemia, CNS abnormalities, decreased renal function, cardiorespiratory distress, and coagulation disorders
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Polycythemia
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- venous hematocrit above 65%.
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Maternal factors that increase the chance of bearing an LGA
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diabetes mellitus or glucose intolerance, multiparity, prior history of a macrosomic infant, postdates gestation, maternal obesity, male fetus, and genetics
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Risk for LGA newborns
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shoulder dystocia, clavicular fractures, facial palsies, cesarean birth, asphyxia, hypoglycemia, polycythemia
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Characteristics of a LGA newborn
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large body and appears plump and full-faced, increase in body size is proportional, however, head circumstance and body length are in the upper limits of intrauterine growth; poor motor skills and have difficulty in regulating behavioral states; more difficult to arouse to a quiet alert state
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What do you assess in an LGA newborn at birth ?
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traumatic birth injuries such as fractured clavicles, brachial palsy, facial paralysis, phrenic nerve palsy, skull fractures, or hematomes; neurologic abnormalities such as immobility of the upper arm
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What are LGA at risk for developing?
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hypoglycemia related to early depletion of glycogen stores in their liver
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Clinical signs of a hypoglycemia newborn
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often subtle and include lethargy, apathy, drowsiness, irritability, tachypnea, weak cry, temperature instability, jitteriness, seizures, apnea, bradycardia, cyanosis or pallor, feeble suck and poor feeding, hypotonia, and coma
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Disorders present in LGA newborns
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septicemia, severe respiratory distress, congenital heart disease, polycythemia and hyperbilirubinemia
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Treatment for hypoglycemic newborns
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asymptomatic- supervised breast-feeding or formula; symptomatic- continuous infusion of parental dextrose
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Nursing interventions for stabilizing the LGA newborn
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monitor blood glucose levels within 30 minutes of birth and repeat the screening every hour, recheck levels before feedings; to prevent hypoglycemia, initiate feedings with formula or breast milk, with intravenous glucose supplementation as needed; increase fluid volume to decrease blood viscosity
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How do you treat hyperbilirubinemia?
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hydration, early feedings, and phototherapy
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Typical complications associated with a postterm newborn
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perinatal asphyxia (caused by placental aging or oligohydramnios [decreased amniotic fluid]), hypoglycemia (caused by acute episodes of hypoxia related to cord compression which exhausts carbohydrate reserves), hypothermia (caused by loss of fat), and polycythemia (caused by an increased production of RBCs)
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What do you monitor in a postterm newborn?
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blood glucose levels (IV dextrose 10% and/or early initiaion of feedings), skin temperature, RR characteristics, results of blood studies (ABGs and serum bilirubin levels and neurologic status), hypothermia
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Clinical findings in postterm newborns - postmature infant
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dry, cracked, peeling, wrinkled skin, vernix caseosa and lanugo are absent, long, thin extremities, creases that cover the entire soles of the feet, wide-eyed, alert expression, abundant hair on scalp, thin umbilical cord, limited vernix and lanugo, meconium-stained skin and fingernails, long nails
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What condition is the leading cause of death within the first month of life and the second leading cause of all infant deaths?
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prematurity
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Common complications in preterm newborns
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respiratory distress syndrome, periventricular-intraventricular hemorrhage, bronchopulmonary dysplasia, retinopathy of prematurity, hyperbilirubinemia, anemia, necrotizing, enterocolitis, hypoglycemia, infection or septicema, delayed growth and development, and mental or motor delays
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Leading cause of preterm births
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Infections/inflammation. Maternal or fetal stress. Bleeding. Stretching.
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Preterm infants at high risk for
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-neurodevelopmental disorders cerebral palsy, intellectual disability, intraventricular hemorrhage, congenital anomalies, neurosensory impairment, behavioral problems, high frequency attention problems, psychiatric disorders, and chronic lung disease
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Cardiovascular problems in preterm newborns
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blood flow may bypass the lungs, increased incidence of congenital anomalies associated with continued fetal circulation, impaired regulation of BP may cause fluctuations, intracranial hemorrhage can occur
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Gastrointestinal problems in preterm newborns
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lack the coordination to maintain the suck, swallow, and breathing regimen necessary for sufficient calorie and fluid intake to support growth, hypoxia stops blood from reaching the heart and brain, ischemia and damage to the intestinal wall, small stomach, weak abdominal muscles, limited ability to digest proteins and absorb nutrients
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Problems that affect the preterm newborn's breathing ability
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-surfactant deficiency, leading to the development of respiratory distress syndrome, -unstable chest wall, leading to atelectasis, -apnea, - increased risk for obstruction, -inability to clear fluid from passages, leading to transient tachypnea
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Renal system problems in preterm newborns
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reduces ability to concentrate urine and slows the GFR--> risk for fluid retention, with fluid and electrolyte disturbances, drug toxicity
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Immune system problems in preterm newborns
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deficiency in IgG, impaired ability to manufacture antibodies to fight infection, thin skin and fragile blood vessels provide a limited protective barrier, adding to the increased risk for infection
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Central nervous system problems in newborns
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difficulty in temperature regulation and maintaining stability- inadequate amounts of fat, lack of muscle tone and flexion, inadequate brown fat, limited muscle mass activity, inability to shiver to generate heat, and an immature temperature-regulating center in the brain; especially susceptible to hypoglycemia due to immature glucose control mechanisms, decreased glucose stores, and reduced ketone bodies
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Maternal risk factors associated with preterm birth
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previous preterm delivery, low socioeconomic status, preeclampsia, hypertension, poor maternal nutrition, smoking, multiple gestation, infection, advanced maternal age, and substance abuse
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Asphyxia (preterm)
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a newborn who fails to establish adequate, sustained respiration after birth due to impairment in gas exchange resulting in a decrease in oxygen in the blood (hypoxemia) and an excess of carbon dioxide or hypercapnia that leads to acidosis
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Common physical characteristics of preterm infants
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birthweight <5.5 lbs, scrawny appearance, head larger than chest circumference, poor muscle tone and flexion, minimal subcutaneous fat, undescended testes, plentiful lanugo (soft, downy hair), especially over the face and back, poorly formed ear pinna, with soft, pliable cartilage, fused eyelids, soft and spongy scull bones, few creases in the palms and soles, thin transparent skin with visible veins, vernix caseosa
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Why do preterms have a limited ability to retain air?
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insufficient surfactant; therefore, newborns develop atelectasis quickly without alveoli stabilization; inability to initiate and establish respirations leads to hypoxemia and ultimately hypoxia (decreased oxygen), acidosis (decreased pH), and hypercarbia (increased carbon dioxide)
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Determine the need for resuscitation by following three questions
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what is the gestational age of this newborn and was the amniotic fluid clear of meconium?, is the newborn breathing or crying now?, does the newborn have good muscle tone?,
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Routine care for newborns who need resuscitation
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provide warmth, clear the airway, dry the newborn, and assess the color; resuscitation measures are continued until the newborn has a pulse above 100 bpm, a good cry or good breathing efforts, and a pink tongue (good oxygen to the brain)
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A for Airway (ABCs of Newborn resuscitation)
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place infant's head in "sniffling" position, suction mouth, then nose, suction trachea is meconium-stained and newborn is NOT vigorous (strong respiratory effort, good muscle tone, and heart rate>100 bpm)
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Sequence of resuscitating actions
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1. Stabilization. Dry the newborn thoroughly with a warm towel; position the head in a neutral position to open the airway; clear the airway with a bulb syringe or suction catheter; and stimulate breathing. At times, rubbing the newborn with a dry towel may be all that is needed to stimulate respiration. 2. Ventilation 3. Chest compression 4. Administration of epinephrine and/or volume expansion
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B for Breathing (ABCs of Newborn resuscitation)
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use positive-pressure ventilation (PPV) for apnea, grasping, or pulse <100 bpm, ventilate at rate of 40-60 breaths/minute, listen for raising heart rate, audible breath sounds, look for slight chest movement with each breath, use carbon dioxide detector after intubation
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C for Circulation (ABCs of Newborn resuscitation)
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start compression if heart rate is <60 after 30 seconds of effective PPV, give 3 compressions: 1 breath every 2 seconds, compress one third of the anterior-posterior diameter of the chest
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What causes respiratory distress?
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deficiency of surfactant, retained fluid in the lungs (wet lung syndrome), meconium aspiration, pneumonia, hypothermia, or anemia
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How do maintain thermal regulation immediately after birth?
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dry the newborn with a warmed towel and then place him or her in a second warm, dry towel before performing the assessment; "kangaroo care"- stable newborns placed on mother's chest
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D is for Drugs
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Give epinephrine if heart rate is ,60 after 30 seconds of compressions and ventilation. -Caution: Epinephrine dosage is different for endotracheal and IV routes!
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What prevents a newborn from attaining good thermal regulation?
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inadequate supply of brown fat because he or she left the uterus before it was adequate, decreased muscle tone, which reduces the amount of skin exposed to a cooler environment, large body surface areas compared to weight, which allows an increased transfer of heat from their bodies to the environment
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Retinopathy of prematurity (ROP)
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- Major cause of blindness in preterm newborns in the past - Use O2 cautiously
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Characteristics of a newborn that is having problems with thermal regulation
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cool to cold to the touch, hands, feet, and tongue may appear cyanotic, respirations are shallow or slow, or signs of respiratory distress are present, lethargic and hypotonic, feeds poorly, and has a feeble cry, blood glucose levels are low, leading to hypoglycemia, due to the energy expended to keep warm
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Signs of hyperthermia
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tachycardia, tachypnea, apnea, warm to touch, flushed skin, lethargy, weak or absent cry, and CNS depression
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Signs of dehydration
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decrease in urinary output, sunken fontanels, temperature elevation, lethargy, and tachypnea
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Signs of cold stress
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-respiratory distress, -central cyanosis, -hypoglycemia, -lethargy, -weak cry, -abdominal distention, - -apnea, -bradycardia, -acidosis.
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Clinical manifestations of a infection in a preterm newborn
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apnea, diminished activity, poor feeding, temperature instability, respiratory distress, seizures, tachycardia, hypotonia, irritability, pallor, jaundice, and hypoglycemia
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Signs of pain in a preterm newborn
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sudden high-pitched cry, facial grimace with furrowing of brown and quivering chin, increased muscle tone, oxygen desaturation, body posturing, such as squirming, kicking, arching, limb withdrawal and thrashing movements, increase in heart rate, blood pressure, pulse, and respirations, fussiness and irritability
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Goals of pain management
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preventing, limiting, or avoiding noxious stimuli
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Nonpharmacologic techniques to reduce pain the preterm newborn
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gentle handling, rocking, caressing, cuddling, and massaging; rest periods before and after painful procedures; kangaroo care (skin-to-skin contact) during procedure; breastfeeding, if able, to reduce pain from minor procedures; use of a facilitated tuck (holding arms and legs in a flexed position); nonnutritive sucking (pacifier dipped in sucrose) prior to procedure; minimal use of tape, with gentle removal to avoid skin tears; warm blankets; reduce stimuli such as noise and bright lights; distraction, such as with colored objects or mobiles
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What is a perinatal loss?
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any pregnancy loss and/or neonatal death up to 1 month of age
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Promoting growth and development for preterm newborns
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promote rest and conserve the infant's energy, flexed positioning to stimulate in utero positioning, reduce noise and visual stimulation, kangaroo care to promote skin-to-skin sensation, placement of twin in the same isolette or open crib to reduce stress, parent-infant bonding,
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Activities to promote self-regulation and state regulation
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surrounding the newborn with nesting rolls/devices, swaddling with a blanket to maintain the flexed position, providing sheepskin or a waterbed to stimulate the uterine environment, providing nonnutritive sucking, providing objects to grasp (comforts the newborn)
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Health risks for the late preterm infant
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Respiratory system dysfunction—due to surfactant deficiency leading to distress Glycemic instability—due to increased energy demands needed for temperature regulation and increased respiratory effort, which cause the blood glucose to remain low for prolonged periods of time Jaundice—due to feeding dificulties and inability of liver to conjugate bilirubin Inadequate oral intake—due to a decreased ability to suck and swallow Susceptibility to infection—due to immaturity of the immune system Neurologic immaturity—due to reduced cortical development, which occurs during the 34th and 40th week of gestation
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